Evidence map›Paper›PMID 35711422›Full record

ReviewFrontiers in immunology2022

Potential Benefits of TNF Targeting Therapy in Blau Syndrome, a NOD2-Associated Systemic Autoinflammatory Granulomatosis.

Tomoko Matsuda, Naotomo Kambe, Riko Takimoto-Ito, Yoko Ueki, Satoshi Nakamizo, Megumu K Saito, Syuji Takei, Nobuo Kanazawa

Open access · goldAbstract readReview
In one paragraph

Review in Frontiers in immunology, 2022. The graph could read no effect estimate from its abstract, so it casts no vote on the map. Cited by 17 papers.

0numbers the graph read from it
0cells of the map it votes in
17citing papers in PubMed
2.8field-weighted citation impact, top 8% of its field
1 · What the graph read from it

What it found

Each row is one number read from the abstract, on the scale the paper reported it, with its interval. Left of the dashed line favours the treatment, right favours the comparator. Under each row is the sentence it came from. New to these charts? A ten-minute tutorial.

The abstract states no effect estimate the extractor could read, or names no intervention and outcome on the map, so this paper lights no cell and moves no belief. It is still indexed, cited and linked below.

2 · The registry

The trial behind it

Trials whose registry record cites this paper, or whose number appears in the abstract. A trial that started after this paper was published is citing it as background, not reporting it.

Neither the registry nor the abstract names a trial number. If this is a trial report, that itself is worth knowing.

3 · Its place in the literature

Who cites it

17 citing papers in PubMed, 27 citations in OpenAlex.

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  15. Implications of combinedFrontiers in immunology · 2023
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4 · The record

Corrections and comments

PubMed lists nothing against this paper. Absence here is not a guarantee, only a check that was made.

5 · Who and what money

Authors and funding

8 authors at 4 institutions in 1 country.

Tomoko MatsudaDepartment of Dermatology, Kansai Medical University, Hirakata, Japan.
Naotomo KambeDepartment of Dermatology, Kansai Medical University, Hirakata, Japan.
Riko Takimoto-ItoDepartment of Dermatology, Kyoto University Graduate School of Medicine, Kyoto, Japan.
Yoko UekiDepartment of Dermatology, Kansai Medical University, Hirakata, Japan.
Satoshi NakamizoDepartment of Dermatology, Kyoto University Graduate School of Medicine, Kyoto, Japan.
Megumu K SaitoDepartment of Clinical Application, Center for iPS Cell Research and Application, Kyoto University, Kyoto, Japan.
Syuji TakeiDepartment of Pediatrics, Kagoshima University, Kagoshima, Japan.
Nobuo KanazawaDepartment of Dermatology, Hyogo Medical University, Nishinomiya, Japan.
Kansai Medical University · JPKyoto University · JPHyogo University · JPKagoshima University · JP

Funding

No grant is acknowledged in the PubMed record.

6 · The paper itself

Abstract

Blau syndrome is a systemic autoinflammatory granulomatous disease caused by mutations in the nucleotide-binding oligomerization domain 2 (

Indexed as

SynovitisUveitisArthritisChildCytokinesHereditary Autoinflammatory DiseasesHumansNod2 Signaling Adaptor ProteinSarcoidosisTumor Necrosis Factor InhibitorsCytokinesNOD2 protein, humanNod2 Signaling Adaptor ProteinTumor Necrosis Factor InhibitorsBlau syndromegranulomaIFNγNOD2TNF

Identifiers

PMID35711422
PMCPMC9195515
OpenAlexW4282031125

What OpenQuestion holds

Textmetadata
LicenceCC BY
Read underepoch 390

Registered trials

None linked

Read under generation 80e0d062 · epoch 390. Bibliography from PubMed, PubMed Central and OpenAlex; grants from NIH RePORTER; trial links from ClinicalTrials.gov; estimates, votes and beliefs from the OpenQuestion graph.