Evidence map›Paper›PMID 35743026›Full record

ReviewInternational journal of molecular sciences2022

Ferroptosis: A Potential Therapeutic Target in Acute Kidney Injury.

Keiko Hosohata, Tanisorn Harnsirikarn, Susama Chokesuwattanaskul

Open access · goldAbstract readReview
In one paragraph

Review in International journal of molecular sciences, 2022. The graph could read no effect estimate from its abstract, so it casts no vote on the map. Cited by 40 papers.

0numbers the graph read from it
0cells of the map it votes in
40citing papers in PubMed
8.8field-weighted citation impact, top 1% of its field
1 · What the graph read from it

What it found

Each row is one number read from the abstract, on the scale the paper reported it, with its interval. Left of the dashed line favours the treatment, right favours the comparator. Under each row is the sentence it came from. New to these charts? A ten-minute tutorial.

The abstract states no effect estimate the extractor could read, or names no intervention and outcome on the map, so this paper lights no cell and moves no belief. It is still indexed, cited and linked below.

2 · The registry

The trial behind it

Trials whose registry record cites this paper, or whose number appears in the abstract. A trial that started after this paper was published is citing it as background, not reporting it.

Neither the registry nor the abstract names a trial number. If this is a trial report, that itself is worth knowing.

3 · Its place in the literature

Who cites it

40 citing papers in PubMed, 64 citations in OpenAlex.

  1. Article
  2. Article
  3. Article
  4. Review
  5. Article
  6. Effects of nTiOToxics · 2026
    Article
  7. Article
  8. Article
  9. Article
  10. Article
  11. Journal of clinical biochemistry and nutrition · 2025
    Article
  12. Review
  13. Article
  14. Article
  15. Review
  16. Article
  17. Article
  18. Article
  19. Article
  20. Review
4 · The record

Corrections and comments

PubMed lists nothing against this paper. Absence here is not a guarantee, only a check that was made.

5 · Who and what money

Authors and funding

3 authors at 3 institutions in 2 countries.

Keiko HosohataEducation and Research Center for Clinical Pharmacy, Osaka Medical and Pharmaceutical University, Osaka 569-1094, Japan.ORCID 0000-0001-8882-8846
Tanisorn HarnsirikarnDivision of Nephrology, Department of Internal Medicine, Bhumibol Adulyadej Hospital, Royal Thai Air Force, Bangkok 10220, Thailand.
Susama ChokesuwattanaskulDepartment of Internal Medicine, Charoenkrung Pracharak Hospital, Bangkok 10120, Thailand.
Bhumibol Adulyadej Hospital · THCharoenkrung Pracharak Hospital · THOsaka University of Pharmaceutical Sciences · JP

Funding

No grant is acknowledged in the PubMed record.

6 · The paper itself

Abstract

Ferroptosis is a recently recognized form of nonapoptotic cell death that is triggered by reactive oxidative species (ROS) due to iron overload, lipid peroxidation accumulation, or the inhibition of phospholipid hydroperoxidase glutathione peroxidase 4 (GPX4). Recent studies have reported that ferroptosis plays a vital role in the pathophysiological process of multiple systems such as the nervous, renal, and pulmonary systems. In particular, the kidney has higher rates of O

Indexed as

Acute Kidney InjuryFerroptosisReperfusion InjuryCell DeathHumansLipid PeroxidationReactive Oxygen SpeciesReactive Oxygen Speciesacute kidney injuryferroptosislipid peroxidationoxidative stressreactive oxygen/nitrogen species

Identifiers

PMID35743026
PMCPMC9223765
OpenAlexW4282822303

What OpenQuestion holds

Textmetadata
LicenceCC BY
Read underepoch 390

Registered trials

None linked

Read under generation 80e0d062 · epoch 390. Bibliography from PubMed, PubMed Central and OpenAlex; grants from NIH RePORTER; trial links from ClinicalTrials.gov; estimates, votes and beliefs from the OpenQuestion graph.