Evidence map›Paper›PMID 34526433›Full record

Observational studyJournal of atherosclerosis and thrombosis2022

Familial Hypercholesterolemia in Patients with Acute Coronary Syndrome: Genetic Insights from EXPLORE-J.

Mariko Harada-Shiba, Junya Ako, Atsushi Hirayama, Masato Nakamura, Atsushi Nohara, Kayoko Sato, Yoshitaka Murakami, Ryusuke Koshida, Asuka Ozaki, Hidenori Arai

Open access · diamondAbstract readMulticenter StudyObservational Study
In one paragraph

Observational study in Journal of atherosclerosis and thrombosis, 2022. The graph could read no effect estimate from its abstract, so it casts no vote on the map. Cited by 9 papers.

0numbers the graph read from it
0cells of the map it votes in
9citing papers in PubMed
2.1field-weighted citation impact, top 13% of its field
1 · What the graph read from it

What it found

Each row is one number read from the abstract, on the scale the paper reported it, with its interval. Left of the dashed line favours the treatment, right favours the comparator. Under each row is the sentence it came from. New to these charts? A ten-minute tutorial.

The abstract states no effect estimate the extractor could read, or names no intervention and outcome on the map, so this paper lights no cell and moves no belief. It is still indexed, cited and linked below.

2 · The registry

The trial behind it

Trials whose registry record cites this paper, or whose number appears in the abstract. A trial that started after this paper was published is citing it as background, not reporting it.

Neither the registry nor the abstract names a trial number. If this is a trial report, that itself is worth knowing.

3 · Its place in the literature

Who cites it

9 citing papers in PubMed, 11 citations in OpenAlex.

  1. Observational
  2. Article
  3. Exploration Continues.Journal of atherosclerosis and thrombosis · 2025
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  8. Review
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4 · The record

Corrections and comments

PubMed lists nothing against this paper. Absence here is not a guarantee, only a check that was made.

5 · Who and what money

Authors and funding

10 authors at 9 institutions in 2 countries.

Mariko Harada-ShibaDepartment of Molecular Pathogenesis, National Cerebral and Cardiovascular Center Research Institute.
Junya AkoDepartment of Cardiovascular Medicine, Kitasato University.
Atsushi HirayamaCardiovascular Division, Osaka Police Hospital.
Masato NakamuraDivision of Cardiovascular Medicine, Toho University Ohashi Medical Center.
Atsushi NoharaDepartment of Clinical Genetics, Ishikawa Prefectural Central Hospital.
Kayoko SatoDepartment of Cardiology, Tokyo Women's Medical University.
Yoshitaka MurakamiDepartment of Medical Statistics, Toho University.
Ryusuke KoshidaSanofi K.K.
Asuka OzakiSanofi K.K.
Hidenori AraiNational Center for Geriatrics and Gerontology.
Sanofi (Mexico) · MXIshikawa Prefectural Central Hospital · JPKitasato University · JPNational Center for Geriatrics and Gerontology · JPNational Cerebral and Cardiovascular Center · JPOsaka Police Hospital · JPToho University · JPToho University Ohashi Medical Center · JPTokyo Women's Medical University · JP

Funding

No grant is acknowledged in the PubMed record.

6 · The paper itself

Abstract

aimGenetic testing can provide a definitive diagnosis of familial hypercholesterolemia (FH). However, accessibility of genetic testing may be limited in certain countries where it is not considered "standard of care," including Japan. In addition, mutations responsible for FH cannot be identified in approximately 30% of patients.

methodsEXPLORE-J is a multicenter, prospective, observational study of patients presenting with acute coronary syndrome (ACS). The genetic data were analyzed and adjudicated as pathogenic, indeterminate, or nondetectable pathogenic variant.

resultsOf 1,944 patients, 431 underwent genetic screening. Overall, most patients had nonpathogenic variants of LDLR, LDLRAP1, or PCSK9 (n=396, 91.9%). Of the 25 (5.8%) patients with pathogenic variants, variants of the LDLR gene and the PCSK9 gene were seen in 10 and 15 patients, respectively. Indeterminate variants were observed in 10 (2.3%) patients. Of the 431 patients, eight (1.9%) met the criteria for a diagnosis of FH using the Japanese Atherosclerosis Society (JAS) 2017 guidelines. When genetic data were incorporated, 33 (7.7%) patients met the JAS guidelines. No patients with FH pathogenic variants satisfied the JAS clinical criteria for a diagnosis of FH.

conclusionsThe results revealed a higher prevalence of genetic mutations of FH among Japanese patients with ACS and a low sensitivity of the FH diagnostic criteria of the JAS 2017 guidelines. These findings highlight the difficulties of FH diagnosis in patients with ACS in the acute phase and suggest the importance of genetic testing and family history.

Indexed as

Acute Coronary SyndromeAtherosclerosisHyperlipoproteinemia Type IIHumansMutationPhenotypeProprotein Convertase 9Prospective StudiesReceptors, LDLRisk FactorsPCSK9 protein, humanProprotein Convertase 9Receptors, LDLAcute coronary syndromeFamilial hypercholesterolemiaGenetic testingJapan

Identifiers

PMID34526433
PMCPMC9371751
OpenAlexW3200557106

What OpenQuestion holds

Texttitle and abstract
LicenceCC BY-NC-SA
Read underepoch 390

Registered trials

None linked

Read under generation 80e0d062 · epoch 390. Bibliography from PubMed, PubMed Central and OpenAlex; grants from NIH RePORTER; trial links from ClinicalTrials.gov; estimates, votes and beliefs from the OpenQuestion graph.