ArticleJournal of clinical laboratory analysis2022
Serum amyloid a, C-reactive protein, and procalcitonin levels in children with Mycoplasma pneumoniae infection.
Article in Journal of clinical laboratory analysis, 2022. The graph could read no effect estimate from its abstract, so it casts no vote on the map. Cited by 27 papers.
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Who cites it
27 citing papers in PubMed, 35 citations in OpenAlex.
- S100A8/A9 as a novel predictor for severe Mycoplasma pneumoniae pneumonia in children: A prospective observational study.Clinics (Sao Paulo, Brazil) · 2026Article
- Human biomarker navigator.iMeta · 2026Review
- Clinical Characteristics and Early Risk Factors for SevereInfection and drug resistance · 2026Article
- Immune dysregulation inFrontiers in immunology · 2026Review
- Development of a nomogram for predicting refractoryTranslational pediatrics · 2025Article
- Correlation between 25-hydroxyvitamin D, N-terminal pro-brain natriuretic peptide, inflammatory factors and myocardial injury in children with mycoplasma pneumoniae pneumonia.BMC pediatrics · 2025Article
- Diagnostic efficacy of Mycoplasma pneumoniae antibody and procalcitonin in children with Mycoplasma pneumonia.BMC pulmonary medicine · 2025Article
- The diagnostic value plasma procalcitonin (PCT) and serum amyloid A protein (SAA) in children with mycoplasma pneumonia.Journal of medical biochemistry · 2025Article
- Bronchoalveolar lavage cytokine profile and clinical features as risk predictors of plastic bronchitis in children with Mycoplasma pneumoniae pneumonia.Italian journal of pediatrics · 2025Article
- Comparative analysis of the clinical characteristics of severe Mycoplasma pneumoniae pneumonia and severe bacterial pneumonia in children.BMC pediatrics · 2025Article
- Navigating the outbreak: a comprehensive analysis of pediatricMicrobiology spectrum · 2025Article
- C-reactive protein-induced injury inMicrobiology spectrum · 2025Article
- Biomarkers associated with the diagnosis and prognosis ofFrontiers in cellular and infection microbiology · 2025Review
- Differential Diagnosis Value of Neutrophil Gelatinase Associated Lipocalin as a Noninvasive Biomarker in Perianal Fistulizing Crohn's Disease.Journal of inflammation research · 2025Article
- Clinical Diagnostic Prediction Model For Mycoplasma pneumoniae Pneumonia in Children.Infection and drug resistance · 2025Article
- The predictive values of C-reactive protein-neutrophil to lymphocyte ratio for the risk of refractoryEpidemiology and infection · 2024Article
- Clinical characteristics and risk factors of pulmonary embolism with Mycoplasma pneumoniae pneumonia in children.Scientific reports · 2024Article
- A comparative study of general and severe mycoplasma pneumoniae pneumonia in children.BMC infectious diseases · 2024Article
- Procalcitonin and Presepsin as Markers of Infectious Respiratory Diseases in Children: A Scoping Review of the Literature.Children (Basel, Switzerland) · 2024Article
- Clinical characteristics and risk factors forFrontiers in pediatrics · 2024Article
Corrections and comments
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Authors and funding
9 authors at 2 institutions in 2 countries.
Funding
Abstract
backgroundMycoplasma pneumoniae (MP) is a common pathogen of community-acquired pneumonia in children. In the present study, serum amyloid A (SAA), C-reactive protein (CRP), and procalcitonin (PCT) levels in children with MP infection were analyzed and the differential diagnoses of MP evaluated.
methodsThe study included 152 children with MP infection hospitalized in Tai'an Central Hospital in Shandong Province and 50 healthy children as controls. SAA, CRP, and PCT, as well as serum immunoglobulins and T lymphocyte subsets were analyzed during the acute and convalescent phases. Among the MP-infected children, 30 cases were selected to monitor the SAA, immunoglobulins, and T lymphocyte subset levels for a week.
resultsThe SAA, CRP, PCT, IgA, and IgM levels were significantly higher in the MP-infected group than in the control group (F
conclusionSAA, CRP, and PCT were specific markers for diagnosing early MP infection in children. These findings are important in the differential diagnosis of MP infection and clinical guidance for MP treatment.
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