Evidence map›Paper›PMID 35052559›Full record

ReviewAntioxidants (Basel, Switzerland)2021

Role of Nitric Oxide and Protein S-Nitrosylation in Ischemia-Reperfusion Injury.

Hyang-Mi Lee, Ji Woong Choi, Min Sik Choi

Open access · goldAbstract readReview
In one paragraph

Review in Antioxidants (Basel, Switzerland), 2021. The graph could read no effect estimate from its abstract, so it casts no vote on the map. Cited by 19 papers, 1 of them a synthesis that pooled it.

0numbers the graph read from it
0cells of the map it votes in
19citing papers in PubMed, 1 pooled it
1.8field-weighted citation impact, top 15% of its field
1 · What the graph read from it

What it found

Each row is one number read from the abstract, on the scale the paper reported it, with its interval. Left of the dashed line favours the treatment, right favours the comparator. Under each row is the sentence it came from. New to these charts? A ten-minute tutorial.

The abstract states no effect estimate the extractor could read, or names no intervention and outcome on the map, so this paper lights no cell and moves no belief. It is still indexed, cited and linked below.

2 · The registry

The trial behind it

Trials whose registry record cites this paper, or whose number appears in the abstract. A trial that started after this paper was published is citing it as background, not reporting it.

Neither the registry nor the abstract names a trial number. If this is a trial report, that itself is worth knowing.

3 · Its place in the literature

Who cites it

19 citing papers in PubMed, 1 synthesis or guideline pooled it, 28 citations in OpenAlex.

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  17. Antioxidant Activity ofAntioxidants (Basel, Switzerland) · 2022
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4 · The record

Corrections and comments

PubMed lists nothing against this paper. Absence here is not a guarantee, only a check that was made.

5 · Who and what money

Authors and funding

3 authors at 2 institutions in 1 country.

Hyang-Mi LeeCollege of Pharmacy, Dongduk Women's University, Seoul 02748, Korea.
Ji Woong ChoiCollege of Pharmacy, Gachon University, Incheon 21936, Korea.
Min Sik ChoiLaboratory of Pharmacology, College of Pharmacy, Dongduk Women's University, Seoul 02748, Korea.
Dongduk Women's University · KRGachon University · KR

Funding

National Research Foundation of Korea NRF-2020M3A9E4104384National Research Foundation of Korea NRF-2020R1A6A1A03043708National Research Foundation of Korea NRF-2021R1A2C1005520
6 · The paper itself

Abstract

Ischemia-reperfusion injury (IRI) is a process in which damage is induced in hypoxic tissue when oxygen supply is resumed after ischemia. During IRI, restoration of reduced nitric oxide (NO) levels may alleviate reperfusion injury in ischemic organs. The protective mechanism of NO is due to anti-inflammatory effects, antioxidant effects, and the regulation of cell signaling pathways. On the other hand, it is generally known that S-nitrosylation (SNO) mediates the detrimental or protective effect of NO depending on the action of the nitrosylated target protein, and this is also applied in the IRI process. In this review, the effect of each change of NO and SNO during the IRI process was investigated.

Indexed as

ischemia-reperfusion injurynitric oxideprotein S-nitrosylation

Identifiers

PMID35052559
PMCPMC8772765
OpenAlexW4200592209

What OpenQuestion holds

Textmetadata
LicenceCC BY
Read underepoch 390

Registered trials

None linked

Read under generation 80e0d062 · epoch 390. Bibliography from PubMed, PubMed Central and OpenAlex; grants from NIH RePORTER; trial links from ClinicalTrials.gov; estimates, votes and beliefs from the OpenQuestion graph.