Evidence map›Paper›PMID 34804306›Full record

ArticleJournal of medical cases2021

Dilated Cardiomyopathy due to the Novel

Sinda Zarrouk, Josef Finsterer, Sounira Mehri, Fatma Ourda, Saida Ben Arab, Raafik Boussada

Open access · diamondAbstract readCase Reports
PubMed Full text at PMC Publisher
In one paragraph

Article in Journal of medical cases, 2021. The graph could read no effect estimate from its abstract, so it casts no vote on the map. Cited by 2 papers.

0numbers the graph read from it
0cells of the map it votes in
2citing papers in PubMed
–field-weighted citation impact, top 89% of its field
1 · What the graph read from it

What it found

Each row is one number read from the abstract, on the scale the paper reported it, with its interval. Left of the dashed line favours the treatment, right favours the comparator. Under each row is the sentence it came from. New to these charts? A ten-minute tutorial.

The abstract states no effect estimate the extractor could read, or names no intervention and outcome on the map, so this paper lights no cell and moves no belief. It is still indexed, cited and linked below.

2 · The registry

The trial behind it

Trials whose registry record cites this paper, or whose number appears in the abstract. A trial that started after this paper was published is citing it as background, not reporting it.

Neither the registry nor the abstract names a trial number. If this is a trial report, that itself is worth knowing.

3 · Its place in the literature

Who cites it

2 citing papers in PubMed, 3 citations in OpenAlex.

  1. Article
  2. Review
4 · The record

Corrections and comments

PubMed lists nothing against this paper. Absence here is not a guarantee, only a check that was made.

5 · Who and what money

Authors and funding

6 authors at 3 institutions in 2 countries.

Sinda ZarroukDepartment of Genetic and Molecular Epidemiology, Medical University of Tunis, Tunisia.
Josef FinstererCity Hospital Landstrasse, Messerli Institute, Vienna, Austria.
Sounira MehriDepartment of Genetic and Molecular Epidemiology, Medical University of Tunis, Tunisia.
Fatma OurdaDepartment of Functional Cardiology, La Rabta Hospital of Tunis, Tunisia.
Saida Ben ArabDepartment of Genetic and Molecular Epidemiology, Medical University of Tunis, Tunisia.
Raafik BoussadaDepartment of Functional Cardiology, La Rabta Hospital of Tunis, Tunisia.
Tunis University · TNHôpital La Rabta · TNVienna General Hospital · AT

Funding

No grant is acknowledged in the PubMed record.

6 · The paper itself

Abstract

Mitochondrial DNA (mtDNA) mutations frequently manifest with multisystem disease, including cardiomyopathy (CM). Various studies described mutations in protein-encoding mtDNA genes, such as cytochrome-b, manifesting with CM. A detailed clinical, biochemical, and molecular genetic analysis was performed in a 40-year-old male with dilated CM (DCM) to detect the underlying mtDNA defect. Muscle biopsy showed complex-III deficiency, and sequencing of the cytochrome-b gene revealed the pathogenic variant m.14757T>C in

Indexed as

Cytochrome-b geneDilated cardiomyopathyMitochondriaMitochondrialmtDNAOxidative phosphorylationSystolic dysfunction

Identifiers

PMID34804306
PMCPMC8577615
OpenAlexW3211681752

What OpenQuestion holds

Textmetadata
LicenceCC BY-NC
Read underepoch 390

Registered trials

None linked

Read under generation 80e0d062 · epoch 390. Bibliography from PubMed, PubMed Central and OpenAlex; grants from NIH RePORTER; trial links from ClinicalTrials.gov; estimates, votes and beliefs from the OpenQuestion graph.