Article in Physiological reports, 2022. The graph could read no effect estimate from its abstract, so it casts no vote on the map. Cited by 2 papers.
0numbers the graph read from it
0cells of the map it votes in
2citing papers in PubMed
0.3field-weighted citation impact, top 42% of its field
1 · What the graph read from it
What it found
Each row is one number read from the abstract, on the scale the paper reported it, with its interval. Left of the dashed line favours the treatment, right favours the comparator. Under each row is the sentence it came from. New to these charts? A ten-minute tutorial.
The abstract states no effect estimate the extractor could read, or names no intervention and outcome on the map, so this paper lights no cell and moves no belief. It is still indexed, cited and linked below.
2 · The registry
The trial behind it
Trials whose registry record cites this paper, or whose number appears in the abstract. A trial that started after this paper was published is citing it as background, not reporting it.
Neither the registry nor the abstract names a trial number. If this is a trial report, that itself is worth knowing.
3 · Its place in the literature
Who cites it
2 citing papers in PubMed, 2 citations in OpenAlex.
PubMed lists nothing against this paper. Absence here is not a guarantee, only a check that was made.
5 · Who and what money
Authors and funding
3 authors at 1 institution in 1 country.
Elizabeth Martinez-HernandezDepartment of Physiology & Biophysics, Rush University Medical Center, Chicago, Illinois, USA.ORCID 0000-0002-5004-2239
Lothar A BlatterDepartment of Physiology & Biophysics, Rush University Medical Center, Chicago, Illinois, USA.ORCID 0000-0003-3409-8678
Giedrius KanaporisDepartment of Physiology & Biophysics, Rush University Medical Center, Chicago, Illinois, USA.ORCID 0000-0003-3593-1182
Rush University Medical Center · US
Funding
Limiting Pathological Calcium Induced Calcium Release in HeartR01HL057832 · NHLBI · RUSH UNIVERSITY MEDICAL CENTER · PI FILL, MICHAEL, GILLESPIE, DIRK · 1997 to 2024
$9.0M
Atrial Excitation-Contraction Coupling, Calcium Signaling and Electro-Mechanical AlternansR01HL164453 · NHLBI · RUSH UNIVERSITY MEDICAL CENTER · PI BANACH, KATHRIN, KANAPORIS, GIEDRIUS · 2022 to 2025
$2.8M
IP3 receptor, NOX2 and calcium signaling domains in atrial physiology and pathophysiologyR01HL155762 · NHLBI · RUSH UNIVERSITY MEDICAL CENTER · PI BANACH, KATHRIN · 2022 to 2025
$2.7M
Pathophysiological Regulation of Atrial Alternans and Atrial FibrillationR01HL132871 · NHLBI · RUSH UNIVERSITY MEDICAL CENTER · PI BANACH, KATHRIN, BLATTER, LOTHAR A · 2017 to 2020
$1.6M
Pathophysiological Regulation of Atrial Myocyte Excitation-Contraction Coupling and Calcium SignalingR01HL134781 · NHLBI · RUSH UNIVERSITY MEDICAL CENTER · PI BLATTER, LOTHAR A · 2017 to 2020
Adaptation of the myocardium to varying workloads critically depends on the recovery from inactivation (RFI) of L-type Ca
Indexed as
Calcium-Calmodulin-Dependent Protein Kinase Type 2Myocytes, CardiacAction PotentialsAnimalsCalciumMembrane PotentialsMyocardiumRabbitsSarcoplasmic ReticulumSodiumSodium-Calcium ExchangerCalciumCalcium-Calmodulin-Dependent Protein Kinase Type 2SodiumSodium-Calcium ExchangeratriaCa2+/calmodulin-dependent kinase IICa2+ channelCa2+ cyclingrecovery from inactivation
Read under generation 80e0d062 · epoch 390. Bibliography from PubMed, PubMed Central and OpenAlex; grants from NIH RePORTER; trial links from ClinicalTrials.gov; estimates, votes and beliefs from the OpenQuestion graph.