Evidence map›Paper›PMID 35796914›Full record

ArticleNeurochemical research2022

Aluminum Induced Necroptosis of PC12 Cells via TNFR1-RIP1/RIP3 Signalling Pathway.

Yue Zhou, Qin Feng, Yaqin Li, Qun Liu, Xiaoyan Zhao, Chunmei Duan, Jingsi Zhang, Qiao Niu

Abstract read
PubMed Publisher
In one paragraph

Article in Neurochemical research, 2022. The graph could read no effect estimate from its abstract, so it casts no vote on the map. Cited by 4 papers.

0numbers the graph read from it
0cells of the map it votes in
4citing papers in PubMed
0.9field-weighted citation impact, top 29% of its field
1 · What the graph read from it

What it found

Each row is one number read from the abstract, on the scale the paper reported it, with its interval. Left of the dashed line favours the treatment, right favours the comparator. Under each row is the sentence it came from. New to these charts? A ten-minute tutorial.

The abstract states no effect estimate the extractor could read, or names no intervention and outcome on the map, so this paper lights no cell and moves no belief. It is still indexed, cited and linked below.

2 · The registry

The trial behind it

Trials whose registry record cites this paper, or whose number appears in the abstract. A trial that started after this paper was published is citing it as background, not reporting it.

Neither the registry nor the abstract names a trial number. If this is a trial report, that itself is worth knowing.

3 · Its place in the literature

Who cites it

4 citing papers in PubMed, 12 citations in OpenAlex.

  1. Review
  2. Review
  3. Article
  4. Article
4 · The record

Corrections and comments

PubMed lists nothing against this paper. Absence here is not a guarantee, only a check that was made.

5 · Who and what money

Authors and funding

8 authors at 1 institution in 1 country.

Yue ZhouDepartment of Occupational Health, School of Public Health, Shanxi Medical University, Taiyuan, China.
Qin FengDepartment of Occupational Health, School of Public Health, Shanxi Medical University, Taiyuan, China.
Yaqin LiDepartment of Occupational Health, School of Public Health, Shanxi Medical University, Taiyuan, China.
Qun LiuDepartment of Occupational Health, School of Public Health, Shanxi Medical University, Taiyuan, China.
Xiaoyan ZhaoDepartment of Occupational Health, School of Public Health, Shanxi Medical University, Taiyuan, China.
Chunmei DuanDepartment of Occupational Health, School of Public Health, Shanxi Medical University, Taiyuan, China.
Jingsi ZhangDepartment of Occupational Health, School of Public Health, Shanxi Medical University, Taiyuan, China.
Qiao NiuDepartment of Occupational Health, School of Public Health, Shanxi Medical University, Taiyuan, China. niuqiao55@163.com.ORCID http://orcid.org/0000-0003-0015-3341
Shanxi Medical University · CN

Funding

National Natural Science Foundation of China 81430078National Natural Science Foundation of China 81872599
6 · The paper itself

Abstract

In addition to apoptosis, it has also been reported that aluminum (Al) causes necroptosis, a new form of programmed necrosis, which has recently been discovered, in nerve cells, but its molecular mechanism is not elucidated. In order to explore the answer, in this study, we apply for this method that after PC12 cells were exposed to maltol aluminum [200 μM Al(mal)

Indexed as

NecroptosisReceptor-Interacting Protein Serine-Threonine KinasesAluminumAnimalsApoptosisNecrosisPC12 CellsRatsReceptors, Tumor Necrosis Factor, Type IAluminumReceptor-Interacting Protein Serine-Threonine KinasesReceptors, Tumor Necrosis Factor, Type IMaltol aluminumNecroptosisReceptor interaction proteins 1Receptor interaction proteins 3Tumour necrosis factor receptor 1

Identifiers

PMID35796914
OpenAlexW4284695629

What OpenQuestion holds

Textmetadata
Read underepoch 390

Registered trials

None linked

Read under generation 80e0d062 · epoch 390. Bibliography from PubMed, PubMed Central and OpenAlex; grants from NIH RePORTER; trial links from ClinicalTrials.gov; estimates, votes and beliefs from the OpenQuestion graph.