Evidence map›Paper›PMID 34868253›Full record

ArticleFrontiers in genetics2021

Discovery and Validation of a Recessively Inherited Major-Effect QTL Conferring Resistance to Maize Lethal Necrosis (MLN) Disease.

Ann Murithi, Michael S Olsen, Daniel B Kwemoi, Ogugo Veronica, Berhanu Tadesse Ertiro, Suresh L M, Yoseph Beyene, Biswanath Das, Boddupalli M Prasanna, Manje Gowda

Open access · goldAbstract read
In one paragraph

Article in Frontiers in genetics, 2021. The graph could read no effect estimate from its abstract, so it casts no vote on the map. Cited by 14 papers.

0numbers the graph read from it
0cells of the map it votes in
14citing papers in PubMed
2.9field-weighted citation impact, top 9% of its field
1 · What the graph read from it

What it found

Each row is one number read from the abstract, on the scale the paper reported it, with its interval. Left of the dashed line favours the treatment, right favours the comparator. Under each row is the sentence it came from. New to these charts? A ten-minute tutorial.

The abstract states no effect estimate the extractor could read, or names no intervention and outcome on the map, so this paper lights no cell and moves no belief. It is still indexed, cited and linked below.

2 · The registry

The trial behind it

Trials whose registry record cites this paper, or whose number appears in the abstract. A trial that started after this paper was published is citing it as background, not reporting it.

Neither the registry nor the abstract names a trial number. If this is a trial report, that itself is worth knowing.

3 · Its place in the literature

Who cites it

14 citing papers in PubMed, 19 citations in OpenAlex.

  1. Article
  2. Targeted knockout of a host peroxisomal peptidase confers field resistance to maize lethal necrosis.Proceedings of the National Academy of Sciences of the United States of America · 2026
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  14. Gene Editing to Accelerate Crop Breeding.Frontiers in plant science · 2022
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4 · The record

Corrections and comments

PubMed lists nothing against this paper. Absence here is not a guarantee, only a check that was made.

5 · Who and what money

Authors and funding

10 authors at 2 institutions in 2 countries.

Ann MurithiInternational Maize and Wheat Improvement Center (CIMMYT), Nairobi, Kenya.
Michael S OlsenInternational Maize and Wheat Improvement Center (CIMMYT), Nairobi, Kenya.
Daniel B KwemoiNational Crops Resources Research Institute (NaCRRI), Namulonge, Uganda.
Ogugo VeronicaInternational Maize and Wheat Improvement Center (CIMMYT), Nairobi, Kenya.
Berhanu Tadesse ErtiroInternational Maize and Wheat Improvement Center (CIMMYT), Nairobi, Kenya.
Suresh L MInternational Maize and Wheat Improvement Center (CIMMYT), Nairobi, Kenya.
Yoseph BeyeneInternational Maize and Wheat Improvement Center (CIMMYT), Nairobi, Kenya.
Biswanath DasInternational Maize and Wheat Improvement Center (CIMMYT), Nairobi, Kenya.
Boddupalli M PrasannaInternational Maize and Wheat Improvement Center (CIMMYT), Nairobi, Kenya.
Manje GowdaInternational Maize and Wheat Improvement Center (CIMMYT), Nairobi, Kenya.
International Maize and Wheat Improvement Center · KENational Agricultural Research Institute · UG

Funding

No grant is acknowledged in the PubMed record.

6 · The paper itself

Abstract

Maize lethal necrosis (MLN) is a viral disease with a devastating effect on maize production. Developing and deploying improved varieties with resistance to the disease is important to effectively control MLN; however, little is known about the causal genes and molecular mechanism(s) underlying MLN resistance. Screening thousands of maize inbred lines revealed KS23-5 and KS23-6 as two of the most promising donors of MLN resistance alleles. KS23-5 and KS23-6 lines were earlier developed at the University of Hawaii, United States, on the basis of a source population constituted using germplasm from Kasetsart University, Thailand. Both linkage mapping and association mapping approaches were used to discover and validate genomic regions associated with MLN resistance. Selective genotyping of resistant and susceptible individuals within large F

Indexed as

disease resistanceF2 populationgenome-wide association studymaize lethal necrosisselective genotyping

Identifiers

PMID34868253
PMCPMC8640137
OpenAlexW3217184069

What OpenQuestion holds

Textmetadata
LicenceCC BY
Read underepoch 390

Registered trials

None linked

Read under generation 80e0d062 · epoch 390. Bibliography from PubMed, PubMed Central and OpenAlex; grants from NIH RePORTER; trial links from ClinicalTrials.gov; estimates, votes and beliefs from the OpenQuestion graph.