Evidence map›Paper›PMID 35056016›Full record

ArticlePathogens (Basel, Switzerland)2022

The E3 Ubiquitin Ligase ATL9 Affects Expression of Defense Related Genes, Cell Death and Callose Deposition in Response to Fungal Infection.

Tingwei Guo, Feng Kong, Carter Burton, Steven Scaglione, Blake Beagles, Justin Ray, Katrina M Ramonell

Open access · goldAbstract read
PubMed Full text at PMC Publisher
In one paragraph

Article in Pathogens (Basel, Switzerland), 2022. The graph could read no effect estimate from its abstract, so it casts no vote on the map. Cited by 2 papers.

0numbers the graph read from it
0cells of the map it votes in
2citing papers in PubMed
2.9field-weighted citation impact, top 10% of its field
1 · What the graph read from it

What it found

Each row is one number read from the abstract, on the scale the paper reported it, with its interval. Left of the dashed line favours the treatment, right favours the comparator. Under each row is the sentence it came from. New to these charts? A ten-minute tutorial.

The abstract states no effect estimate the extractor could read, or names no intervention and outcome on the map, so this paper lights no cell and moves no belief. It is still indexed, cited and linked below.

2 · The registry

The trial behind it

Trials whose registry record cites this paper, or whose number appears in the abstract. A trial that started after this paper was published is citing it as background, not reporting it.

Neither the registry nor the abstract names a trial number. If this is a trial report, that itself is worth knowing.

3 · Its place in the literature

Who cites it

2 citing papers in PubMed, 8 citations in OpenAlex.

  1. Article
  2. International journal of molecular sciences · 2022
    Article
4 · The record

Corrections and comments

PubMed lists nothing against this paper. Absence here is not a guarantee, only a check that was made.

5 · Who and what money

Authors and funding

7 authors at 2 institutions in 1 country.

Tingwei GuoDepartment of Biological Sciences, The University of Alabama, Tuscaloosa, AL 35401, USA.
Feng KongDepartment of Biological Sciences, The University of Alabama, Tuscaloosa, AL 35401, USA.ORCID 0000-0002-2700-8307
Carter BurtonDepartment of Biological Sciences, The University of Alabama, Tuscaloosa, AL 35401, USA.
Steven ScaglioneDepartment of Biological Sciences, The University of Alabama, Tuscaloosa, AL 35401, USA.ORCID 0000-0002-1947-5837
Blake BeaglesDepartment of Biological Sciences, The University of Alabama, Tuscaloosa, AL 35401, USA.
Justin RayDepartment of Biological Sciences, The University of Alabama, Tuscaloosa, AL 35401, USA.
Katrina M RamonellDepartment of Biological Sciences, The University of Alabama, Tuscaloosa, AL 35401, USA.
University of Alabama · USUniversity of Southern California · US

Funding

No grant is acknowledged in the PubMed record.

6 · The paper itself

Abstract

Plants use diverse strategies to defend themselves from biotic stresses in nature, which include the activation of defense gene expression and a variety of signal transduction pathways. Previous studies have shown that protein ubiquitination plays a critical role in plant defense responses, however the details of its function remain unclear. Our previous work has shown that increasing expression levels of

Indexed as

Arabidopsis thalianaE3 ubiquitin ligasefungal infectionplant defenseplant-pathogen interactionsignal transduction pathwaysubiquitination

Identifiers

PMID35056016
PMCPMC8778023
OpenAlexW4206811938

What OpenQuestion holds

Textmetadata
LicenceCC BY
Read underepoch 390

Registered trials

None linked

Read under generation 80e0d062 · epoch 390. Bibliography from PubMed, PubMed Central and OpenAlex; grants from NIH RePORTER; trial links from ClinicalTrials.gov; estimates, votes and beliefs from the OpenQuestion graph.