Evidence map›Paper›PMID 34696365›Full record

ReviewViruses2021

Roles of Virion-Incorporated CD162 (PSGL-1), CD43, and CD44 in HIV-1 Infection of T Cells.

Tomoyuki Murakami, Akira Ono

Open access · goldAbstract readReview
In one paragraph

Review in Viruses, 2021. The graph could read no effect estimate from its abstract, so it casts no vote on the map. Cited by 6 papers.

0numbers the graph read from it
0cells of the map it votes in
6citing papers in PubMed
1.3field-weighted citation impact, top 22% of its field
1 · What the graph read from it

What it found

Each row is one number read from the abstract, on the scale the paper reported it, with its interval. Left of the dashed line favours the treatment, right favours the comparator. Under each row is the sentence it came from. New to these charts? A ten-minute tutorial.

The abstract states no effect estimate the extractor could read, or names no intervention and outcome on the map, so this paper lights no cell and moves no belief. It is still indexed, cited and linked below.

2 · The registry

The trial behind it

Trials whose registry record cites this paper, or whose number appears in the abstract. A trial that started after this paper was published is citing it as background, not reporting it.

Neither the registry nor the abstract names a trial number. If this is a trial report, that itself is worth knowing.

3 · Its place in the literature

Who cites it

6 citing papers in PubMed, 11 citations in OpenAlex.

  1. Article
  2. PIPScience advances · 2025
    Article
  3. Review
  4. Article
  5. Article
  6. Article
4 · The record

Corrections and comments

PubMed lists nothing against this paper. Absence here is not a guarantee, only a check that was made.

5 · Who and what money

Authors and funding

2 authors at 1 institution in 1 country.

Tomoyuki MurakamiDepartment of Microbiology & Immunology, University of Michigan Medical School, Ann Arbor, MI 48109-0620, USA.ORCID 0000-0002-6645-2240
Akira OnoDepartment of Microbiology & Immunology, University of Michigan Medical School, Ann Arbor, MI 48109-0620, USA.ORCID 0000-0001-7841-851X
University of Michigan · US

Funding

Mechanisms that determine subcellular sites of HIV-1 assemblyR37AI071727 · NIAID · UNIVERSITY OF MICHIGAN AT ANN ARBOR · PI Akira Ono · 2017 to 2026
$6.2M
Effects of virion-incorporated CD43, CD44, and PSGL-1 on HIV-1 infectionR21AI148381 · NIAID · UNIVERSITY OF MICHIGAN AT ANN ARBOR · PI ONO, AKIRA · 2019 to 2020
$429k
NIAID NIH HHS R21 AI148381NIAID NIH HHS R37 AI071727NIH HHS R21 AI 148381NIH HHS R37 AI 071727
6 · The paper itself

Abstract

Nascent HIV-1 particles incorporate the viral envelope glycoprotein and multiple host transmembrane proteins during assembly at the plasma membrane. At least some of these host transmembrane proteins on the surface of virions are reported as pro-viral factors that enhance virus attachment to target cells or facilitate trans-infection of CD4

Indexed as

Cell Membranegag Gene Products, Human Immunodeficiency VirusHIV-1HIV InfectionsHost-Pathogen InteractionsHumansHyaluronan ReceptorsLeukosialinMembrane GlycoproteinsMembrane ProteinsT-LymphocytesVirionVirus AssemblyVirus AttachmentCD44 protein, humangag Gene Products, Human Immunodeficiency VirusHyaluronan ReceptorsLeukosialinMembrane GlycoproteinsMembrane ProteinsP-selectin ligand proteinSPN protein, humanCD43CD44PSGL-1trans-infectiontransmembrane proteinsvirion incorporationvirus attachment

Identifiers

PMID34696365
PMCPMC8541244
OpenAlexW3202270089

What OpenQuestion holds

Textmetadata
LicenceCC BY
Read underepoch 390

Registered trials

None linked

Read under generation 80e0d062 · epoch 390. Bibliography from PubMed, PubMed Central and OpenAlex; grants from NIH RePORTER; trial links from ClinicalTrials.gov; estimates, votes and beliefs from the OpenQuestion graph.