Evidence map›Paper›PMID 34440516›Full record

ArticleLife (Basel, Switzerland)2021

A Comprehensive Analysis of Hungarian MODY Patients-Part II: Glucokinase MODY Is the Most Prevalent Subtype Responsible for about 70% of Confirmed Cases.

Zsolt Gaál, Zsuzsanna Szűcs, Irén Kántor, Andrea Luczay, Péter Tóth-Heyn, Orsolya Benn, Enikő Felszeghy, Zsuzsanna Karádi, László Madar, István Balogh

Open access · goldAbstract read
PubMed Full text at PMC Publisher
In one paragraph

Article in Life (Basel, Switzerland), 2021. The graph could read no effect estimate from its abstract, so it casts no vote on the map. Cited by 2 papers.

0numbers the graph read from it
0cells of the map it votes in
2citing papers in PubMed
1.5field-weighted citation impact, top 18% of its field
1 · What the graph read from it

What it found

Each row is one number read from the abstract, on the scale the paper reported it, with its interval. Left of the dashed line favours the treatment, right favours the comparator. Under each row is the sentence it came from. New to these charts? A ten-minute tutorial.

The abstract states no effect estimate the extractor could read, or names no intervention and outcome on the map, so this paper lights no cell and moves no belief. It is still indexed, cited and linked below.

2 · The registry

The trial behind it

Trials whose registry record cites this paper, or whose number appears in the abstract. A trial that started after this paper was published is citing it as background, not reporting it.

Neither the registry nor the abstract names a trial number. If this is a trial report, that itself is worth knowing.

3 · Its place in the literature

Who cites it

2 citing papers in PubMed, 8 citations in OpenAlex.

  1. Review
  2. Article
4 · The record

Corrections and comments

PubMed lists nothing against this paper. Absence here is not a guarantee, only a check that was made.

5 · Who and what money

Authors and funding

10 authors at 4 institutions in 1 country.

Zsolt Gaál4th Department of Medicine, Jósa András Teaching Hospital, 4400 Nyíregyháza, Hungary.
Zsuzsanna SzűcsDivision of Clinical Genetics, Department of Laboratory Medicine, Faculty of Medicine, University of Debrecen, 4032 Debrecen, Hungary.
Irén KántorDepartment of Pediatrics, Jósa András Teaching Hospital, 4400 Nyíregyháza, Hungary.ORCID 0000-0002-6372-5805
Andrea Luczay1st Department of Pediatrics, Semmelweis University, 1085 Budapest, Hungary.
Péter Tóth-Heyn1st Department of Pediatrics, Semmelweis University, 1085 Budapest, Hungary.ORCID 0000-0002-7521-5044
Orsolya BennDepartment of Pediatrics, Szent György Hospital of Fejér County, 8000 Székesfehérvár, Hungary.
Enikő FelszeghyDepartment of Pediatrics, Faculty of Medicine, University of Debrecen, 4032 Debrecen, Hungary.
Zsuzsanna KarádiDepartment of Pediatrics, Szent György Hospital of Fejér County, 8000 Székesfehérvár, Hungary.
László MadarDivision of Clinical Genetics, Department of Laboratory Medicine, Faculty of Medicine, University of Debrecen, 4032 Debrecen, Hungary.
István BaloghDivision of Clinical Genetics, Department of Laboratory Medicine, Faculty of Medicine, University of Debrecen, 4032 Debrecen, Hungary.ORCID 0000-0003-3397-2829
University of Debrecen · HUFejér Megyei Szent György Egyetemi Oktató Kórház · HUJósa András Múzeum · HUSemmelweis University · HU

Funding

Hungarian Scientific Research Fund K109076Ministry of National Economy GINOP-2.3.2-15-2016-00039
6 · The paper itself

Abstract

MODY2 is caused by heterozygous inactivating mutations in the glucokinase (

Indexed as

GCK-MODYGCK mutationsHungaryMODY2

Identifiers

PMID34440516
PMCPMC8400228
OpenAlexW3187587643

What OpenQuestion holds

Textmetadata
LicenceCC BY
Read underepoch 390

Registered trials

None linked

Read under generation 80e0d062 · epoch 390. Bibliography from PubMed, PubMed Central and OpenAlex; grants from NIH RePORTER; trial links from ClinicalTrials.gov; estimates, votes and beliefs from the OpenQuestion graph.