Evidence map›Paper›PMID 35229066›Full record

ArticleJBMR plus2022

The Posterior Part Influences the Anterior Part of the Mouse Cranial Base Development.

Honghao Zhang, Ke'ale W Louie, Anshul K Kulkarni, Karen Zapien-Guerra, Jingwen Yang, Yuji Mishina

Open access · goldAbstract read
In one paragraph

Article in JBMR plus, 2022. The graph could read no effect estimate from its abstract, so it casts no vote on the map. Cited by 5 papers.

0numbers the graph read from it
0cells of the map it votes in
5citing papers in PubMed
0.4field-weighted citation impact, top 40% of its field
1 · What the graph read from it

What it found

Each row is one number read from the abstract, on the scale the paper reported it, with its interval. Left of the dashed line favours the treatment, right favours the comparator. Under each row is the sentence it came from. New to these charts? A ten-minute tutorial.

The abstract states no effect estimate the extractor could read, or names no intervention and outcome on the map, so this paper lights no cell and moves no belief. It is still indexed, cited and linked below.

2 · The registry

The trial behind it

Trials whose registry record cites this paper, or whose number appears in the abstract. A trial that started after this paper was published is citing it as background, not reporting it.

Neither the registry nor the abstract names a trial number. If this is a trial report, that itself is worth knowing.

3 · Its place in the literature

Who cites it

5 citing papers in PubMed, 6 citations in OpenAlex.

  1. Neural crest-specific disruption of Evc2 provides an animal model to study the temporomandibular joint development and homeostasis in response to jaw loading.Journal of bone and mineral research : the official journal of the American Society for Bone and Mineral Research · 2026
    Article
  2. Article
  3. Review
  4. Cranial Base Synchondrosis: Chondrocytes at the Hub.International journal of molecular sciences · 2022
    Review
  5. Article
4 · The record

Corrections and comments

PubMed lists nothing against this paper. Absence here is not a guarantee, only a check that was made.

5 · Who and what money

Authors and funding

6 authors at 1 institution in 1 country.

Honghao ZhangDepartment of Biologic and Materials Sciences & Prosthodontics, School of Dentistry University of Michigan Ann Arbor MI USA.ORCID https://orcid.org/0000-0002-2338-6992
Ke'ale W LouieDepartment of Biologic and Materials Sciences & Prosthodontics, School of Dentistry University of Michigan Ann Arbor MI USA.
Anshul K KulkarniDepartment of Biologic and Materials Sciences & Prosthodontics, School of Dentistry University of Michigan Ann Arbor MI USA.
Karen Zapien-GuerraDepartment of Biologic and Materials Sciences & Prosthodontics, School of Dentistry University of Michigan Ann Arbor MI USA.
Jingwen YangDepartment of Biologic and Materials Sciences & Prosthodontics, School of Dentistry University of Michigan Ann Arbor MI USA.
Yuji MishinaDepartment of Biologic and Materials Sciences & Prosthodontics, School of Dentistry University of Michigan Ann Arbor MI USA.ORCID https://orcid.org/0000-0002-6268-4204
University of Michigan · US

Funding

No grant is acknowledged in the PubMed record.

6 · The paper itself

Abstract

The cranial base is a critical structure in the head, which is composed of endoskeletal and dermal skeletal. The braincase floor, part of the cranial base, is a midline structure of the head. Because it is a midline structure connecting the posterior skull with the facial region, braincase floor is critical for the orientation of the facial structure. Shortened braincase floor leads to mid-facial hypoplasia and malocclusions. During embryonic development, elongation of the braincase floor occurs through endochondral ossification in the parachordal cartilage, hypophyseal cartilage, and trabecular cartilage, which leads to formation of basioccipital (BO), basisphenoid (BS), and presphenoid (PS) bones, respectively. Currently, little is known about whether maturation of parachordal cartilage, hypophyseal cartilage, and trabecular cartilage occurs in a simultaneous or sequential manner and if the formation of one impacts the others. Our previous studies demonstrated that loss of function of ciliary protein

Indexed as

BONE MODELING AND REMODELINGCELL/TISSUE SIGNALING—PARACRINE PATHWAYSCHONDROCYTE AND CARTILAGE BIOLOGYDENTAL BIOLOGYDEVELOPMENTAL MODELINGGROWTH PLATEHEDGEHOGS

Identifiers

PMID35229066
PMCPMC8861986
OpenAlexW4200418187

What OpenQuestion holds

Textmetadata
LicenceCC BY
Read underepoch 390

Registered trials

None linked

Read under generation 80e0d062 · epoch 390. Bibliography from PubMed, PubMed Central and OpenAlex; grants from NIH RePORTER; trial links from ClinicalTrials.gov; estimates, votes and beliefs from the OpenQuestion graph.