Evidence map›Paper›PMID 35186174›Full record

ArticleCanadian respiratory journal2022

Association of

Jong-Uk Lee, Ji-Yeon Jeong, Min Kyung Kim, Sun A Min, Jong-Sook Park, Choon-Sik Park

Open access · goldAbstract read
In one paragraph

Article in Canadian respiratory journal, 2022. The graph could read no effect estimate from its abstract, so it casts no vote on the map. Cited by 1 paper.

0numbers the graph read from it
0cells of the map it votes in
1citing papers in PubMed
0.4field-weighted citation impact, top 43% of its field
1 · What the graph read from it

What it found

Each row is one number read from the abstract, on the scale the paper reported it, with its interval. Left of the dashed line favours the treatment, right favours the comparator. Under each row is the sentence it came from. New to these charts? A ten-minute tutorial.

The abstract states no effect estimate the extractor could read, or names no intervention and outcome on the map, so this paper lights no cell and moves no belief. It is still indexed, cited and linked below.

2 · The registry

The trial behind it

Trials whose registry record cites this paper, or whose number appears in the abstract. A trial that started after this paper was published is citing it as background, not reporting it.

Neither the registry nor the abstract names a trial number. If this is a trial report, that itself is worth knowing.

3 · Its place in the literature

Who cites it

1 citing paper in PubMed, 5 citations in OpenAlex.

  1. The Leucocyte Telomere Length,Current issues in molecular biology · 2022
    Article
4 · The record

Corrections and comments

PubMed lists nothing against this paper. Absence here is not a guarantee, only a check that was made.

5 · Who and what money

Authors and funding

6 authors at 1 institution in 1 country.

Jong-Uk LeeDivision of Allergy and Respiratory Medicine, Department of Internal Medicine, Soonchunhyang University Bucheon Hospital, Bucheon, Republic of Korea.ORCID 0000-0002-0232-5381
Ji-Yeon JeongDivision of Allergy and Respiratory Medicine, Department of Internal Medicine, Soonchunhyang University Bucheon Hospital, Bucheon, Republic of Korea.ORCID 0000-0002-8251-2039
Min Kyung KimDepartment of Interdisciplinary Program in Biomedical Science Major, Soonchuhyang University, Asan, Republic of Korea.ORCID 0000-0003-2996-0198
Sun A MinDepartment of Interdisciplinary Program in Biomedical Science Major, Soonchuhyang University, Asan, Republic of Korea.
Jong-Sook ParkDivision of Allergy and Respiratory Medicine, Department of Internal Medicine, Soonchunhyang University Bucheon Hospital, Bucheon, Republic of Korea.ORCID 0000-0003-4128-9085
Choon-Sik ParkDivision of Allergy and Respiratory Medicine, Department of Internal Medicine, Soonchunhyang University Bucheon Hospital, Bucheon, Republic of Korea.ORCID 0000-0003-2977-0255
Soonchunhyang University · KR

Funding

No grant is acknowledged in the PubMed record.

6 · The paper itself

Abstract

backgroundToluene diisocyanate (TDI) causes occupational asthma by generating oxidative stress, leading to tissue injury and inflammation. Glutathione transferases (GSTs) are detoxifying enzymes that eliminate oxidative stress. We examined whether the genotypes of the

methodsThe study population consisted of 26 asthmatics with a positive response to the TDI challenge (TDI-PA) and 27 asthmatics with negative responses (TDI-NA).

resultsThe

conclusionsThe

Indexed as

Asthma, OccupationalToluene 2,4-DiisocyanateCase-Control StudiesGenetic Predisposition to DiseaseGenotypeGlutathione TransferaseHumansPolymorphism, GeneticRisk Factorsglutathione S-transferase T1Glutathione TransferaseToluene 2,4-Diisocyanate

Identifiers

PMID35186174
PMCPMC8856815
OpenAlexW4211055189

What OpenQuestion holds

Textmetadata
LicenceCC BY
Read underepoch 390

Registered trials

None linked

Read under generation 80e0d062 · epoch 390. Bibliography from PubMed, PubMed Central and OpenAlex; grants from NIH RePORTER; trial links from ClinicalTrials.gov; estimates, votes and beliefs from the OpenQuestion graph.