Evidence map›Paper›PMID 34924789›Full record

ArticleNoro psikiyatri arsivi2021

Proteasome Modulator 9 Gene rs14259 Polymorphism in Patients with Diabetic Polyneuropathy.

Celal Şalçini, Gülin Sunter, Fatih Özen, Yağmur Özer, Belkıs Atasever Arslan

Open access · greenAbstract read
In one paragraph

Article in Noro psikiyatri arsivi, 2021. The graph could read no effect estimate from its abstract, so it casts no vote on the map. Cited by 1 paper.

0numbers the graph read from it
0cells of the map it votes in
1citing papers in PubMed
0.2field-weighted citation impact, top 52% of its field
1 · What the graph read from it

What it found

Each row is one number read from the abstract, on the scale the paper reported it, with its interval. Left of the dashed line favours the treatment, right favours the comparator. Under each row is the sentence it came from. New to these charts? A ten-minute tutorial.

The abstract states no effect estimate the extractor could read, or names no intervention and outcome on the map, so this paper lights no cell and moves no belief. It is still indexed, cited and linked below.

2 · The registry

The trial behind it

Trials whose registry record cites this paper, or whose number appears in the abstract. A trial that started after this paper was published is citing it as background, not reporting it.

Neither the registry nor the abstract names a trial number. If this is a trial report, that itself is worth knowing.

3 · Its place in the literature

Who cites it

1 citing paper in PubMed, 2 citations in OpenAlex.

  1. AiScience · 2023
    Article
4 · The record

Corrections and comments

PubMed lists nothing against this paper. Absence here is not a guarantee, only a check that was made.

5 · Who and what money

Authors and funding

5 authors at 2 institutions in 1 country.

Celal ŞalçiniDepartment of Neurology, NP İstanbul Brain Hospital, Üsküdar University, İstanbul, Turkey.
Gülin SunterDepartment of Neurology, Marmara Üniversitesi School of Medicine, İstanbul, Turkey.
Fatih ÖzenDepartment of Bioengineering, Üsküdar University, İstanbul, Turkey.
Yağmur ÖzerNeuroscience Program, Health Sciences Institute, Üsküdar University, İstanbul, Turkey.
Belkıs Atasever ArslanDepartment of Molecular Biology and Genetics, Üsküdar University, İstanbul, Turkey.
Üsküdar University · TRMarmara University · TR

Funding

No grant is acknowledged in the PubMed record.

6 · The paper itself

Abstract

introductionDiabetic polyneuropathy (DPN) is a major chronic neurological complication of diabetes mellitus (DM) and typically presents as diabetic sensory polyneuropathy (DSPN). Whereas some patients with similar risk factors develop polyneuropathy, others don't, which suggests that genetics plays an important role in the progression of disease. The

methodsThe study included 31 DM patients with DSPN and 29 healthy controls. All participants underwent electrophysiological investigation. In addition, DNA was isolated from peripheral blood samples for the genotyping of

resultsMean age in the DSPN and control groups was 58.03±9.59 years and 57.62±12.32 years, respectively. There were significant differences between the DSPN and controls groups in the frequencies of the genotype for AA (n=9 and n=12, respectively), AG (n=10 and n=15, respectively), and GG (n=12 and n=2, respectively). According to the distribution of

conclusionThe present findings show that the GG genotype and G allele of

Indexed as

diabetes mellitusdiabetic polyneuropathyPSMD9rs14259

Identifiers

PMID34924789
PMCPMC8665284
OpenAlexW2998882999

What OpenQuestion holds

Textmetadata
LicenceCC BY-NC-SA
Read underepoch 390

Registered trials

None linked

Read under generation 80e0d062 · epoch 390. Bibliography from PubMed, PubMed Central and OpenAlex; grants from NIH RePORTER; trial links from ClinicalTrials.gov; estimates, votes and beliefs from the OpenQuestion graph.