Evidence map›Paper›PMID 35625651›Full record

ArticleBiomolecules2022

Preferential Expression of Ca

Yosuke Okamoto, Naing Ye Aung, Masahiro Tanaka, Yuji Takeda, Daichi Takagi, Wataru Igarashi, Kuniaki Ishii, Mitsunori Yamakawa, Kyoichi Ono

Open access · goldAbstract read
In one paragraph

Article in Biomolecules, 2022. The graph could read no effect estimate from its abstract, so it casts no vote on the map. Cited by 4 papers.

0numbers the graph read from it
0cells of the map it votes in
4citing papers in PubMed
1.0field-weighted citation impact, top 24% of its field
1 · What the graph read from it

What it found

Each row is one number read from the abstract, on the scale the paper reported it, with its interval. Left of the dashed line favours the treatment, right favours the comparator. Under each row is the sentence it came from. New to these charts? A ten-minute tutorial.

The abstract states no effect estimate the extractor could read, or names no intervention and outcome on the map, so this paper lights no cell and moves no belief. It is still indexed, cited and linked below.

2 · The registry

The trial behind it

Trials whose registry record cites this paper, or whose number appears in the abstract. A trial that started after this paper was published is citing it as background, not reporting it.

Neither the registry nor the abstract names a trial number. If this is a trial report, that itself is worth knowing.

3 · Its place in the literature

Who cites it

4 citing papers in PubMed, 6 citations in OpenAlex.

  1. Enhanced Late IInternational journal of molecular sciences · 2024
    Article
  2. Function and Role of Histamine HPharmaceuticals (Basel, Switzerland) · 2023
    Review
  3. Review
  4. Article
4 · The record

Corrections and comments

PubMed lists nothing against this paper. Absence here is not a guarantee, only a check that was made.

5 · Who and what money

Authors and funding

9 authors at 2 institutions in 1 country.

Yosuke OkamotoDepartment of Cell Physiology, Akita Graduate School of Medicine, Hondo 010-8543, Japan.ORCID 0000-0001-5119-3840
Naing Ye AungPathological and Image Analysis Center, Cancer Research Center, Faculty of Medicine, Yamagata University, Iida-Nishi 990-9585, Japan.
Masahiro TanakaDepartment of Pharmacology, Faculty of Medicine, Yamagata University, Iida-Nishi 990-9585, Japan.
Yuji TakedaDepartment of Immunology, Faculty of Medicine, Yamagata University, Iida-Nishi 990-9585, Japan.ORCID 0000-0002-0823-1802
Daichi TakagiDepartment of Cardiovascular Surgery, Akita Graduate School of Medicine, Hondo 010-8543, Japan.
Wataru IgarashiDepartment of Cardiovascular Surgery, Akita Graduate School of Medicine, Hondo 010-8543, Japan.
Kuniaki IshiiDepartment of Pharmacology, Faculty of Medicine, Yamagata University, Iida-Nishi 990-9585, Japan.
Mitsunori YamakawaPathological and Image Analysis Center, Cancer Research Center, Faculty of Medicine, Yamagata University, Iida-Nishi 990-9585, Japan.
Kyoichi OnoDepartment of Cell Physiology, Akita Graduate School of Medicine, Hondo 010-8543, Japan.
Yamagata University · JPAkita University · JP

Funding

No grant is acknowledged in the PubMed record.

6 · The paper itself

Abstract

Ectopic excitability in pulmonary veins (PVs) is the major cause of atrial fibrillation. We previously reported that the inositol trisphosphate receptor in rat PV cardiomyocytes cooperates with the Na

Indexed as

Atrial FibrillationPulmonary VeinsAdenylyl CyclasesAnimalsMyocytes, CardiacNorepinephrineRatsSodium-Calcium Exchangeradenylate cyclase 3Adenylyl CyclasesNorepinephrineSodium-Calcium Exchangeradenylyl cyclasearrhythmiaatrial fibrillationpulmonary veinsupraventricular areat-tubule

Identifiers

PMID35625651
PMCPMC9138642
OpenAlexW4280641452

What OpenQuestion holds

Textmetadata
LicenceCC BY
Read underepoch 390

Registered trials

None linked

Read under generation 80e0d062 · epoch 390. Bibliography from PubMed, PubMed Central and OpenAlex; grants from NIH RePORTER; trial links from ClinicalTrials.gov; estimates, votes and beliefs from the OpenQuestion graph.