Evidence map›Paper›PMID 35163450›Full record

ReviewInternational journal of molecular sciences2022

Roles of α-Synuclein and Disease-Associated Factors in

Mari Suzuki, Kazunori Sango, Yoshitaka Nagai

Open access · goldAbstract readReview
In one paragraph

Review in International journal of molecular sciences, 2022. The graph could read no effect estimate from its abstract, so it casts no vote on the map. Cited by 17 papers.

0numbers the graph read from it
0cells of the map it votes in
17citing papers in PubMed
2.1field-weighted citation impact, top 12% of its field
1 · What the graph read from it

What it found

Each row is one number read from the abstract, on the scale the paper reported it, with its interval. Left of the dashed line favours the treatment, right favours the comparator. Under each row is the sentence it came from. New to these charts? A ten-minute tutorial.

The abstract states no effect estimate the extractor could read, or names no intervention and outcome on the map, so this paper lights no cell and moves no belief. It is still indexed, cited and linked below.

2 · The registry

The trial behind it

Trials whose registry record cites this paper, or whose number appears in the abstract. A trial that started after this paper was published is citing it as background, not reporting it.

Neither the registry nor the abstract names a trial number. If this is a trial report, that itself is worth knowing.

3 · Its place in the literature

Who cites it

17 citing papers in PubMed, 19 citations in OpenAlex.

  1. Review
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  3. Utilizing amicroPublication biology · 2026
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  11. Validating and Optimizing amicroPublication biology · 2025
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  17. Role ofInternational journal of molecular sciences · 2022
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4 · The record

Corrections and comments

PubMed lists nothing against this paper. Absence here is not a guarantee, only a check that was made.

5 · Who and what money

Authors and funding

3 authors at 2 institutions in 1 country.

Mari SuzukiDiabetic Neuropathy Project, Department of Diseases and Infection, Tokyo Metropolitan Institute of Medical Science, Setagaya, Tokyo 156-8506, Japan.ORCID 0000-0001-7495-1675
Kazunori SangoDiabetic Neuropathy Project, Department of Diseases and Infection, Tokyo Metropolitan Institute of Medical Science, Setagaya, Tokyo 156-8506, Japan.ORCID 0000-0002-9750-9596
Yoshitaka NagaiDepartment of Neurotherapeutics, Graduate School of Medicine, Osaka University, Suita 565-0871, Japan.
Tokyo Metropolitan Institute of Medical Science · JPKindai University · JP

Funding

Japan Agency for Medical Research and Development JP16ek0109018Japan Agency for Medical Research and Development JP19ek0109222Ministry of Education, Culture, Sports, Science and Technology 17H05699Ministry of Education, Culture, Sports, Science and Technology 17K19658Ministry of Education, Culture, Sports, Science and Technology 19K07987National Center of Neurology and Psychiatry 30-3National Center of Neurology and Psychiatry 30-9National Center of Neurology and Psychiatry 3-9
6 · The paper itself

Abstract

α-Synuclein (αSyn) plays a major role in the pathogenesis of Parkinson's disease (PD), which is the second most common neurodegenerative disease after Alzheimer's disease. The accumulation of αSyn is a pathological hallmark of PD, and mutations in the

Indexed as

Mutationalpha-SynucleinAnimalsAnimals, Genetically ModifiedDisease Models, AnimalDrosophila melanogasterHumansParkinson Diseasealpha-SynucleinSNCA protein, humanDrosophilaParkinson’s diseaseα-Synuclein

Identifiers

PMID35163450
PMCPMC8835920
OpenAlexW4210388741

What OpenQuestion holds

Textmetadata
LicenceCC BY
Read underepoch 390

Registered trials

None linked

Read under generation 80e0d062 · epoch 390. Bibliography from PubMed, PubMed Central and OpenAlex; grants from NIH RePORTER; trial links from ClinicalTrials.gov; estimates, votes and beliefs from the OpenQuestion graph.