Evidence map›Paper›PMID 35309327›Full record

ReviewFrontiers in immunology2022

Dysregulation of Survivin-Targeting microRNAs in Autoimmune Diseases: New Perspectives for Novel Therapies.

Navid Shomali, Marwah Suliman Maashi, Behzad Baradaran, Amin Daei Sorkhabi, Aila Sarkesh, Hamed Mohammadi, Maryam Hemmatzadeh, Faroogh Marofi, Siamak Sandoghchian Shotorbani, Mostafa Jarahian

RetractedOpen access · goldAbstract readReviewRetracted Publication
In one paragraph

Review in Frontiers in immunology, 2022. The graph could read no effect estimate from its abstract, so it casts no vote on the map. It has been retracted, and should not be counted. Cited by 14 papers.

0numbers the graph read from it
0cells of the map it votes in
14citing papers in PubMed
2.6field-weighted citation impact, top 9% of its field
1 · What the graph read from it

What it found

Each row is one number read from the abstract, on the scale the paper reported it, with its interval. Left of the dashed line favours the treatment, right favours the comparator. Under each row is the sentence it came from. New to these charts? A ten-minute tutorial.

The abstract states no effect estimate the extractor could read, or names no intervention and outcome on the map, so this paper lights no cell and moves no belief. It is still indexed, cited and linked below.

2 · The registry

The trial behind it

Trials whose registry record cites this paper, or whose number appears in the abstract. A trial that started after this paper was published is citing it as background, not reporting it.

Neither the registry nor the abstract names a trial number. If this is a trial report, that itself is worth knowing.

3 · Its place in the literature

Who cites it

14 citing papers in PubMed, 27 citations in OpenAlex.

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  5. CXC chemokine receptor 4 (CXCR4) blockade in cancer treatment.Journal of cancer research and clinical oncology · 2023
    Review
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4 · The record

Corrections and comments

5 · Who and what money

Authors and funding

10 authors at 4 institutions in 3 countries.

Navid ShomaliImmunology Research Center, Tabriz University of Medical Sciences, Tabriz, Iran.
Marwah Suliman MaashiMedical Laboratory Technology Department, Faculty of Applied Medical Sciences, King Abdulaziz University, Jeddah, Saudi Arabia.
Behzad BaradaranImmunology Research Center, Tabriz University of Medical Sciences, Tabriz, Iran.
Amin Daei SorkhabiStudent Research Committee, Tabriz University of Medical Sciences, Tabriz, Iran.
Aila SarkeshStudent Research Committee, Tabriz University of Medical Sciences, Tabriz, Iran.
Hamed MohammadiNon-Communicable Diseases Research Center, Alborz University of Medical Sciences, Karaj, Iran.
Maryam HemmatzadehDepartment of Immunology, Faculty of Medicine, Tabriz University of Medical Sciences, Tabriz, Iran.
Faroogh MarofiImmunology Research Center, Tabriz University of Medical Sciences, Tabriz, Iran.
Siamak Sandoghchian ShotorbaniImmunology Research Center, Tabriz University of Medical Sciences, Tabriz, Iran.
Mostafa JarahianGerman Cancer Research Center, Toxicology and Chemotherapy Unit (G401), Heidelberg, Germany.
Tabriz University of Medical Sciences · IRGerman Cancer Research Center · DEJahrom University of Medical Sciences · IRKing Abdulaziz University · SA

Funding

No grant is acknowledged in the PubMed record.

6 · The paper itself

Abstract

It has been well established that the etiopathogenesis of diverse autoimmune diseases is rooted in the autoreactive immune cells' excessively proliferative state and impaired apoptotic machinery. Survivin is an anti-apoptotic and mitotic factor that has sparked a considerable research interest in this field. Survivin overexpression has been shown to contribute significantly to the development of autoimmune diseases

Indexed as

Autoimmune DiseasesMicroRNAsAutoimmunityHumansSurvivinMicroRNAsSurvivinand multiple sclerosis (MS)autoimmune diseaseinflammatory bowel disease (IBD)microRNApsoriasisrheumatoid arthritis (RA)survivinsystemic lupus erythematosus (SLE)

Identifiers

PMID35309327
PMCPMC8927965
OpenAlexW4214834623

What OpenQuestion holds

Textmetadata
LicenceCC BY
Read underepoch 390

Registered trials

None linked

Read under generation 80e0d062 · epoch 390. Bibliography from PubMed, PubMed Central and OpenAlex; grants from NIH RePORTER; trial links from ClinicalTrials.gov; estimates, votes and beliefs from the OpenQuestion graph.