Evidence map›Paper›PMID 34574656›Full record

ArticleInternational journal of environmental research and public health2021

Arsenic Secondary Methylation Capacity Is Inversely Associated with Arsenic Exposure-Related Muscle Mass Reduction.

Md Khalequzzaman Sarker, Selim Reza Tony, Abu Eabrahim Siddique, Md Rezaul Karim, Nazmul Haque, Zohurul Islam, Md Shofikul Islam, Moriom Khatun, Jahidul Islam, Shakhawoat Hossain and 6 more

Open access · goldAbstract read
In one paragraph

Article in International journal of environmental research and public health, 2021. The graph could read no effect estimate from its abstract, so it casts no vote on the map. Cited by 13 papers.

0numbers the graph read from it
0cells of the map it votes in
13citing papers in PubMed
1.2field-weighted citation impact, top 23% of its field
1 · What the graph read from it

What it found

Each row is one number read from the abstract, on the scale the paper reported it, with its interval. Left of the dashed line favours the treatment, right favours the comparator. Under each row is the sentence it came from. New to these charts? A ten-minute tutorial.

The abstract states no effect estimate the extractor could read, or names no intervention and outcome on the map, so this paper lights no cell and moves no belief. It is still indexed, cited and linked below.

2 · The registry

The trial behind it

Trials whose registry record cites this paper, or whose number appears in the abstract. A trial that started after this paper was published is citing it as background, not reporting it.

Neither the registry nor the abstract names a trial number. If this is a trial report, that itself is worth knowing.

3 · Its place in the literature

Who cites it

13 citing papers in PubMed, 16 citations in OpenAlex.

  1. Article
  2. Underexplored terrain: effects of high priority environmental toxicants on skeletal muscle.Journal of toxicology and environmental health. Part B, Critical reviews · 2026
    Review
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  8. Arsenic disrupts extracellular vesicle-mediated signaling in regenerating myofibers.Toxicological sciences : an official journal of the Society of Toxicology · 2023
    Article
  9. Article
  10. Article
  11. Reproductive-Toxicity-Related Endpoints inJournal of developmental biology · 2023
    Article
  12. Review
  13. Review
4 · The record

Corrections and comments

PubMed lists nothing against this paper. Absence here is not a guarantee, only a check that was made.

5 · Who and what money

Authors and funding

16 authors at 5 institutions in 3 countries.

Md Khalequzzaman SarkerInstitute of Biological Sciences, University of Rajshahi, Rajshahi 6205, Bangladesh.
Selim Reza TonyDepartment of Biochemistry and Molecular Biology, University of Rajshahi, Rajshahi 6205, Bangladesh.ORCID 0000-0002-1601-0727
Abu Eabrahim SiddiqueDepartment of Biochemistry and Molecular Biology, University of Rajshahi, Rajshahi 6205, Bangladesh.ORCID 0000-0003-1762-6379
Md Rezaul KarimDepartment of Applied Nutrition and Food Technology, Islamic University, Kushtia 7003, Bangladesh.
Nazmul HaqueDepartment of Biochemistry and Molecular Biology, University of Rajshahi, Rajshahi 6205, Bangladesh.
Zohurul IslamDepartment of Biochemistry and Molecular Biology, University of Rajshahi, Rajshahi 6205, Bangladesh.
Md Shofikul IslamDepartment of Applied Nutrition and Food Technology, Islamic University, Kushtia 7003, Bangladesh.
Moriom KhatunDepartment of Biochemistry and Molecular Biology, University of Rajshahi, Rajshahi 6205, Bangladesh.
Jahidul IslamDepartment of Biochemistry and Molecular Biology, University of Rajshahi, Rajshahi 6205, Bangladesh.
Shakhawoat HossainDepartment of Biochemistry and Molecular Biology, University of Rajshahi, Rajshahi 6205, Bangladesh.
Zahangir Alam SaudDepartment of Biochemistry and Molecular Biology, University of Rajshahi, Rajshahi 6205, Bangladesh.
Hideki MiyatakaLaboratory of Molecular Nutrition and Toxicology, Faculty of Pharmaceutical Sciences, Tokushima Bunri University, Tokushima 770-8514, Japan.
Daigo SumiLaboratory of Molecular Nutrition and Toxicology, Faculty of Pharmaceutical Sciences, Tokushima Bunri University, Tokushima 770-8514, Japan.ORCID 0000-0002-1585-8013
Aaron BarchowskyDepartment of Environmental and Occupational Health, University of Pittsburgh, Pittsburgh, PA 15261, USA.
Seiichiro HimenoLaboratory of Molecular Nutrition and Toxicology, Faculty of Pharmaceutical Sciences, Tokushima Bunri University, Tokushima 770-8514, Japan.ORCID 0000-0002-0773-2373
Khaled HossainDepartment of Biochemistry and Molecular Biology, University of Rajshahi, Rajshahi 6205, Bangladesh.
University of Rajshahi · BDIslamic University · BDTokushima Bunri University · JPShowa University · JPUniversity of Pittsburgh · US

Funding

No grant is acknowledged in the PubMed record.

6 · The paper itself

Abstract

Skeletal muscle mass reduction has been implicated in insulin resistance (IR) that promotes cardiometabolic diseases. We have previously reported that arsenic exposure increases IR concomitantly with the reduction of skeletal muscle mass among individuals exposed to arsenic. The arsenic methylation capacity is linked to the susceptibility to some arsenic exposure-related diseases. However, it remains unknown whether the arsenic methylation capacity affects the arsenic-induced reduction of muscle mass and elevation of IR. Therefore, this study examined the associations between the arsenic methylation status and skeletal muscle mass measures with regard to IR by recruiting 437 participants from low- and high-arsenic exposure areas in Bangladesh. The subjects' skeletal muscle mass was estimated by their lean body mass (LBM) and serum creatinine levels. Subjects' drinking water arsenic concentrations were positively associated with total urinary arsenic concentrations and the percentages of MMA, as well as inversely associated with the percentages of DMA and the secondary methylation index (SMI). Subjects' LBM and serum creatinine levels were positively associated with the percentage of DMA and SMI, as well as inversely associated with the percentage of MMA. HOMA-IR showed an inverse association with SMI, with a confounding effect of sex. Our results suggest that reduced secondary methylation capacity is involved in the arsenic-induced skeletal muscle loss that may be implicated in arsenic-induced IR and cardiometabolic diseases.

Indexed as

ArsenicArsenicalsEnvironmental ExposureHumansMethylationMuscle, SkeletalArsenicArsenicalsarseniccardiometabolic diseasediabetesinsulin resistancemetabolitesmuscle mass

Identifiers

PMID34574656
PMCPMC8472591
OpenAlexW3199219841

What OpenQuestion holds

Textmetadata
LicenceCC BY
Read underepoch 390

Registered trials

None linked

Read under generation 80e0d062 · epoch 390. Bibliography from PubMed, PubMed Central and OpenAlex; grants from NIH RePORTER; trial links from ClinicalTrials.gov; estimates, votes and beliefs from the OpenQuestion graph.