Evidence map›Paper›PMID 35167804›Full record

ArticleCurrent biology : CB2022

ECM degradation in the Drosophila abdominal epidermis initiates tissue growth that ceases with rapid cell-cycle exit.

John Robert Davis, Anna P Ainslie, John J Williamson, Ana Ferreira, Alejandro Torres-Sánchez, Andreas Hoppe, Federica Mangione, Matthew B Smith, Enrique Martin-Blanco, Guillaume Salbreux and 1 more

Open access · hybridAbstract read
In one paragraph

Article in Current biology : CB, 2022. The graph could read no effect estimate from its abstract, so it casts no vote on the map. Cited by 9 papers.

0numbers the graph read from it
0cells of the map it votes in
9citing papers in PubMed
2.6field-weighted citation impact, top 11% of its field
1 · What the graph read from it

What it found

Each row is one number read from the abstract, on the scale the paper reported it, with its interval. Left of the dashed line favours the treatment, right favours the comparator. Under each row is the sentence it came from. New to these charts? A ten-minute tutorial.

The abstract states no effect estimate the extractor could read, or names no intervention and outcome on the map, so this paper lights no cell and moves no belief. It is still indexed, cited and linked below.

2 · The registry

The trial behind it

Trials whose registry record cites this paper, or whose number appears in the abstract. A trial that started after this paper was published is citing it as background, not reporting it.

Neither the registry nor the abstract names a trial number. If this is a trial report, that itself is worth knowing.

3 · Its place in the literature

Who cites it

9 citing papers in PubMed, 24 citations in OpenAlex.

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4 · The record

Corrections and comments

5 · Who and what money

Authors and funding

11 authors at 4 institutions in 3 countries.

John Robert DavisApoptosis and Proliferation Control Laboratory, The Francis Crick Institute, 1 Midland Road, London NW1 1AT, UK.
Anna P AinslieApoptosis and Proliferation Control Laboratory, The Francis Crick Institute, 1 Midland Road, London NW1 1AT, UK.
John J WilliamsonTheoretical Physics of Biology Laboratory, The Francis Crick Institute, 1 Midland Road, London NW1 1AT, UK.
Ana FerreiraApoptosis and Proliferation Control Laboratory, The Francis Crick Institute, 1 Midland Road, London NW1 1AT, UK.
Alejandro Torres-SánchezTheoretical Physics of Biology Laboratory, The Francis Crick Institute, 1 Midland Road, London NW1 1AT, UK.
Andreas HoppeFaculty of Science, Engineering and Computing, Kingston University, Kingston-upon-Thames KT1 2EE, UK.
Federica MangioneApoptosis and Proliferation Control Laboratory, The Francis Crick Institute, 1 Midland Road, London NW1 1AT, UK.
Matthew B SmithTheoretical Physics of Biology Laboratory, The Francis Crick Institute, 1 Midland Road, London NW1 1AT, UK.
Enrique Martin-BlancoInstituto de Biología Molecular de Barcelona, Consejo Superior de Investigaciones Científicas, Parc Científic de Barcelona, C/Baldiri Reixac, 4-8, Torre R, 3era Planta, 08028 Barcelona, Spain.
Guillaume SalbreuxTheoretical Physics of Biology Laboratory, The Francis Crick Institute, 1 Midland Road, London NW1 1AT, UK; Department of Genetics and Evolution, University of Geneva, Quai Ernest Ansermet 30, 1211 Geneva, Switzerland. Electronic address: guillaume.salbreux@unige.ch.
Nicolas TaponApoptosis and Proliferation Control Laboratory, The Francis Crick Institute, 1 Midland Road, London NW1 1AT, UK. Electronic address: nic.tapon@crick.ac.uk.
The Francis Crick Institute · GBConsejo Superior de Investigaciones Científicas · ESKingston University · GBUniversity of Geneva · CH

Funding

Cancer Research UK 17064Cancer Research UK FC001175Cancer Research UK FC001317Medical Research Council FC001175Medical Research Council FC001317Wellcome Trust 107885/Z/15/ZWellcome Trust 201358/Z/16/ZWellcome Trust FC001175Wellcome Trust FC001317
6 · The paper itself

Abstract

During development, multicellular organisms undergo stereotypical patterns of tissue growth in space and time. How developmental growth is orchestrated remains unclear, largely due to the difficulty of observing and quantitating this process in a living organism. Drosophila histoblast nests are small clusters of progenitor epithelial cells that undergo extensive growth to give rise to the adult abdominal epidermis and are amenable to live imaging. Our quantitative analysis of histoblast proliferation and tissue mechanics reveals that tissue growth is driven by cell divisions initiated through basal extracellular matrix degradation by matrix metalloproteases secreted by the neighboring larval epidermal cells. Laser ablations and computational simulations show that tissue mechanical tension does not decrease as the histoblasts fill the abdominal epidermal surface. During tissue growth, the histoblasts display oscillatory cell division rates until growth termination occurs through the rapid emergence of G0/G1 arrested cells, rather than a gradual increase in cell-cycle time as observed in other systems such as the Drosophila wing and mouse postnatal epidermis. Different developing tissues can therefore achieve their final size using distinct growth termination strategies. Thus, adult abdominal epidermal development is characterized by changes in the tissue microenvironment and a rapid exit from the cell cycle.

Indexed as

DrosophilaEpidermal CellsAnimalsCell CycleCell DivisionEpidermisMicecell divisiondevelopmentDrosophilaextracellular matrixgrowth controlmechanicsquantitative biologysimulations

Identifiers

PMID35167804
PMCPMC8967408
OpenAlexW4213211382

What OpenQuestion holds

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LicenceCC BY
Read underepoch 390

Registered trials

None linked

Read under generation 80e0d062 · epoch 390. Bibliography from PubMed, PubMed Central and OpenAlex; grants from NIH RePORTER; trial links from ClinicalTrials.gov; estimates, votes and beliefs from the OpenQuestion graph.