Evidence map›Paper›PMID 34566890›Full record

ReviewFrontiers in endocrinology2021

Mechanisms of Post-Pancreatitis Diabetes Mellitus and Cystic Fibrosis-Related Diabetes: A Review of Preclinical Studies.

Eleonóra Gál, Jurij Dolenšek, Andraž Stožer, László Czakó, Attila Ébert, Viktória Venglovecz

Open access · goldAbstract readReview
In one paragraph

Review in Frontiers in endocrinology, 2021. The graph could read no effect estimate from its abstract, so it casts no vote on the map. Cited by 8 papers.

0numbers the graph read from it
0cells of the map it votes in
8citing papers in PubMed
2.5field-weighted citation impact, top 10% of its field
1 · What the graph read from it

What it found

Each row is one number read from the abstract, on the scale the paper reported it, with its interval. Left of the dashed line favours the treatment, right favours the comparator. Under each row is the sentence it came from. New to these charts? A ten-minute tutorial.

The abstract states no effect estimate the extractor could read, or names no intervention and outcome on the map, so this paper lights no cell and moves no belief. It is still indexed, cited and linked below.

2 · The registry

The trial behind it

Trials whose registry record cites this paper, or whose number appears in the abstract. A trial that started after this paper was published is citing it as background, not reporting it.

Neither the registry nor the abstract names a trial number. If this is a trial report, that itself is worth knowing.

3 · Its place in the literature

Who cites it

8 citing papers in PubMed, 14 citations in OpenAlex.

  1. Article
  2. Article
  3. Review
  4. Review
  5. Article
  6. Review
  7. Review
  8. Article
4 · The record

Corrections and comments

PubMed lists nothing against this paper. Absence here is not a guarantee, only a check that was made.

5 · Who and what money

Authors and funding

6 authors at 2 institutions in 2 countries.

Eleonóra GálDepartment of Pharmacology and Pharmacotherapy, University of Szeged, Szeged, Hungary.
Jurij DolenšekFaculty of Medicine, University of Maribor, Maribor, Slovenia.
Andraž StožerFaculty of Medicine, University of Maribor, Maribor, Slovenia.
László CzakóFirst Department of Medicine, University of Szeged, Szeged, Hungary.
Attila ÉbertDepartment of Pharmacology and Pharmacotherapy, University of Szeged, Szeged, Hungary.
Viktória VengloveczDepartment of Pharmacology and Pharmacotherapy, University of Szeged, Szeged, Hungary.
University of Szeged · HUUniversity of Maribor · SI

Funding

No grant is acknowledged in the PubMed record.

6 · The paper itself

Abstract

Anatomical proximity and functional correlations between the exocrine and endocrine pancreas warrant reciprocal effects between the two parts. Inflammatory diseases of the exocrine pancreas, such as acute or chronic pancreatitis, or the presence of cystic fibrosis disrupt endocrine function, resulting in diabetes of the exocrine pancreas. Although novel mechanisms are being increasingly identified, the intra- and intercellular pathways regulating exocrine-endocrine interactions are still not fully understood, making the development of new and more effective therapies difficult. Therefore, this review sought to accumulate current knowledge regarding the pathogenesis of diabetes in acute and chronic pancreatitis, as well as cystic fibrosis.

Indexed as

AnimalsCystic FibrosisDiabetes MellitusHumansPancreatitisacute pancreatitis (AP)chronic pancreatitis (CP)cystic fibrosis (CF)diabetes of the exocrine pancreas (DEP)interaction

Identifiers

PMID34566890
PMCPMC8461102
OpenAlexW3199357779

What OpenQuestion holds

Textmetadata
LicenceCC BY
Read underepoch 390

Registered trials

None linked

Read under generation 80e0d062 · epoch 390. Bibliography from PubMed, PubMed Central and OpenAlex; grants from NIH RePORTER; trial links from ClinicalTrials.gov; estimates, votes and beliefs from the OpenQuestion graph.