Evidence map›Paper›PMID 35602830›Full record

ArticleCureus2022

Hereditary Gingival Fibromatosis and Developmental Anomalies: A Case Report.

Rhaina A Afonso, Géssica V Godinho, Cristhiane A Silva, Everton J Silva, Luiz E Volpato

Open access · diamondAbstract readCase Reports
In one paragraph

Article in Cureus, 2022. The graph could read no effect estimate from its abstract, so it casts no vote on the map. Cited by 1 paper.

0numbers the graph read from it
0cells of the map it votes in
1citing papers in PubMed
0.3field-weighted citation impact, top 39% of its field
1 · What the graph read from it

What it found

Each row is one number read from the abstract, on the scale the paper reported it, with its interval. Left of the dashed line favours the treatment, right favours the comparator. Under each row is the sentence it came from. New to these charts? A ten-minute tutorial.

The abstract states no effect estimate the extractor could read, or names no intervention and outcome on the map, so this paper lights no cell and moves no belief. It is still indexed, cited and linked below.

2 · The registry

The trial behind it

Trials whose registry record cites this paper, or whose number appears in the abstract. A trial that started after this paper was published is citing it as background, not reporting it.

Neither the registry nor the abstract names a trial number. If this is a trial report, that itself is worth knowing.

3 · Its place in the literature

Who cites it

1 citing paper in PubMed, 1 citations in OpenAlex.

  1. Surgical Management of Hereditary Gingival Fibromatosis: Case Series.Clinical, cosmetic and investigational dentistry · 2024
    Article
4 · The record

Corrections and comments

PubMed lists nothing against this paper. Absence here is not a guarantee, only a check that was made.

5 · Who and what money

Authors and funding

5 authors at 2 institutions in 1 country.

Rhaina A AfonsoDepartment of Dentistry, Hospital de Câncer de Mato Grosso, Cuiabá, BRA.
Géssica V GodinhoDepartment of Dentistry, Hospital de Câncer de Mato Grosso, Cuiabá, BRA.
Cristhiane A SilvaDepartment of Environment and Health, Universidade de Cuiabá, Cuiabá, BRA.
Everton J SilvaDepartment of Dentistry, Hospital de Câncer de Mato Grosso, Cuiabá, BRA.
Luiz E VolpatoDepartment of Dentistry, Hospital de Câncer de Mato Grosso, Cuiabá, BRA.
Centro Universitário Unirondon · BRUniversidade de Cuiabá · BR

Funding

No grant is acknowledged in the PubMed record.

6 · The paper itself

Abstract

Hereditary gingival fibromatosis is the most common genetic form of gingival fibromatosis that develops as a slow, progressive, benign, localized, or generalized enlargement of the keratinized gingiva. It is a genetically heterogeneous disorder transmitted as an autosomal dominant or autosomal recessive trait or appears sporadically. Here, we report a case of a male patient with generalized gingival hyperplasia with great tissue extension to the palatal region, bilateral mandibular torus, bilateral exostosis in the maxillary posterior region, anterior open bite, and diastema in anterior maxilla and mandible teeth. The mucous membranes were healthy and normal colored, with pale pink gums and firm teeth upon palpation. Computed tomography also revealed images suggestive of supernumerary teeth. The patient reported that his mother and a maternal aunt have the same gingival condition. Considering the gingival characteristic, the patient's family history, and the absence of other possible etiological factors of gingival hyperplasia, the diagnostic hypothesis was hereditary gingival fibromatosis. The surgical removal of the enlarged tissue through gingivectomy with internal bevel, osteoplasty, and removal of supernumerary teeth with a subsequent filling of the surgical sites with platelet and leukocyte-rich fibrin membranes in the same surgical time presented good functional and aesthetic results for the young patient with hereditary gingival fibromatosis. It is a viable possibility for clinical management of similar cases.

Indexed as

autosomal dominant genetic disordergingival fibromatosisgingivectomyplatelet-rich fibrinrare genetic disorder

Identifiers

PMID35602830
PMCPMC9117822
OpenAlexW4224112582

What OpenQuestion holds

Textmetadata
LicenceCC BY
Read underepoch 390

Registered trials

None linked

Read under generation 80e0d062 · epoch 390. Bibliography from PubMed, PubMed Central and OpenAlex; grants from NIH RePORTER; trial links from ClinicalTrials.gov; estimates, votes and beliefs from the OpenQuestion graph.