Evidence map›Paper›PMID 35336733›Full record

ArticleBiology2022

Endoglin and Activin Receptor-like Kinase 1 (Alk1) Modify Adrenomedullin Expression in an Organ-Specific Manner in Mice.

Josune García-Sanmartín, Judit Narro-Íñiguez, Alicia Rodríguez-Barbero, Alfredo Martínez

Open access · goldAbstract read
In one paragraph

Article in Biology, 2022. The graph could read no effect estimate from its abstract, so it casts no vote on the map. Cited by 5 papers.

0numbers the graph read from it
0cells of the map it votes in
5citing papers in PubMed
0.5field-weighted citation impact, top 35% of its field
1 · What the graph read from it

What it found

Each row is one number read from the abstract, on the scale the paper reported it, with its interval. Left of the dashed line favours the treatment, right favours the comparator. Under each row is the sentence it came from. New to these charts? A ten-minute tutorial.

The abstract states no effect estimate the extractor could read, or names no intervention and outcome on the map, so this paper lights no cell and moves no belief. It is still indexed, cited and linked below.

2 · The registry

The trial behind it

Trials whose registry record cites this paper, or whose number appears in the abstract. A trial that started after this paper was published is citing it as background, not reporting it.

Neither the registry nor the abstract names a trial number. If this is a trial report, that itself is worth knowing.

3 · Its place in the literature

Who cites it

5 citing papers in PubMed, 5 citations in OpenAlex.

  1. Article
  2. Article
  3. Article
  4. Frontiers in bioengineering and biotechnology · 2023
    Article
  5. Article
4 · The record

Corrections and comments

PubMed lists nothing against this paper. Absence here is not a guarantee, only a check that was made.

5 · Who and what money

Authors and funding

4 authors at 2 institutions in 1 country.

Josune García-SanmartínAngiogenesis Unit, Oncology Area, Center for Biomedical Research of La Rioja (CIBIR), 26006 Logrono, Spain.ORCID 0000-0003-4391-5537
Judit Narro-ÍñiguezAngiogenesis Unit, Oncology Area, Center for Biomedical Research of La Rioja (CIBIR), 26006 Logrono, Spain.ORCID 0000-0001-5336-4930
Alicia Rodríguez-BarberoVascular Endothelium Pathophysiology (ENDOVAS) Unit, Department of Physiology and Pharmacology, University of Salamanca, 37007 Salamanca, Spain.ORCID 0000-0002-6774-7147
Alfredo MartínezAngiogenesis Unit, Oncology Area, Center for Biomedical Research of La Rioja (CIBIR), 26006 Logrono, Spain.ORCID 0000-0003-4882-4044
Center for Biomedical Research of La Rioja · ESUniversidad de Salamanca · ES

Funding

Fundación Rioja Salud 001
6 · The paper itself

Abstract

Hereditary hemorrhagic telangiectasia (HHT) is a rare disease characterized by vascular malformations and profuse bleeding. The disease is caused by mutations in the components of the BMP-9 receptor: endoglin (

Indexed as

adrenomedullinALK1BMP-9endoglinhereditary hemorrhagic telangiectasia (HHT)

Identifiers

PMID35336733
PMCPMC8945164
OpenAlexW4213434922

What OpenQuestion holds

Textmetadata
LicenceCC BY
Read underepoch 390

Registered trials

None linked

Read under generation 80e0d062 · epoch 390. Bibliography from PubMed, PubMed Central and OpenAlex; grants from NIH RePORTER; trial links from ClinicalTrials.gov; estimates, votes and beliefs from the OpenQuestion graph.