Evidence map›Paper›PMID 34839402›Full record

ArticleHuman genetics2022

A common founder effect of the splice site variant c.-23 + 1G > A in GJB2 gene causing autosomal recessive deafness 1A (DFNB1A) in Eurasia.

Aisen V Solovyev, Alena Kushniarevich, Elena Bliznetz, Marita Bady-Khoo, Maria R Lalayants, Tatiana G Markova, Gabriel Minárik, L'udevít Kádasi, Ene Metspalu, Vera G Pshennikova and 7 more

Abstract read
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In one paragraph

Article in Human genetics, 2022. The graph could read no effect estimate from its abstract, so it casts no vote on the map. Cited by 5 papers.

0numbers the graph read from it
0cells of the map it votes in
5citing papers in PubMed
–field-weighted citation impact
1 · What the graph read from it

What it found

Each row is one number read from the abstract, on the scale the paper reported it, with its interval. Left of the dashed line favours the treatment, right favours the comparator. Under each row is the sentence it came from. New to these charts? A ten-minute tutorial.

The abstract states no effect estimate the extractor could read, or names no intervention and outcome on the map, so this paper lights no cell and moves no belief. It is still indexed, cited and linked below.

2 · The registry

The trial behind it

Trials whose registry record cites this paper, or whose number appears in the abstract. A trial that started after this paper was published is citing it as background, not reporting it.

Neither the registry nor the abstract names a trial number. If this is a trial report, that itself is worth knowing.

3 · Its place in the literature

Who cites it

5 citing papers in PubMed.

  1. Review
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  3. Article
  4. TheGenes · 2023
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  5. Review
4 · The record

Corrections and comments

PubMed lists nothing against this paper. Absence here is not a guarantee, only a check that was made.

5 · Who and what money

Authors and funding

17 authors.

Aisen V SolovyevLaboratory of Molecular Biology, Institute of Natural Sciences, M.K. Ammosov North-Eastern Federal University, Kulakovskogo 46, 677010, Yakutsk, Russia.ORCID http://orcid.org/0000-0003-0914-3609
Alena KushniarevichEstonian Biocentre, Institute of Genomics, University of Tartu, Riia 23b, 51010, Tartu, Estonia.ORCID http://orcid.org/0000-0003-1608-917X
Elena BliznetzResearch Centre for Medical Genetics, Moskvorechye st., 1, 115478, Moscow, Russia.ORCID http://orcid.org/0000-0002-5339-5566
Marita Bady-KhooFederal Research Center Institute of Cytology and Genetics, Siberian Branch of the Russian Academy of Sciences, Prospekt Lavrentyeva 10, 630090, Novosibirsk, Russia.
Maria R LalayantsNational Research Centre for Audiology and Hearing Rehabilitation, Leninsky prospect 123, 117513, Moscow, Russia.ORCID http://orcid.org/0000-0003-3975-7759
Tatiana G MarkovaNational Research Centre for Audiology and Hearing Rehabilitation, Leninsky prospect 123, 117513, Moscow, Russia.ORCID http://orcid.org/0000-0002-1086-588X
Gabriel MinárikInstitute of Molecular Biomedicine, Comenius University Faculty of Medicine, Sasinkova, 4811 08, Bratislava, Slovakia.ORCID http://orcid.org/0000-0001-9399-2202
L'udevít KádasiInstitute of Molecular Physiology and Genetics, Slovak Academy of Sciences, Dubravska cesta 9, 840 05, Bratislava, Slovakia.ORCID http://orcid.org/0000-0002-8323-099X
Ene MetspaluEstonian Biocentre, Institute of Genomics, University of Tartu, Riia 23b, 51010, Tartu, Estonia.ORCID http://orcid.org/0000-0001-6247-7291
Vera G PshennikovaLaboratory of Molecular Genetics, Yakut Science Centre of Complex Medical Problems, Yaroslavskogo 6/3, 677019, Yakutsk, Russia.ORCID http://orcid.org/0000-0001-6866-9462
Fedor M TeryutinLaboratory of Molecular Biology, Institute of Natural Sciences, M.K. Ammosov North-Eastern Federal University, Kulakovskogo 46, 677010, Yakutsk, Russia.ORCID http://orcid.org/0000-0002-8659-0886
Elza K KhusnutdinovaLaboratory of Molecular Biology, Institute of Natural Sciences, M.K. Ammosov North-Eastern Federal University, Kulakovskogo 46, 677010, Yakutsk, Russia.ORCID http://orcid.org/0000-0003-2987-3334
Alexander PoliakovResearch Centre for Medical Genetics, Moskvorechye st., 1, 115478, Moscow, Russia.
Mait MetspaluEstonian Biocentre, Institute of Genomics, University of Tartu, Riia 23b, 51010, Tartu, Estonia.ORCID http://orcid.org/0000-0003-3099-9161
Olga L PosukhFederal Research Center Institute of Cytology and Genetics, Siberian Branch of the Russian Academy of Sciences, Prospekt Lavrentyeva 10, 630090, Novosibirsk, Russia.ORCID http://orcid.org/0000-0003-1352-3591
Nikolay A BarashkovLaboratory of Molecular Genetics, Yakut Science Centre of Complex Medical Problems, Yaroslavskogo 6/3, 677019, Yakutsk, Russia. barashkov2004@mail.ru.ORCID http://orcid.org/0000-0002-6984-7934
Sardana A FedorovaLaboratory of Molecular Biology, Institute of Natural Sciences, M.K. Ammosov North-Eastern Federal University, Kulakovskogo 46, 677010, Yakutsk, Russia.ORCID http://orcid.org/0000-0002-6952-3868

Funding

european regional development fund 2014-2020.4.01.15-0012ministry of science and higher education of the russian federation FSRG-2020-0016russian foundation for basic research 18-05-60035_Arctikarussian foundation for basic research 20-015-00328_ARussian Foundation for Basic Research 19-34-60023_PerspektivaRussian State Budjet program 0259-2021-0014
6 · The paper itself

Abstract

Mutations in the GJB2 gene are known to be a major cause of autosomal recessive deafness 1A (OMIM 220290). The most common pathogenic variants of the GJB2 gene have a high ethno-geographic specificity in their distribution, being attributed to a founder effect related to the Neolithic migration routes of Homo sapiens. The c.-23 + 1G > A splice site variant is frequently found among deaf patients of both Caucasian and Asian origins. It is currently unknown whether the spread of this mutation across Eurasia is a result of the founder effect or if it could have multiple local centers of origin. To determine the origin of c.-23 + 1G > A, we reconstructed haplotypes by genotyping SNPs on an Illumina OmniExpress 730 K platform of 23 deaf individuals homozygous for this variant from different populations of Eurasia. The analyses revealed the presence of common regions of homozygosity in different individual genomes in the sample. These data support the hypothesis of the common founder effect in the distribution of the c.-23 + 1G > A variant of the GJB2 gene. Based on the published data on the c.-23 + 1G > A prevalence among 16,177 deaf people and the calculation of the TMRCA of the modified f2-haplotypes carrying this variant, we reconstructed the potential migration routes of the carriers of this mutation around the world. This analysis indicates that the c.-23 + 1G > A variant in the GJB2 gene may have originated approximately 6000 years ago in the territory of the Caucasus or the Middle East then spread throughout Europe, South and Central Asia and other regions of the world.

Indexed as

DeafnessFounder EffectConnexin 26ConnexinsHearing Loss, SensorineuralHumansMutationConnexin 26ConnexinsGJB2 protein, human

Identifiers

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Read under generation 80e0d062 · epoch 390. Bibliography from PubMed, PubMed Central and OpenAlex; grants from NIH RePORTER; trial links from ClinicalTrials.gov; estimates, votes and beliefs from the OpenQuestion graph.