Evidence map›Paper›PMID 35129134›Full record

ArticleJournal of genetics2022

Examination of

Lan Enli, Yuto Moronuki, Takahisa Yamada, Hiroyuki Kose

Abstract read
PubMed
In one paragraph

Article in Journal of genetics, 2022. The graph could read no effect estimate from its abstract, so it casts no vote on the map. Not yet cited in PubMed.

0numbers the graph read from it
0cells of the map it votes in
0citing papers in PubMed
0.1field-weighted citation impact, top 65% of its field
1 · What the graph read from it

What it found

Each row is one number read from the abstract, on the scale the paper reported it, with its interval. Left of the dashed line favours the treatment, right favours the comparator. Under each row is the sentence it came from. New to these charts? A ten-minute tutorial.

The abstract states no effect estimate the extractor could read, or names no intervention and outcome on the map, so this paper lights no cell and moves no belief. It is still indexed, cited and linked below.

2 · The registry

The trial behind it

Trials whose registry record cites this paper, or whose number appears in the abstract. A trial that started after this paper was published is citing it as background, not reporting it.

Neither the registry nor the abstract names a trial number. If this is a trial report, that itself is worth knowing.

3 · Its place in the literature

Who cites it

0 citing papers in PubMed, 1 citations in OpenAlex.

No citing paper in PubMed yet.

4 · The record

Corrections and comments

PubMed lists nothing against this paper. Absence here is not a guarantee, only a check that was made.

5 · Who and what money

Authors and funding

4 authors at 2 institutions in 1 country.

Lan EnliDepartment of Natural Sciences, International Christian University, Mitaka, Tokyo 181-8585, Japan. kose@icu.ac.jp.
Yuto Moronuki
Takahisa Yamada
Hiroyuki Kose
International Christian University · JPNiigata University · JP

Funding

No grant is acknowledged in the PubMed record.

6 · The paper itself

Abstract

Type 2 diabetes mellitus (T2D) is a metabolic disorder caused by a complex interplay between genetic and environmental factors. While remarkable progress have been made in our understanding of the genetic components that lead to disease expression, the 'diabetes genes' identified to date are inadequate for assessing disease risk, which suggests that many other genes remain to be discovered. Here, we used Drosophila to examine the protein-coding genes annotated in the hyperglycaemic locus of Otsuka Long-Evans Tokushima Fatty (OLETF) rats, a well-characterized model of T2D. We identified

Indexed as

Diabetes Mellitus, Type 2AnimalsBlood GlucoseDisease Models, AnimalDrosophila melanogasterDrosophila ProteinsFemaleLiverNeuropeptidesNuclear ProteinsRatsRats, Inbred OLETFTranscription FactorsBlood GlucoseDrosophila ProteinsGAL4 protein, DrosophilaILP2 protein, Drosophilalilli protein, DrosophilaNeuropeptidesNuclear ProteinsTranscription Factors

Identifiers

PMID35129134
OpenAlexW4210752587

What OpenQuestion holds

Textmetadata
Read underepoch 390

Registered trials

None linked

Read under generation 80e0d062 · epoch 390. Bibliography from PubMed, PubMed Central and OpenAlex; grants from NIH RePORTER; trial links from ClinicalTrials.gov; estimates, votes and beliefs from the OpenQuestion graph.