Evidence map›Paper›PMID 35844617›Full record

ReviewFrontiers in immunology2022

Endocrine Autoimmunity in Pregnancy.

Renata Świątkowska-Stodulska, Agata Berlińska, Katarzyna Stefańska, Maciej Zieliński, Sebastian Kwiatkowski, Joanna Połom, Elżbieta Andrysiak-Mamos, Piotr Wydra, Krzysztof Sworczak

Open access · goldAbstract readReview
In one paragraph

Review in Frontiers in immunology, 2022. The graph could read no effect estimate from its abstract, so it casts no vote on the map. Cited by 5 papers.

0numbers the graph read from it
0cells of the map it votes in
5citing papers in PubMed
2.0field-weighted citation impact, top 12% of its field
1 · What the graph read from it

What it found

Each row is one number read from the abstract, on the scale the paper reported it, with its interval. Left of the dashed line favours the treatment, right favours the comparator. Under each row is the sentence it came from. New to these charts? A ten-minute tutorial.

The abstract states no effect estimate the extractor could read, or names no intervention and outcome on the map, so this paper lights no cell and moves no belief. It is still indexed, cited and linked below.

2 · The registry

The trial behind it

Trials whose registry record cites this paper, or whose number appears in the abstract. A trial that started after this paper was published is citing it as background, not reporting it.

Neither the registry nor the abstract names a trial number. If this is a trial report, that itself is worth knowing.

3 · Its place in the literature

Who cites it

5 citing papers in PubMed, 16 citations in OpenAlex.

  1. Article
  2. SARS-CoV-2 and thyroid diseases.Journal of translational autoimmunity · 2023
    Review
  3. Article
  4. Review
  5. Article
4 · The record

Corrections and comments

PubMed lists nothing against this paper. Absence here is not a guarantee, only a check that was made.

5 · Who and what money

Authors and funding

9 authors at 3 institutions in 1 country.

Renata Świątkowska-StodulskaDepartment of Endocrinology and Internal Medicine, Faculty of Medicine, Medical University of Gdańsk, Gdańsk, Poland.
Agata BerlińskaDepartment of Endocrinology and Internal Medicine, Faculty of Medicine, Medical University of Gdańsk, Gdańsk, Poland.
Katarzyna StefańskaDivision of Gynecology and Obstetrics, Faculty of Medicine, Medical University of Gdańsk, Gdańsk, Poland.
Maciej ZielińskiDepartment of Medical Immunology, Faculty of Medicine, Medical University of Gdańsk, Gdańsk, Poland.
Sebastian KwiatkowskiDepartment of Obstetrics and Gynecology, Pomeranian Medical University of Szczecin, Szczecin, Poland.
Joanna PołomDepartment of Internal Medicine, Connective Tissue Diseases and Geriatrics, Faculty of Medicine, Medical University of Gdańsk, Gdańsk, Poland.
Elżbieta Andrysiak-MamosDepartment of Endocrinology, Metabolic Diseases and Internal Diseases Pomeranian Medical University, Szczecin, Poland.
Piotr WydraDepartment of Medical Immunology, Faculty of Medicine, Medical University of Gdańsk, Gdańsk, Poland.
Krzysztof SworczakDepartment of Endocrinology and Internal Medicine, Faculty of Medicine, Medical University of Gdańsk, Gdańsk, Poland.
Gdańsk Medical University · PLPomeranian Medical University · PLUniversity of Szczecin · PL

Funding

No grant is acknowledged in the PubMed record.

6 · The paper itself

Abstract

Human gestation leads to a number of physiological alterations which peak at the development of placentta known for, among many other functions, being a transient but highly potent endocrine organ. Hormonal activity of placenta is marked by its ability to continuously produce and secrete high levels of progesterone. Progesterone guards the well-being of the fetoplacental unit throughout the gestation and one of the proposed mechanisms of this principle involves the development of local and systemic immune tolerance mainly due to impediment of CD4+ lymphocyte activation. However, though these alterations are present and well-established, autoimmunity is not entirely rare and a wide spectrum of diseases can continue, or develop

Indexed as

Autoimmune DiseasesGraves DiseaseAutoimmunityFemaleHumansPlacentaPregnancyProgesteroneProgesteroneAddison’s diseaseautoimmune thyroid disordergestationGraves’ diseaseHashimoto’s diseaseHashimoto’s thyroiditispostpartum thyroiditispregnancy

Identifiers

PMID35844617
PMCPMC9277138
OpenAlexW4285818955

What OpenQuestion holds

Textmetadata
LicenceCC BY
Read underepoch 390

Registered trials

None linked

Read under generation 80e0d062 · epoch 390. Bibliography from PubMed, PubMed Central and OpenAlex; grants from NIH RePORTER; trial links from ClinicalTrials.gov; estimates, votes and beliefs from the OpenQuestion graph.