ReviewJournal of applied genetics2022
Congenital amusia-pathology of musical disorder.
Review in Journal of applied genetics, 2022. The graph could read no effect estimate from its abstract, so it casts no vote on the map. Cited by 4 papers.
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The abstract states no effect estimate the extractor could read, or names no intervention and outcome on the map, so this paper lights no cell and moves no belief. It is still indexed, cited and linked below.
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Who cites it
4 citing papers in PubMed.
- Expressive rhythm training improves consistency of rhythm production abilities in patients with schizophrenia.Fukushima journal of medical science · 2026Article
- The neuroscience of music perception: a narrative review.Arquivos de neuro-psiquiatria · 2025Review
- In Vitro Fertilization and Hearing Impairment in Twins: A Comprehensive Analysis of Perinatal Factors.Journal of the American Academy of Audiology · 2025Article
- Associations between major depressive disorder and performance-based and self-reported music cognition.Frontiers in psychology · 2024Article
Corrections and comments
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Authors and funding
2 authors.
Funding
No grant is acknowledged in the PubMed record.
Abstract
Amusia also known as tone deafness affects roughly 1.5% population. Congenital amusia appears from birth and lasts over life span. Usually, it is not associated with other diseases. Its link to hearing impairment has been definitively excluded. Neurobiological studies point to asymmetrical processing of musical signals in auditory cortex of left and right brain hemispheres. The finding was supported by discovering microlesions in the right-side gray matter. Because of its connection with asymmetry, amusia has been classified to disconnection syndromes. Alternatively to the neurobiological explanation of amusia background, an attention was turned to the significance of genetic factors. The studies done on relatives and twins indicated familial aggregation of amusia. Molecular genetic investigations linked amusia with deletion of 22q11.2 chromosome region. Until now no specific genes responsible for development of amusia were found.
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