Evidence map›Paper›PMID 34664800›Full record

ArticleJournal of Korean medical science2021

Innate Type 2 Response to

Arum Park, Eun Lee, Hyojung Park, Mee-Na Park, Jiho Lee, Kun Baek Song, Jisun Yoon, Sungsu Jung, Nayoung Suh, Jin Yoon and 1 more

Open access · goldAbstract read
PubMed Full text at PMC Publisher
In one paragraph

Article in Journal of Korean medical science, 2021. The graph could read no effect estimate from its abstract, so it casts no vote on the map. Cited by 3 papers.

0numbers the graph read from it
0cells of the map it votes in
3citing papers in PubMed
0.4field-weighted citation impact, top 42% of its field
1 · What the graph read from it

What it found

Each row is one number read from the abstract, on the scale the paper reported it, with its interval. Left of the dashed line favours the treatment, right favours the comparator. Under each row is the sentence it came from. New to these charts? A ten-minute tutorial.

The abstract states no effect estimate the extractor could read, or names no intervention and outcome on the map, so this paper lights no cell and moves no belief. It is still indexed, cited and linked below.

2 · The registry

The trial behind it

Trials whose registry record cites this paper, or whose number appears in the abstract. A trial that started after this paper was published is citing it as background, not reporting it.

Neither the registry nor the abstract names a trial number. If this is a trial report, that itself is worth knowing.

3 · Its place in the literature

Who cites it

3 citing papers in PubMed, 6 citations in OpenAlex.

  1. Article
  2. Article
  3. Article
4 · The record

Corrections and comments

PubMed lists nothing against this paper. Absence here is not a guarantee, only a check that was made.

5 · Who and what money

Authors and funding

11 authors at 4 institutions in 1 country.

Arum Park *Asan Institute for Life Sciences, Asan Medical Center, Seoul, Korea.ORCID https://orcid.org/0000-0001-7415-8220
Eun Lee *Department of Pediatrics, Chonnam National University Hospital, Chonnam National University Medical School, Gwangju, Korea.ORCID https://orcid.org/0000-0002-0145-7067
Hyojung ParkAsan Institute for Life Sciences, Asan Medical Center, Seoul, Korea.ORCID https://orcid.org/0000-0002-5540-1413
Mee-Na ParkAsan Institute for Life Sciences, Asan Medical Center, Seoul, Korea.ORCID https://orcid.org/0000-0002-8066-0134
Jiho LeeDepartment of Medical Science, Asan Medical Institute of Convergence Science and Technology, Asan Medical Center, University of Ulsan College of Medicine, Seoul, Korea.ORCID https://orcid.org/0000-0002-8609-6386
Kun Baek SongDepartment of Pediatrics, Asan Medical Center, University of Ulsan College of Medicine, Seoul, Korea.ORCID https://orcid.org/0000-0001-6981-6292
Jisun YoonDepartment of Pediatrics, Mediplex Sejong Hospital, Incheon, Korea.ORCID https://orcid.org/0000-0002-4904-9118
Sungsu JungDepartment of Pediatrics, Pusan National University Yangsan Hospital, Yangsan, Korea.ORCID https://orcid.org/0000-0002-0559-4982
Nayoung SuhDepartment of Pharmaceutical Engineering, College of Medical Sciences and Department of Medical Sciences, General Graduate School, Soon Chun Hyang University, Asan, Korea.ORCID https://orcid.org/0000-0002-2755-9520
Jin YoonAsan Institute for Life Sciences, Asan Medical Center, Seoul, Korea. yoonjin8787@gmail.com.ORCID https://orcid.org/0000-0003-4355-6888
Jinho YuDepartment of Pediatrics, Asan Medical Center, University of Ulsan College of Medicine, Seoul, Korea. jyu3922@gmail.com.ORCID https://orcid.org/0000-0002-1226-8077
Asan Medical Center · KRUlsan College · KRChonnam National University Hospital · KRPusan National University Yangsan Hospital · KR

Funding

National Research Foundation of Korea NRF-2018R1D1A1B07049168
6 · The paper itself

Abstract

backgroundAtopic dermatitis (AD) is a chronic and relapsing inflammatory skin disease mediated by T helper type 2 (Th2) cells in acute phase. Group 2 innate lymphoid cells (ILCs) play a role in the initiation of the Th2 response. Although mold exposure is associated with the development of AD, studies on the underlying mechanisms are lacking. This study investigated whether group 2 ILCs are involved in inflammation in AD-like skin induced by

methodsWe investigated changes of group 2 ILCs population in

resultsThe clinical parameters were higher in the

conclusionsGroup 2 ILCs may play a role in a murine model of

Indexed as

Immunity, InnateAnimalsAntibodies, MonoclonalAspergillus fumigatusDermatitis, AtopicDisease Models, AnimalFemaleHomeodomain ProteinsImmunoglobulin EInterleukin-33MiceMice, Inbred BALB CMice, KnockoutSkinTh2 CellsThy-1 AntigensAntibodies, MonoclonalHomeodomain ProteinsImmunoglobulin EInterleukin-33RAG-1 proteinThy-1 AntigensAspergillus fumigatusAtopic DermatitisGroup 2 Innate Lymphoid CellsT Helper 2 Response

Identifiers

PMID34664800
PMCPMC8524237
OpenAlexW3199261510

What OpenQuestion holds

Textmetadata
LicenceCC BY-NC
Read underepoch 390

Registered trials

None linked

Read under generation 80e0d062 · epoch 390. Bibliography from PubMed, PubMed Central and OpenAlex; grants from NIH RePORTER; trial links from ClinicalTrials.gov; estimates, votes and beliefs from the OpenQuestion graph.