Evidence map›Paper›PMID 35469048›Full record

ArticleScientific reports2022

A novel ND1 mitochondrial DNA mutation is maternally inherited in growth hormone transgenesis in amago salmon (Oncorhynchus masou ishikawae).

Tomohiko Sato, Naoko Goto-Inoue, Masaya Kimishima, Jike Toyoharu, Ryuhei Minei, Atsushi Ogura, Hiroyuki Nagoya, Tsukasa Mori

Open access · goldAbstract read
PubMed Full text at PMC Publisher
In one paragraph

Article in Scientific reports, 2022. The graph could read no effect estimate from its abstract, so it casts no vote on the map. Cited by 1 paper.

0numbers the graph read from it
0cells of the map it votes in
1citing papers in PubMed
0.4field-weighted citation impact, top 34% of its field
1 · What the graph read from it

What it found

Each row is one number read from the abstract, on the scale the paper reported it, with its interval. Left of the dashed line favours the treatment, right favours the comparator. Under each row is the sentence it came from. New to these charts? A ten-minute tutorial.

The abstract states no effect estimate the extractor could read, or names no intervention and outcome on the map, so this paper lights no cell and moves no belief. It is still indexed, cited and linked below.

2 · The registry

The trial behind it

Trials whose registry record cites this paper, or whose number appears in the abstract. A trial that started after this paper was published is citing it as background, not reporting it.

Neither the registry nor the abstract names a trial number. If this is a trial report, that itself is worth knowing.

3 · Its place in the literature

Who cites it

1 citing paper in PubMed, 2 citations in OpenAlex.

  1. Roles of Mitochondrial DNA Damage in Kidney Diseases: A New Biomarker.International journal of molecular sciences · 2022
    Review
4 · The record

Corrections and comments

PubMed lists nothing against this paper. Absence here is not a guarantee, only a check that was made.

5 · Who and what money

Authors and funding

8 authors at 3 institutions in 1 country.

Tomohiko SatoDepartment of Marine Science and Resources, Nihon University College of Bioresource Sciences, Kameino 1866, Fujisawa, 252-0880, Japan.
Naoko Goto-InoueDepartment of Marine Science and Resources, Nihon University College of Bioresource Sciences, Kameino 1866, Fujisawa, 252-0880, Japan.
Masaya KimishimaDepartment of Marine Science and Resources, Nihon University College of Bioresource Sciences, Kameino 1866, Fujisawa, 252-0880, Japan.
Jike ToyoharuResearch Institute of Medical Research Support Center Electron Microscope Laboratory, School of Medicine, Nihon University, Tokyo, 173-8610, Japan.
Ryuhei MineiDepartment of Computer Bioscience, Nagahama Institute of Bio-Science and Technology, Nagahama, 526-0829, Japan.
Atsushi OguraDepartment of Computer Bioscience, Nagahama Institute of Bio-Science and Technology, Nagahama, 526-0829, Japan.
Hiroyuki NagoyaNational Research Institute of Aquaculture, Fisheries Research and Education Agency, Minamiise, 516-0193, Japan.
Tsukasa MoriDepartment of Marine Science and Resources, Nihon University College of Bioresource Sciences, Kameino 1866, Fujisawa, 252-0880, Japan. mori.tsukasa@nihon-u.ac.jp.
Nihon University · JPNagahama Institute of Bio-Science and Technology · JPJapan Fisheries Research and Education Agency · JP

Funding

No grant is acknowledged in the PubMed record.

6 · The paper itself

Abstract

Growth hormone (GH) transgenesis can be used to manipulate the growth performance of fish and mammals. In this study, homozygous and hemizygous GH-transgenic amago salmon (Oncorhynchus masou ishikawae) derived from a single female exhibited hypoglycemia. Proteomic and signal network analyses using iTRAQ indicated a decreased NAD

Indexed as

Human Growth HormoneOncorhynchusAnimalsAnimals, Genetically ModifiedDNA, MitochondrialFemaleGene Transfer TechniquesGrowth HormoneMammalsMaternal InheritanceMutationProteomicsReactive Oxygen SpeciesSalmonDNA, MitochondrialGrowth HormoneHuman Growth HormoneReactive Oxygen Species

Identifiers

PMID35469048
PMCPMC9038734
OpenAlexW4224440126

What OpenQuestion holds

Textmetadata
LicenceCC BY
Read underepoch 390

Registered trials

None linked

Read under generation 80e0d062 · epoch 390. Bibliography from PubMed, PubMed Central and OpenAlex; grants from NIH RePORTER; trial links from ClinicalTrials.gov; estimates, votes and beliefs from the OpenQuestion graph.