Evidence map›Paper›PMID 35289116›Full record

ArticleThe journal of pathology. Clinical research2022

CD24 is a surrogate for 'immune-cold' phenotype in aggressive large B-cell lymphoma.

Morihiro Higashi, Shuji Momose, Natsuko Takayanagi, Yuka Tanaka, Tomoe Anan, Takahisa Yamashita, Jun Kikuchi, Michihide Tokuhira, Masahiro Kizaki, Jun-Ichi Tamaru

Open access · goldAbstract read
In one paragraph

Article in The journal of pathology. Clinical research, 2022. The graph could read no effect estimate from its abstract, so it casts no vote on the map. Cited by 11 papers.

0numbers the graph read from it
0cells of the map it votes in
11citing papers in PubMed
2.1field-weighted citation impact, top 13% of its field
1 · What the graph read from it

What it found

Each row is one number read from the abstract, on the scale the paper reported it, with its interval. Left of the dashed line favours the treatment, right favours the comparator. Under each row is the sentence it came from. New to these charts? A ten-minute tutorial.

The abstract states no effect estimate the extractor could read, or names no intervention and outcome on the map, so this paper lights no cell and moves no belief. It is still indexed, cited and linked below.

2 · The registry

The trial behind it

Trials whose registry record cites this paper, or whose number appears in the abstract. A trial that started after this paper was published is citing it as background, not reporting it.

Neither the registry nor the abstract names a trial number. If this is a trial report, that itself is worth knowing.

3 · Its place in the literature

Who cites it

11 citing papers in PubMed, 14 citations in OpenAlex.

  1. Review
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  7. Emerging Immune Checkpoint Molecules on Cancer Cells: CD24 and CD200.International journal of molecular sciences · 2023
    Review
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4 · The record

Corrections and comments

PubMed lists nothing against this paper. Absence here is not a guarantee, only a check that was made.

5 · Who and what money

Authors and funding

10 authors at 1 institution in 1 country.

Morihiro HigashiDepartment of Pathology, Saitama Medical Center, Saitama Medical University, Kawagoe, Japan.ORCID 0000-0002-0093-8977
Shuji MomoseDepartment of Pathology, Saitama Medical Center, Saitama Medical University, Kawagoe, Japan.
Natsuko TakayanagiDepartment of Pathology, Saitama Medical Center, Saitama Medical University, Kawagoe, Japan.
Yuka TanakaDepartment of Hematology, Saitama Medical Center, Saitama Medical University, Kawagoe, Japan.ORCID 0000-0001-9603-9839
Tomoe AnanDepartment of Hematology, Saitama Medical Center, Saitama Medical University, Kawagoe, Japan.
Takahisa YamashitaDepartment of Pathology, Saitama Medical Center, Saitama Medical University, Kawagoe, Japan.
Jun KikuchiDepartment of Pathology, Saitama Medical Center, Saitama Medical University, Kawagoe, Japan.
Michihide TokuhiraHematology, Saitama Medical Center, Japan Community Health Care Organization, Kawagoe, Japan.
Masahiro KizakiDepartment of Hematology, Saitama Medical Center, Saitama Medical University, Kawagoe, Japan.
Jun-Ichi TamaruDepartment of Pathology, Saitama Medical Center, Saitama Medical University, Kawagoe, Japan.
Social Insurance Saitama Chuo Hospital · JP

Funding

No grant is acknowledged in the PubMed record.

6 · The paper itself

Abstract

The tumor microenvironment (TME) is a critical regulator of the development of malignant lymphoma. Therapeutics targeting the TME, especially immune checkpoint molecules, are changing the treatment strategy for lymphoma. However, the overall response to these therapeutics for diffuse large B-cell lymphoma (DLBCL) is modest and new targets of immunotherapy are needed. To find critical immune checkpoint molecules for DLBCL, we explored the prognostic impact of immune checkpoint molecules and their ligands using publicly available datasets of gene expression profiles. In silico analysis of three independent datasets (GSE117556, GSE10846, and GSE181063) revealed that DLBCL expressing CD24 had a poor prognosis and had a high frequency of MYC aberrations. Moreover, gene set enrichment analysis showed that the 'MYC-targets-hallmark' (false discovery rate [FDR] = 0.024) and 'inflammatory-response-hallmark' (FDR = 0.001) were enriched in CD24-high and CD24-low DLBCL, respectively. In addition, the expression of cell-specific markers of various immune cells was higher in CD24-low DLBCL than in CD24-high DLBCL. CIBERSORT analysis of the datasets showed fewer macrophages in CD24-high DLBCL than in CD24-low DLBCL. Additionally, immunohistochemical analysis of 335 cases of DLBCL showed that few TME cells were found in CD24-high DLBCL, although statistical differences were not observed. These data indicate that CD24 expression suppresses immune cell components of the TME in DLBCL, suggesting that CD24 may be a target for cancer immunotherapy in aggressive large B-cell lymphoma.

Indexed as

CD24 AntigenImmune Checkpoint ProteinsLymphoma, Large B-Cell, DiffuseHumansPhenotypePrognosisTumor MicroenvironmentCD24 AntigenCD24 protein, humanImmune Checkpoint ProteinsDLBCLlymphomatumor microenvironment

Identifiers

PMID35289116
PMCPMC9161324
OpenAlexW4220919417

What OpenQuestion holds

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LicenceCC BY
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Registered trials

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Read under generation 80e0d062 · epoch 390. Bibliography from PubMed, PubMed Central and OpenAlex; grants from NIH RePORTER; trial links from ClinicalTrials.gov; estimates, votes and beliefs from the OpenQuestion graph.