Evidence map›Paper›PMID 35838945›Full record

ArticleProbiotics and antimicrobial proteins2023

Impact of Oral Probiotics in Amelioration of Immunological and Inflammatory Responses on Experimentally Induced Acute Diverticulitis.

Maha G Soliman, Hanaa A Mansour, Wedad A Hassan, Eman Shawky

Open access · hybridAbstract read
In one paragraph

Article in Probiotics and antimicrobial proteins, 2023. The graph could read no effect estimate from its abstract, so it casts no vote on the map. Cited by 4 papers.

0numbers the graph read from it
0cells of the map it votes in
4citing papers in PubMed
1.3field-weighted citation impact, top 20% of its field
1 · What the graph read from it

What it found

Each row is one number read from the abstract, on the scale the paper reported it, with its interval. Left of the dashed line favours the treatment, right favours the comparator. Under each row is the sentence it came from. New to these charts? A ten-minute tutorial.

The abstract states no effect estimate the extractor could read, or names no intervention and outcome on the map, so this paper lights no cell and moves no belief. It is still indexed, cited and linked below.

2 · The registry

The trial behind it

Trials whose registry record cites this paper, or whose number appears in the abstract. A trial that started after this paper was published is citing it as background, not reporting it.

Neither the registry nor the abstract names a trial number. If this is a trial report, that itself is worth knowing.

3 · Its place in the literature

Who cites it

4 citing papers in PubMed, 6 citations in OpenAlex.

  1. Review
  2. Review
  3. Article
  4. Article
4 · The record

Corrections and comments

PubMed lists nothing against this paper. Absence here is not a guarantee, only a check that was made.

5 · Who and what money

Authors and funding

4 authors at 2 institutions in 1 country.

Maha G SolimanDepartment of Zoology, Faculty of Science, Al-Azhar University, Cairo, Egypt.
Hanaa A MansourDepartment of Pharmacology, National Organization for Drug Control and Research (NODCAR), Giza, Egypt.
Wedad A HassanDepartment of Pharmacology, National Organization for Drug Control and Research (NODCAR), Giza, Egypt.
Eman ShawkyDepartment of Zoology, Faculty of Science, Al-Azhar University, Cairo, Egypt. Eshawky_sci@yahoo.com.ORCID http://orcid.org/0000-0002-3321-0384
Al-Azhar University · EGNational Organization for Drug Control and Research · EG

Funding

No grant is acknowledged in the PubMed record.

6 · The paper itself

Abstract

Acute diverticulitis is inflammation of a colon diverticulum; it represents a major cause of morbidity and mortality. The alteration of gut microbiota contributes to the promotion of inflammation and the development of acute diverticulitis disease. Probiotics can modify the gut microbiota, so they are considered a promising option for managing diverticulitis disease. This study aimed to investigate the potential protective effect of probiotics, alone or in combination with amoxicillin, on the experimentally induced model of acute diverticulitis disease. Forty-two rats were divided into seven groups as follows: control group: received water and food only; DSS group: received 3% dextran sulfate sodium (DSS) daily for 7 days; LPS group: injected with lipopolysaccharide (LPS) enema at the dose of (4 mg/kg); probiotics group: treated with probiotics (Lactobacillus acidophilus and Bifidobacterium lactis) each of which (4 × 10

Indexed as

ColitisDiverticulitisProbioticsAmoxicillinAnimalsDisease Models, AnimalInflammationLipopolysaccharidesRatsAmoxicillinLipopolysaccharidesAcute diverticulitisInflammatory responsesLipopolysaccharideProbiotics

Identifiers

PMID35838945
PMCPMC10491525
OpenAlexW4285592511

What OpenQuestion holds

Textmetadata
LicenceCC BY
Read underepoch 390

Registered trials

None linked

Read under generation 80e0d062 · epoch 390. Bibliography from PubMed, PubMed Central and OpenAlex; grants from NIH RePORTER; trial links from ClinicalTrials.gov; estimates, votes and beliefs from the OpenQuestion graph.