Evidence map›Paper›PMID 34731533›Full record

ArticleJournal of thrombosis and haemostasis : JTH2022

Persistent endotheliopathy in the pathogenesis of long COVID syndrome: Comment from von Meijenfeldt et al.

Fien A von Meijenfeldt, Sebastian Havervall, Jelle Adelmeijer, Charlotte Thalin, Ton Lisman

Registry-linked trialAbstract readLetterComment
In one paragraph

Article in Journal of thrombosis and haemostasis : JTH, 2022. The graph could read no effect estimate from its abstract, so it casts no vote on the map. It is linked to trial NCT06784739 (COMMUNITY - the COVID-19 Immunity Study), which is not on this map. Cited by 8 papers.

0numbers the graph read from it
0cells of the map it votes in
8citing papers in PubMed
–field-weighted citation impact
1 · What the graph read from it

What it found

Each row is one number read from the abstract, on the scale the paper reported it, with its interval. Left of the dashed line favours the treatment, right favours the comparator. Under each row is the sentence it came from. New to these charts? A ten-minute tutorial.

The abstract states no effect estimate the extractor could read, or names no intervention and outcome on the map, so this paper lights no cell and moves no belief. It is still indexed, cited and linked below.

2 · The registry

The trial behind it

Trials whose registry record cites this paper, or whose number appears in the abstract. A trial that started after this paper was published is citing it as background, not reporting it.

NCT06784739 recruitingnot on this map

COMMUNITY - the COVID-19 Immunity Study

TypeobservationalSponsorDanderyd HospitalRan2020 to 2027Enrolled5,500ConditionsCOVID 19, Respiratory Synctial Virus, Influenza Virus, SARS-CoV-2 VirusArmsBlood sampling, Mucosal sampling, PCR-test
3 · Its place in the literature

Who cites it

8 citing papers in PubMed.

  1. Article
  2. Article
  3. Neurobiology of long-COVID: Hypotheses and unanswered questions.Anaesthesia, critical care & pain medicine · 2023
    Article
  4. Article
  5. The suboptimal fibrinolytic response in COVID-19 is dictated by high PAI-1.Journal of thrombosis and haemostasis : JTH · 2022
    Article
  6. Article
  7. Review
  8. Article
4 · The record

Corrections and comments

5 · Who and what money

Authors and funding

5 authors.

Fien A von MeijenfeldtSurgical Research Laboratory, Department of Surgery, University of Groningen, University Medical Center Groningen, Groningen, the Netherlands.ORCID 0000-0003-0274-2414
Sebastian HavervallDivision of Internal Medicine, Department of Clinical Sciences, Danderyd Hospital, Karolinska Institutet, Stockholm, Sweden.
Jelle AdelmeijerSurgical Research Laboratory, Department of Surgery, University of Groningen, University Medical Center Groningen, Groningen, the Netherlands.
Charlotte ThalinDivision of Internal Medicine, Department of Clinical Sciences, Danderyd Hospital, Karolinska Institutet, Stockholm, Sweden.
Ton LismanSurgical Research Laboratory, Department of Surgery, University of Groningen, University Medical Center Groningen, Groningen, the Netherlands.ORCID 0000-0002-3503-7140

Funding

No grant is acknowledged in the PubMed record.

6 · The paper itself

Abstract

PubMed holds no abstract for this paper.

Indexed as

COVID-19Endothelial CellsHumansPost-Acute COVID-19 SyndromeSARS-CoV-2

Identifiers

PMID34731533
PMCPMC8646477

What OpenQuestion holds

Textmetadata
Read underepoch 390

Registered trials

Read under generation 80e0d062 · epoch 390. Bibliography from PubMed, PubMed Central and OpenAlex; grants from NIH RePORTER; trial links from ClinicalTrials.gov; estimates, votes and beliefs from the OpenQuestion graph.