ArticleBeyoglu eye journal2021
Assessment of the Effect of Attention-Deficit Hyperactivity Disorder on Choroidal Thickness Using Spectral Domain Optical Coherence Tomography.
Article in Beyoglu eye journal, 2021. The graph could read no effect estimate from its abstract, so it casts no vote on the map. Cited by 2 papers.
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Who cites it
2 citing papers in PubMed.
- Retinal fundus imaging as biomarker for ADHD using machine learning for screening and visual attention stratification.NPJ digital medicine · 2025Article
- An examination of the ocular effects of methylphenidate used in children and adolescents diagnosed with attention-deficit hyperactivity disorder.International ophthalmology · 2025Article
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Authors and funding
4 authors.
Funding
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Abstract
objectivesThis study was a comparison of the thickness of the choroid in pediatric patients with attention-deficit hyperactivity disorder (ADHD) and healthy pediatric controls.
methodsThis study was comparative, cross-sectional, and observational in design. The healthy controls were age- and sex-matched with the members of the ADHD group and had no history of psychosis. Choroidal thickness was determined using spectral-domain optical coherence tomography.
resultsA total of 138 patients were enrolled with a male:female ratio of 54 (69.2%): 24 (30.8%) in the ADHD group and 41 (68.3%): 19 (31.7%) in the control group (p=0.910). The ADHD patients had a mean age of 9.4±1.9 years (range: 6-12 years) and the controls had a mean age of 9.9±2.2 years (range: 6-12 years) (p=0.213). The ADHD group (n=78 eyes tested) had a significantly higher mean choroidal thickness at 1.5 mm (temporal-to-fovea, TTF) measurement than the controls (n=60 eyes tested) (281.12±46.63 μm vs. 264.40±48.61 μm, p=0.042). There were no significant differences in any of the other choroidal thickness measurements (p>0.05).
conclusionThe choroidal thickness measurement (TTF) at 1.5 mm was significantly greater in the ADHD patients. These findings suggest that choroidal thickness alterations may have a potential role in the underlying etiology of ADHD.
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