Evidence map›Paper›PMID 35580065›Full record

ArticleJournal of cellular physiology2022

Modulation of I

Xue An, Jiwon Lee, Ga Hye Kim, Hyun-Ji Kim, Hyun-Jeong Pyo, Ilmin Kwon, Hana Cho

Open access · hybridAbstract read
In one paragraph

Article in Journal of cellular physiology, 2022. The graph could read no effect estimate from its abstract, so it casts no vote on the map. Cited by 8 papers.

0numbers the graph read from it
0cells of the map it votes in
8citing papers in PubMed
0.7field-weighted citation impact, top 34% of its field
1 · What the graph read from it

What it found

Each row is one number read from the abstract, on the scale the paper reported it, with its interval. Left of the dashed line favours the treatment, right favours the comparator. Under each row is the sentence it came from. New to these charts? A ten-minute tutorial.

The abstract states no effect estimate the extractor could read, or names no intervention and outcome on the map, so this paper lights no cell and moves no belief. It is still indexed, cited and linked below.

2 · The registry

The trial behind it

Trials whose registry record cites this paper, or whose number appears in the abstract. A trial that started after this paper was published is citing it as background, not reporting it.

Neither the registry nor the abstract names a trial number. If this is a trial report, that itself is worth knowing.

3 · Its place in the literature

Who cites it

8 citing papers in PubMed, 8 citations in OpenAlex.

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  8. Modulation of IJournal of cellular physiology · 2022
    Article
4 · The record

Corrections and comments

PubMed lists nothing against this paper. Absence here is not a guarantee, only a check that was made.

5 · Who and what money

Authors and funding

7 authors at 1 institution in 1 country.

Xue AnDepartment of Physiology, Sungkyunkwan University School of Medicine, Suwon, Korea.
Jiwon LeeDepartment of Anatomy and Cell Biology, Sungkyunkwan University School of Medicine, Suwon, Korea.
Ga Hye KimDepartment of Anatomy and Cell Biology, Sungkyunkwan University School of Medicine, Suwon, Korea.
Hyun-Ji KimDepartment of Physiology, Sungkyunkwan University School of Medicine, Suwon, Korea.
Hyun-Jeong PyoDepartment of Physiology, Sungkyunkwan University School of Medicine, Suwon, Korea.
Ilmin KwonDepartment of Anatomy and Cell Biology, Sungkyunkwan University School of Medicine, Suwon, Korea.
Hana ChoDepartment of Physiology, Sungkyunkwan University School of Medicine, Suwon, Korea.ORCID 0000-0002-9394-8671
Sungkyunkwan University · KR

Funding

National Research Foundation of Korea NRF-2019R1A2C2003767National Research Foundation of Korea NRF-2020R1A2C2012846
6 · The paper itself

Abstract

Recent studies have shown that protein arginine methyltransferase 1 (PRMT1) is highly expressed in the human heart, and loss of PRMT1 contributes to cardiac remodeling in the heart failure. However, the functional importance of PRMT1 in cardiac ion channels remains uncertain. The slow activating delayed rectifier K

Indexed as

Heart FailureKCNQ1 Potassium ChannelAction PotentialsAnimalsGuinea PigsHEK293 CellsHumansMyocytes, CardiacPhosphatidylinositol PhosphatesPotassium Channels, Voltage-GatedProtein-Arginine N-MethyltransferasesRepressor ProteinsKCNQ1 Potassium ChannelPhosphatidylinositol Phosphatespotassium channel protein I(sk)Potassium Channels, Voltage-GatedPRMT1 protein, humanProtein-Arginine N-MethyltransferasesRepressor Proteinsarrhythmiacardiac myocytesdelayed rectifier potassium channelPIP2PRMT1

Identifiers

PMID35580065
PMCPMC9543859
OpenAlexW4280517746

What OpenQuestion holds

Textmetadata
LicenceCC BY-NC-ND
Read underepoch 390

Registered trials

None linked

Read under generation 80e0d062 · epoch 390. Bibliography from PubMed, PubMed Central and OpenAlex; grants from NIH RePORTER; trial links from ClinicalTrials.gov; estimates, votes and beliefs from the OpenQuestion graph.