Evidence map›Paper›PMID 35163382›Full record

ArticleInternational journal of molecular sciences2022

A Central Role for TRPM4 in Ca

Tamás Csípő, Ágnes Czikora, Gábor Á Fülöp, Hajnalka Gulyás, Ibolya Rutkai, Enikő Pásztorné Tóth, Róbert Pórszász, Andrea Szalai, Kata Bölcskei, Zsuzsanna Helyes and 4 more

Open access · goldAbstract read
PubMed Full text at PMC Publisher
In one paragraph

Article in International journal of molecular sciences, 2022. The graph could read no effect estimate from its abstract, so it casts no vote on the map. Cited by 9 papers.

0numbers the graph read from it
0cells of the map it votes in
9citing papers in PubMed
1.5field-weighted citation impact, top 21% of its field
1 · What the graph read from it

What it found

Each row is one number read from the abstract, on the scale the paper reported it, with its interval. Left of the dashed line favours the treatment, right favours the comparator. Under each row is the sentence it came from. New to these charts? A ten-minute tutorial.

The abstract states no effect estimate the extractor could read, or names no intervention and outcome on the map, so this paper lights no cell and moves no belief. It is still indexed, cited and linked below.

2 · The registry

The trial behind it

Trials whose registry record cites this paper, or whose number appears in the abstract. A trial that started after this paper was published is citing it as background, not reporting it.

Neither the registry nor the abstract names a trial number. If this is a trial report, that itself is worth knowing.

3 · Its place in the literature

Who cites it

9 citing papers in PubMed, 13 citations in OpenAlex.

  1. Article
  2. Review
  3. Article
  4. Article
  5. Article
  6. Review
  7. Article
  8. Ion Channels in the Development and Remodeling of the Aortic Valve.International journal of molecular sciences · 2023
    Review
  9. Article
4 · The record

Corrections and comments

PubMed lists nothing against this paper. Absence here is not a guarantee, only a check that was made.

5 · Who and what money

Authors and funding

14 authors at 3 institutions in 2 countries.

Tamás CsípőDivision of Clinical Physiology, Department of Cardiology, Faculty of Medicine, University of Debrecen, 4032 Debrecen, Hungary.
Ágnes CzikoraDivision of Clinical Physiology, Department of Cardiology, Faculty of Medicine, University of Debrecen, 4032 Debrecen, Hungary.
Gábor Á FülöpDivision of Clinical Physiology, Department of Cardiology, Faculty of Medicine, University of Debrecen, 4032 Debrecen, Hungary.
Hajnalka GulyásDivision of Clinical Physiology, Department of Cardiology, Faculty of Medicine, University of Debrecen, 4032 Debrecen, Hungary.
Ibolya RutkaiDivision of Clinical Physiology, Department of Cardiology, Faculty of Medicine, University of Debrecen, 4032 Debrecen, Hungary.
Enikő Pásztorné TóthDivision of Clinical Physiology, Department of Cardiology, Faculty of Medicine, University of Debrecen, 4032 Debrecen, Hungary.
Róbert PórszászDepartment of Pharmacology and Pharmacotherapy, Faculty of Medicine, University of Debrecen, 4032 Debrecen, Hungary.
Andrea SzalaiDepartment of Pharmacology and Pharmacotherapy, Faculty of Medicine, University of Debrecen, 4032 Debrecen, Hungary.
Kata BölcskeiDepartment of Pharmacology and Pharmacotherapy, Medical School, University of Pécs, 7624 Pécs, Hungary.
Zsuzsanna HelyesDepartment of Pharmacology and Pharmacotherapy, Medical School, University of Pécs, 7624 Pécs, Hungary.
Erika PintérDepartment of Pharmacology and Pharmacotherapy, Medical School, University of Pécs, 7624 Pécs, Hungary.ORCID 0000-0001-9898-632X
Zoltán PappDivision of Clinical Physiology, Department of Cardiology, Faculty of Medicine, University of Debrecen, 4032 Debrecen, Hungary.ORCID 0000-0002-4675-1542
Zoltán UngváriVascular Cognitive Impairment and Neurodegeneration Program, Oklahoma Center for Geroscience, Department of Biochemistry and Molecular Biology, University of Oklahoma Health Sciences Center, Oklahoma City, OK 73104, USA.
Attila TóthDivision of Clinical Physiology, Department of Cardiology, Faculty of Medicine, University of Debrecen, 4032 Debrecen, Hungary.ORCID 0000-0001-6503-3653
University of Debrecen · HUUniversity of Pecs · HUSemmelweis University · HU

Funding

Hungarian Scientific Research Fund FK 128809Hungarian Scientific Research Fund K 116940Hungarian Scientific Research Fund K 132623National Research, Development and Innovation Fund of Hungary TKP2020-NKA-04
6 · The paper itself

Abstract

Transient receptor potential melastatin-4 (TRPM4) is activated by an increase in intracellular Ca

Indexed as

Calcium SignalingVasoconstrictionAdministration, IntravenousAnimalsArteriesBlood PressureCalcimycinCalciumEndothelium, VascularHeart RateIonophoresMaleMuscle DevelopmentMuscle, SkeletalMuscle, Smooth, VascularMyocytes, Smooth Muscle9-phenanthrolCalcimycinCalciumIonophoresNorepinephrinePhenanthrenesPotassium ChlorideTRPM4 protein, ratTRPM Cation Channelsblood pressure regulationCa2+ signalingtransient receptor potentialtransient receptor potential melastatin-4vascular smooth muscle

Identifiers

PMID35163382
PMCPMC8836177
OpenAlexW4210412569

What OpenQuestion holds

Textmetadata
LicenceCC BY
Read underepoch 390

Registered trials

None linked

Read under generation 80e0d062 · epoch 390. Bibliography from PubMed, PubMed Central and OpenAlex; grants from NIH RePORTER; trial links from ClinicalTrials.gov; estimates, votes and beliefs from the OpenQuestion graph.