Evidence map›Paper›PMID 35383094›Full record

ArticleNeurology(R) neuroimmunology & neuroinflammation2022

Transcription Factor c-Maf Promotes Immunoregulation of Programmed Cell Death 1-Expressed CD8

Shusuke Koto, Norio Chihara, Ritsu Akatani, Hiroko Nakano, Atsushi Hara, Kenji Sekiguchi, Riki Matsumoto, Tatsushi Toda

Open access · goldAbstract read
In one paragraph

Article in Neurology(R) neuroimmunology & neuroinflammation, 2022. The graph could read no effect estimate from its abstract, so it casts no vote on the map. Cited by 11 papers.

0numbers the graph read from it
0cells of the map it votes in
11citing papers in PubMed
1.4field-weighted citation impact, top 20% of its field
1 · What the graph read from it

What it found

Each row is one number read from the abstract, on the scale the paper reported it, with its interval. Left of the dashed line favours the treatment, right favours the comparator. Under each row is the sentence it came from. New to these charts? A ten-minute tutorial.

The abstract states no effect estimate the extractor could read, or names no intervention and outcome on the map, so this paper lights no cell and moves no belief. It is still indexed, cited and linked below.

2 · The registry

The trial behind it

Trials whose registry record cites this paper, or whose number appears in the abstract. A trial that started after this paper was published is citing it as background, not reporting it.

Neither the registry nor the abstract names a trial number. If this is a trial report, that itself is worth knowing.

3 · Its place in the literature

Who cites it

11 citing papers in PubMed, 16 citations in OpenAlex.

  1. Article
  2. Review
  3. Article
  4. Article
  5. Review
  6. Review
  7. Review
  8. Article
  9. Article
  10. Programmed Cell Death Protein 1-Positive CD8Neurology(R) neuroimmunology & neuroinflammation · 2022
    Article
  11. Review
4 · The record

Corrections and comments

5 · Who and what money

Authors and funding

8 authors at 1 institution in 1 country.

Shusuke KotoFrom the Division of Neurology (S.K., N.C., R.A., H.N., A.H., K.S., R.M.), Kobe University Graduate School of Medicine; and Department of Neurology (T.T.), Graduate School of Medicine, the University of Tokyo, Japan.
Norio ChiharaFrom the Division of Neurology (S.K., N.C., R.A., H.N., A.H., K.S., R.M.), Kobe University Graduate School of Medicine; and Department of Neurology (T.T.), Graduate School of Medicine, the University of Tokyo, Japan.ORCID 0000-0003-4499-0790
Ritsu AkataniFrom the Division of Neurology (S.K., N.C., R.A., H.N., A.H., K.S., R.M.), Kobe University Graduate School of Medicine; and Department of Neurology (T.T.), Graduate School of Medicine, the University of Tokyo, Japan.
Hiroko NakanoFrom the Division of Neurology (S.K., N.C., R.A., H.N., A.H., K.S., R.M.), Kobe University Graduate School of Medicine; and Department of Neurology (T.T.), Graduate School of Medicine, the University of Tokyo, Japan.
Atsushi HaraFrom the Division of Neurology (S.K., N.C., R.A., H.N., A.H., K.S., R.M.), Kobe University Graduate School of Medicine; and Department of Neurology (T.T.), Graduate School of Medicine, the University of Tokyo, Japan.
Kenji SekiguchiFrom the Division of Neurology (S.K., N.C., R.A., H.N., A.H., K.S., R.M.), Kobe University Graduate School of Medicine; and Department of Neurology (T.T.), Graduate School of Medicine, the University of Tokyo, Japan.ORCID 0000-0003-4100-7316
Riki MatsumotoFrom the Division of Neurology (S.K., N.C., R.A., H.N., A.H., K.S., R.M.), Kobe University Graduate School of Medicine; and Department of Neurology (T.T.), Graduate School of Medicine, the University of Tokyo, Japan.ORCID 0000-0003-3985-9210
Tatsushi TodaFrom the Division of Neurology (S.K., N.C., R.A., H.N., A.H., K.S., R.M.), Kobe University Graduate School of Medicine; and Department of Neurology (T.T.), Graduate School of Medicine, the University of Tokyo, Japan.
Kobe University · JP

Funding

No grant is acknowledged in the PubMed record.

6 · The paper itself

Abstract

BACKGROUND AND

objectivesMultiple sclerosis (MS) is an inflammatory demyelinating disease of the CNS. CD8

methodsWe performed a cohort, case-control study for phenotyping analysis of PD-1

resultsIn the disease remission state, PD-1 DISCUSSION: This study uncovered a favorable role of PD-1

Indexed as

CD8-Positive T-LymphocytesMultiple SclerosisProto-Oncogene Proteins c-mafApoptosisCase-Control StudiesHumansInterleukin-10Programmed Cell Death 1 ReceptorIL10 protein, humanInterleukin-10MAF protein, humanProgrammed Cell Death 1 ReceptorProto-Oncogene Proteins c-maf

Identifiers

PMID35383094
PMCPMC8985076
OpenAlexW4223420460

What OpenQuestion holds

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LicenceCC BY-NC-ND
Read underepoch 390

Registered trials

None linked

Read under generation 80e0d062 · epoch 390. Bibliography from PubMed, PubMed Central and OpenAlex; grants from NIH RePORTER; trial links from ClinicalTrials.gov; estimates, votes and beliefs from the OpenQuestion graph.