Evidence map›Paper›PMID 34911964›Full record

ArticleNature communications2021

Directly recruited GATA6 + peritoneal cavity macrophages contribute to the repair of intestinal serosal injury.

Masaki Honda, Masashi Kadohisa, Daiki Yoshii, Yoshihiro Komohara, Taizo Hibi

Open access · goldAbstract read
In one paragraph

Article in Nature communications, 2021. The graph could read no effect estimate from its abstract, so it casts no vote on the map. Cited by 36 papers.

0numbers the graph read from it
0cells of the map it votes in
36citing papers in PubMed
2.9field-weighted citation impact, top 8% of its field
1 · What the graph read from it

What it found

Each row is one number read from the abstract, on the scale the paper reported it, with its interval. Left of the dashed line favours the treatment, right favours the comparator. Under each row is the sentence it came from. New to these charts? A ten-minute tutorial.

The abstract states no effect estimate the extractor could read, or names no intervention and outcome on the map, so this paper lights no cell and moves no belief. It is still indexed, cited and linked below.

2 · The registry

The trial behind it

Trials whose registry record cites this paper, or whose number appears in the abstract. A trial that started after this paper was published is citing it as background, not reporting it.

Neither the registry nor the abstract names a trial number. If this is a trial report, that itself is worth knowing.

3 · Its place in the literature

Who cites it

36 citing papers in PubMed, 61 citations in OpenAlex.

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  5. Peritoneal cavity-derived GATA6Journal of advanced research · 2026
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4 · The record

Corrections and comments

PubMed lists nothing against this paper. Absence here is not a guarantee, only a check that was made.

5 · Who and what money

Authors and funding

5 authors at 2 institutions in 1 country.

Masaki Honda *Department of Transplantation and Pediatric Surgery, Kumamoto University, 1-1-1 Honjo, Chuo-ku, Kumamoto, 860-8556, Japan. honda.masaki@kuh.kumamoto-u.ac.jp.ORCID http://orcid.org/0000-0001-5526-2591
Masashi Kadohisa *Department of Transplantation and Pediatric Surgery, Kumamoto University, 1-1-1 Honjo, Chuo-ku, Kumamoto, 860-8556, Japan.
Daiki YoshiiDepartment of Transplantation and Pediatric Surgery, Kumamoto University, 1-1-1 Honjo, Chuo-ku, Kumamoto, 860-8556, Japan.
Yoshihiro KomoharaDepartment of Cell Pathology, Kumamoto University, 1-1-1 Honjo, Chuo-ku, Kumamoto, 860-8556, Japan.
Taizo HibiDepartment of Transplantation and Pediatric Surgery, Kumamoto University, 1-1-1 Honjo, Chuo-ku, Kumamoto, 860-8556, Japan.
Kumamoto University · JPKumamoto University Hospital · JP

Funding

No grant is acknowledged in the PubMed record.

6 · The paper itself

Abstract

Recruitment of bone marrow derived monocytes via bloodstream and their subsequent conversion to CX3CR1

Indexed as

AnimalsGATA6 Transcription FactorHumansInflammatory Bowel DiseasesIntestinesMacrophages, PeritonealMaleMiceMice, Inbred C57BLMonocytesNuclear Receptor Subfamily 4, Group A, Member 1PeritoneumReceptors, CCR2RegenerationGATA6 Transcription FactorNr4a1 protein, mouseNuclear Receptor Subfamily 4, Group A, Member 1Receptors, CCR2

Identifiers

PMID34911964
PMCPMC8674319
OpenAlexW4200262539

What OpenQuestion holds

Textmetadata
LicenceCC BY
Read underepoch 390

Registered trials

None linked

Read under generation 80e0d062 · epoch 390. Bibliography from PubMed, PubMed Central and OpenAlex; grants from NIH RePORTER; trial links from ClinicalTrials.gov; estimates, votes and beliefs from the OpenQuestion graph.