ArticleiScience2022
Longitudinal 16S rRNA gut microbiota data of infant triplets show partial susceptibility to host genetics.
Article in iScience, 2022. The graph could read no effect estimate from its abstract, so it casts no vote on the map. Cited by 5 papers.
What it found
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The abstract states no effect estimate the extractor could read, or names no intervention and outcome on the map, so this paper lights no cell and moves no belief. It is still indexed, cited and linked below.
The trial behind it
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Who cites it
5 citing papers in PubMed.
- Perinatal Hypoxia and Immune System Activation in Schizophrenia Pathogenesis: Critical Considerations During COVID-19 Pandemic.Physiological research · 2024Review
- Early-life differences in the gut microbiota composition and functionality of infants at elevated likelihood of developing autism spectrum disorder.Translational psychiatry · 2023Article
- Early-life microbiota-immune homeostasis.Frontiers in immunology · 2023Review
- Proof of principle study replicating microbial clusters in connection to birth mode and diet in the early life intestine.PloS one · 2022Article
- How longitudinal data can contribute to our understanding of host genetic effects on the gut microbiome.Gut microbesArticle
Corrections and comments
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Authors and funding
7 authors.
Funding
Abstract
The question of whether host genetics plays a role in the development of the infant gut microbiota does not, as yet, have a clear answer. In order to throw additional light on this question, we have analyzed 16S rRNA amplicon sequences from 99 valid fecal samples of five sets of dichorionic triplet babies born by C-section from 1 to 36 months of age. Beta diversity analysis showed that monozygotic twins were more similar to each other than their dizygotic siblings. Monozygotic twins also tended to share more amplicon sequence variants between them. Heritability analysis showed that the genera
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Registered trials
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