Evidence map›Paper›PMID 35293603›Full record

ArticleMolecular medicine reports2022

Potential impact of

Mohammad Muzaffar Mir, Rashid Mir, Mushabab Ayed Abdullah Alghamdi, Javed Iqbal Wani, Imadeldin Elfaki, Zia Ul Sabah, Muhanad Alhujaily, Mohammed Jeelani, Vijaya Marakala, Muffarah Hamid Alharthi and 1 more

Open access · hybridAbstract read
In one paragraph

Article in Molecular medicine reports, 2022. The graph could read no effect estimate from its abstract, so it casts no vote on the map. Cited by 14 papers.

0numbers the graph read from it
0cells of the map it votes in
14citing papers in PubMed
5.0field-weighted citation impact, top 4% of its field
1 · What the graph read from it

What it found

Each row is one number read from the abstract, on the scale the paper reported it, with its interval. Left of the dashed line favours the treatment, right favours the comparator. Under each row is the sentence it came from. New to these charts? A ten-minute tutorial.

The abstract states no effect estimate the extractor could read, or names no intervention and outcome on the map, so this paper lights no cell and moves no belief. It is still indexed, cited and linked below.

2 · The registry

The trial behind it

Trials whose registry record cites this paper, or whose number appears in the abstract. A trial that started after this paper was published is citing it as background, not reporting it.

Neither the registry nor the abstract names a trial number. If this is a trial report, that itself is worth knowing.

3 · Its place in the literature

Who cites it

14 citing papers in PubMed, 23 citations in OpenAlex.

  1. Article
  2. Article
  3. Review
  4. Probiotics EnhanceInternational journal of molecular sciences · 2024
    Article
  5. Article
  6. Article
  7. A critical review on therapeutic approaches of CRISPR-Cas9 in diabetes mellitus.Naunyn-Schmiedeberg's archives of pharmacology · 2023
    Review
  8. Article
  9. Article
  10. Article
  11. Article
  12. Article
  13. Review
  14. Article
4 · The record

Corrections and comments

PubMed lists nothing against this paper. Absence here is not a guarantee, only a check that was made.

5 · Who and what money

Authors and funding

11 authors at 3 institutions in 1 country.

Mohammad Muzaffar MirDepartment of Basic Medical Sciences, College of Medicine, University of Bisha, Bisha 61922, Kingdom of Saudi Arabia.
Rashid MirPrince Fahd Bin Sultan Research Chair, Department of Medical Laboratory Technology (MLT), Faculty of Applied Medical Sciences, University of Tabuk, Tabuk 71491, Kingdom of Saudi Arabia.
Mushabab Ayed Abdullah AlghamdiDepartment of Internal Medicine, College of Medicine, University of Bisha, Bisha 61922, Kingdom of Saudi Arabia.
Javed Iqbal WaniDepartment of Internal Medicine College of Medicine, King Khalid University, Abha 61421, Kingdom of Saudi Arabia.
Imadeldin ElfakiDepartment of Biochemistry, Faculty of Science, University of Tabuk, Tabuk 71491, Kingdom of Saudi Arabia.
Zia Ul SabahDepartment of Internal Medicine College of Medicine, King Khalid University, Abha 61421, Kingdom of Saudi Arabia.
Muhanad AlhujailyDepartment of Clinical Laboratory Sciences, College of Applied Medical Sciences, University of Bisha, Bisha 61922, Kingdom of Saudi Arabia.
Mohammed JeelaniDepartment of Basic Medical Sciences, College of Medicine, University of Bisha, Bisha 61922, Kingdom of Saudi Arabia.
Vijaya MarakalaDepartment of Basic Medical Sciences, College of Medicine, University of Bisha, Bisha 61922, Kingdom of Saudi Arabia.
Muffarah Hamid AlharthiDepartment of Family Medicine, College of Medicine, University of Bisha, Bisha 61922, Kingdom of Saudi Arabia.
Abdullah M Al-ShahraniDepartment of Family Medicine, College of Medicine, University of Bisha, Bisha 61922, Kingdom of Saudi Arabia.
University of Bisha · SAKing Khalid University · SAUniversity of Tabuk · SA

Funding

No grant is acknowledged in the PubMed record.

6 · The paper itself

Abstract

Type 2 diabetes mellitus (T2DM) is a metabolic disorder characterized by persistent hyperglycemia and is associated with serious complications. The risk factors for T2DM include both genetic and lifestyle factors. Genome‑wide association studies have indicated the association of genetic variations with many diseases, including T2DM. Glucokinase (GCK) plays a key role in the regulation of insulin release in the pancreas and catalyzes the first step in glycolysis in the liver. Genetic alterations in the

Indexed as

Diabetes Mellitus, Type 2MicroRNAsCase-Control StudiesGenetic Predisposition to DiseaseGenome-Wide Association StudyGerminal Center KinasesGlucokinaseHumansPolymorphism, Single NucleotideSaudi ArabiaGerminal Center KinasesGlucokinaseMAP4K2 protein, humanMicroRNAsMIRN423 microRNA, humanglucokinase rs1799884MIR‑196A-2 (rs11614913)MIR‑423 (rs6505162)Saudi Arabiasingle nucleotide polymorphismtetra‑primer amplification refractory mutation system‑polymerase chain reactiontype 2 diabetes mellitus

Identifiers

PMID35293603
PMCPMC8941532
OpenAlexW4220925167

What OpenQuestion holds

Textmetadata
LicenceCC BY-NC-ND
Read underepoch 390

Registered trials

None linked

Read under generation 80e0d062 · epoch 390. Bibliography from PubMed, PubMed Central and OpenAlex; grants from NIH RePORTER; trial links from ClinicalTrials.gov; estimates, votes and beliefs from the OpenQuestion graph.