Evidence map›Paper›PMID 35822241›Full record

ArticleAutophagy2023

Increased

So Jung Park, Rebecca A Frake, David C Rubinsztein

Open access · hybridAbstract read
In one paragraph

Article in Autophagy, 2023. The graph could read no effect estimate from its abstract, so it casts no vote on the map. Cited by 8 papers.

0numbers the graph read from it
0cells of the map it votes in
8citing papers in PubMed
3.7field-weighted citation impact, top 6% of its field
1 · What the graph read from it

What it found

Each row is one number read from the abstract, on the scale the paper reported it, with its interval. Left of the dashed line favours the treatment, right favours the comparator. Under each row is the sentence it came from. New to these charts? A ten-minute tutorial.

The abstract states no effect estimate the extractor could read, or names no intervention and outcome on the map, so this paper lights no cell and moves no belief. It is still indexed, cited and linked below.

2 · The registry

The trial behind it

Trials whose registry record cites this paper, or whose number appears in the abstract. A trial that started after this paper was published is citing it as background, not reporting it.

Neither the registry nor the abstract names a trial number. If this is a trial report, that itself is worth knowing.

3 · Its place in the literature

Who cites it

8 citing papers in PubMed, 24 citations in OpenAlex.

  1. Review
  2. Article
  3. Review
  4. Article
  5. Article
  6. Article
  7. Role of angiomotin family members in human diseases (Review).Experimental and therapeutic medicine · 2024
    Review
  8. Article
4 · The record

Corrections and comments

PubMed lists nothing against this paper. Absence here is not a guarantee, only a check that was made.

5 · Who and what money

Authors and funding

3 authors at 2 institutions in 1 country.

So Jung ParkDepartment of Medical Genetics, Cambridge Institute for Medical Research, Wellcome Trust/MRC Building, Cambridge Biomedical Campus, Cambridge, UK.
Rebecca A FrakeOxford University Hospitals NHS Foundation Trust, John Radcliffe Hospital, Headley Way, Oxford, UK.
David C RubinszteinDepartment of Medical Genetics, Cambridge Institute for Medical Research, Wellcome Trust/MRC Building, Cambridge Biomedical Campus, Cambridge, UK.
Wellcome Trust · GBUniversity of Oxford · GB

Funding

Medical Research Council UKDRI-2002Wellcome Trust 095317/Z/11/ZWellcome Trust 100140/Z/12/Z
6 · The paper itself

Abstract

Impaired autophagosome formation and reduced flux through the macroautophagy/autophagy pathway occurs outside the brain as part of normal aging in various species. We recently identified autophagic decline in mouse brain tissue dependent on aging. This sits alongside significantly increased expression of the

Indexed as

AutophagyTrans-ActivatorsAdaptor Proteins, Signal TransducingAgingAnimalsBrainHumansIntracellular Signaling Peptides and ProteinsMiceTranscriptional Coactivator with PDZ-Binding Motif ProteinsAdaptor Proteins, Signal TransducingIntracellular Signaling Peptides and ProteinsTrans-ActivatorsTranscriptional Coactivator with PDZ-Binding Motif ProteinsWWTR1 protein, humanWwtr1 protein, mouseAutophagybrain agingSORBS3vinexinYAP1-WWTR1/TAZ

Identifiers

PMID35822241
PMCPMC9980698
OpenAlexW4285396441

What OpenQuestion holds

Textmetadata
LicenceCC BY
Read underepoch 390

Registered trials

None linked

Read under generation 80e0d062 · epoch 390. Bibliography from PubMed, PubMed Central and OpenAlex; grants from NIH RePORTER; trial links from ClinicalTrials.gov; estimates, votes and beliefs from the OpenQuestion graph.