ArticleBiological psychiatry global open science2021
Task-general efficiency of evidence accumulation as a computationally-defined neurocognitive trait: Implications for clinical neuroscience.
Article in Biological psychiatry global open science, 2021. The graph could read no effect estimate from its abstract, so it casts no vote on the map. Cited by 33 papers.
What it found
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The abstract states no effect estimate the extractor could read, or names no intervention and outcome on the map, so this paper lights no cell and moves no belief. It is still indexed, cited and linked below.
The trial behind it
Trials whose registry record cites this paper, or whose number appears in the abstract. A trial that started after this paper was published is citing it as background, not reporting it.
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Who cites it
33 citing papers in PubMed, 40 citations in OpenAlex.
- Initial evaluation of a personalized advantage index to determine which individuals may benefit from mindfulness-based cognitive therapy for suicide prevention.Behaviour research and therapy · 2024Trial
- Cognitive markers for efficacy of neurofeedback for attention-deficit hyperactivity disorder - personalized medicine using computational psychiatry in a randomized clinical trial.Journal of clinical and experimental neuropsychology · 2023Trial
- Repetitive negative thinking is associated with conflict-related neural activity that modulates evidence accumulation.Journal of mood and anxiety disorders · 2026Article
- Less systematic decision-making during delay discounting, but not discounting rate, is robustly linked to psychopathology.Journal of psychopathology and clinical science · 2026Article
- Examining Pubertal and Cognitive Differences in Adolescent Depressive Symptoms Using Drift Diffusion Modeling.Research on child and adolescent psychopathology · 2026Article
- Article
- Childhood Material Hardship Linked to Adolescent Neurocognition: A Computational Modeling Approach.Developmental science · 2026Article
- Cognitive Process Models Reveal Meaningful Brain-Behavior Associations in the Adolescent Brain Cognitive Development Study Stop-signal Task.Journal of cognitive neuroscience · 2026Article
- Model-based analysis of stop-signal data reveals robust neural and clinical correlates of evidence accumulation but not inhibition.Neuropsychopharmacology : official publication of the American College of Neuropsychopharmacology · 2026Article
- Joint Cognitive Models Reveal Sources of Robust Individual Differences in Conflict Processing.Computational brain & behavior · 2026Article
- The diffusion model's drift rate parameter primarily reflects efficiency, rather than speed, of evidence accumulation.Psychonomic bulletin & review · 2026Article
- From symptom-based heterogeneity to mechanism-based profiling in youth ADHD: the promise of computational psychiatry.Neuropsychopharmacology : official publication of the American College of Neuropsychopharmacology · 2026Review
- Prefrontal and parieto-occipital neural signatures of evidence accumulation and response to computerised Cognitive Behavioural Therapy in depression.Npj mental health research · 2025Article
- Leveraging bifactor modeling to test prospective direct and indirect effects of adolescent alcohol use and externalizing symptoms on the development of task-general executive functioning.Development and psychopathology · 2025Article
- Efficiency of evidence accumulation as a formal model-based measure of task-general executive functioning in adolescents.Journal of psychopathology and clinical science · 2025Article
- Disentangling sources of variability in decision-making.Nature reviews. Neuroscience · 2025Review
- Neurophysiology of Perceptual Decision-Making and Its Alterations in Attention-Deficit Hyperactivity Disorder.The Journal of neuroscience : the official journal of the Society for Neuroscience · 2025Article
- Acute effects of Δ9-tetrahydrocannabinol on computational measures of neurocognitive processes are related to recent cannabis use among adolescents and young adults.Frontiers in adolescent medicine · 2025Article
- Dissociable Contributions of Goal-Relevant Evidence and Goal-Irrelevant Familiarity to Individual and Developmental Differences in Conflict Recognition.Cognitive science · 2024Article
- Flexible adaptation of task-positive brain networks predicts efficiency of evidence accumulation.Communications biology · 2024Article
Corrections and comments
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Authors and funding
2 authors at 1 institution in 1 country.
Funding
Abstract
Quantifying individual differences in higher-order cognitive functions is a foundational area of cognitive science that also has profound implications for research on psychopathology. For the last two decades, the dominant approach in these fields has been to attempt to fractionate higher-order functions into hypothesized components (e.g., "inhibition", "updating") through a combination of experimental manipulation and factor analysis. However, the putative constructs obtained through this paradigm have recently been met with substantial criticism on both theoretical and empirical grounds. Concurrently, an alternative approach has emerged focusing on parameters of formal computational models of cognition that have been developed in mathematical psychology. These models posit biologically plausible and experimentally validated explanations of the data-generating process for cognitive tasks, allowing them to be used to measure the latent mechanisms that underlie performance. One of the primary insights provided by recent applications of such models is that individual and clinical differences in performance on a wide variety of cognitive tasks, ranging from simple choice tasks to complex executive paradigms, are largely driven by efficiency of evidence accumulation (EEA), a computational mechanism defined by sequential sampling models. This review assembles evidence for the hypothesis that EEA is a central individual difference dimension that explains neurocognitive deficits in multiple clinical disorders and identifies ways in which in this insight can advance clinical neuroscience research. We propose that recognition of EEA as a major driver of neurocognitive differences will allow the field to make clearer inferences about cognitive abnormalities in psychopathology and their links to neurobiology.
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Registered trials
Read under generation 80e0d062 · epoch 390. Bibliography from PubMed, PubMed Central and OpenAlex; grants from NIH RePORTER; trial links from ClinicalTrials.gov; estimates, votes and beliefs from the OpenQuestion graph.