Evidence map›Paper›PMID 35341719›Full record

ArticleBiomedical journal2022

Glycolytic metabolism and activation of Na

Hsin-Yi Lin, Rong-Chi Huang

Open access · goldAbstract read
PubMed Full text at PMC Publisher
In one paragraph

Article in Biomedical journal, 2022. The graph could read no effect estimate from its abstract, so it casts no vote on the map. Cited by 3 papers.

0numbers the graph read from it
0cells of the map it votes in
3citing papers in PubMed
0.3field-weighted citation impact, top 53% of its field
1 · What the graph read from it

What it found

Each row is one number read from the abstract, on the scale the paper reported it, with its interval. Left of the dashed line favours the treatment, right favours the comparator. Under each row is the sentence it came from. New to these charts? A ten-minute tutorial.

The abstract states no effect estimate the extractor could read, or names no intervention and outcome on the map, so this paper lights no cell and moves no belief. It is still indexed, cited and linked below.

2 · The registry

The trial behind it

Trials whose registry record cites this paper, or whose number appears in the abstract. A trial that started after this paper was published is citing it as background, not reporting it.

Neither the registry nor the abstract names a trial number. If this is a trial report, that itself is worth knowing.

3 · Its place in the literature

Who cites it

3 citing papers in PubMed, 3 citations in OpenAlex.

  1. Efficient ocular delivery of siRNAInternational journal of pharmaceutics: X · 2023
    Article
  2. Article
  3. About gladiators and a sacred disease.Biomedical journal · 2022
    Article
4 · The record

Corrections and comments

PubMed lists nothing against this paper. Absence here is not a guarantee, only a check that was made.

5 · Who and what money

Authors and funding

2 authors at 1 institution in 1 country.

Hsin-Yi LinDepartment of Physiology and Pharmacology, College of Medicine, Chang Gung University, Taoyuan, Taiwan.
Rong-Chi HuangDepartment of Physiology and Pharmacology, College of Medicine, Chang Gung University, Taoyuan, Taiwan; Healthy Aging Research Center, Chang Gung University, Taoyuan, Taiwan; Neuroscience Research Center, Chang Gung Memorial Hospital at Linkou, Taoyuan, Taiwan. Electronic address: rongchi@mail.cgu.edu.tw.
Chang Gung University · TW

Funding

No grant is acknowledged in the PubMed record.

6 · The paper itself

Abstract

backgroundThe central clock of the suprachiasmatic nucleus (SCN) controls the metabolism of glucose and is sensitive to glucose shortage. However, it is only beginning to be understood how metabolic signals such as glucose availability regulate the SCN physiology. We previously showed that the ATP-sensitive K

methodsDouble-barreled pH-selective microelectrodes were used to determine the pHe in the SCN in hypothalamic slices.

resultsNKA inhibition with K

conclusionGlycolytic metabolism and activation of glycolysis-fueled NKA Na

Indexed as

GlucoseSuprachiasmatic NucleusCyanidesGlycolysisHumansHydrogen-Ion ConcentrationOxidative StressSodiumCyanidesGlucoseSodiumGlycolysisMetabolismNa(+)/K(+)-ATPasepHSuprachiasmatic nucleus

Identifiers

PMID35341719
PMCPMC9133309
OpenAlexW3133356070

What OpenQuestion holds

Textmetadata
LicenceCC BY-NC-ND
Read underepoch 390

Registered trials

None linked

Read under generation 80e0d062 · epoch 390. Bibliography from PubMed, PubMed Central and OpenAlex; grants from NIH RePORTER; trial links from ClinicalTrials.gov; estimates, votes and beliefs from the OpenQuestion graph.