Evidence map›Paper›PMID 34386847›Full record

ArticlePflugers Archiv : European journal of physiology2021

K

Simon Schemke, Cor de Wit

Open access · hybridAbstract read
In one paragraph

Article in Pflugers Archiv : European journal of physiology, 2021. The graph could read no effect estimate from its abstract, so it casts no vote on the map. Cited by 2 papers.

0numbers the graph read from it
0cells of the map it votes in
2citing papers in PubMed
0.5field-weighted citation impact, top 27% of its field
1 · What the graph read from it

What it found

Each row is one number read from the abstract, on the scale the paper reported it, with its interval. Left of the dashed line favours the treatment, right favours the comparator. Under each row is the sentence it came from. New to these charts? A ten-minute tutorial.

The abstract states no effect estimate the extractor could read, or names no intervention and outcome on the map, so this paper lights no cell and moves no belief. It is still indexed, cited and linked below.

2 · The registry

The trial behind it

Trials whose registry record cites this paper, or whose number appears in the abstract. A trial that started after this paper was published is citing it as background, not reporting it.

Neither the registry nor the abstract names a trial number. If this is a trial report, that itself is worth knowing.

3 · Its place in the literature

Who cites it

2 citing papers in PubMed, 2 citations in OpenAlex.

  1. Article
  2. Review
4 · The record

Corrections and comments

PubMed lists nothing against this paper. Absence here is not a guarantee, only a check that was made.

5 · Who and what money

Authors and funding

2 authors at 1 institution in 1 country.

Simon SchemkeInstitut für Physiologie, Universität zu Lübeck, Ratzeburger Allee 160, 23562, Lübeck, Germany.
Cor de WitInstitut für Physiologie, Universität zu Lübeck, Ratzeburger Allee 160, 23562, Lübeck, Germany. dewit@uni-luebeck.de.ORCID http://orcid.org/0000-0003-4621-0399
German Centre for Cardiovascular Research · DE

Funding

No grant is acknowledged in the PubMed record.

6 · The paper itself

Abstract

Functional hyperemia is fundamental to provide enhanced oxygen delivery during exercise in skeletal muscle. Different mechanisms are suggested to contribute, mediators from skeletal muscle, transmitter spillover from the neuromuscular synapse as well as endothelium-related dilators. We hypothesized that redundant mechanisms that invoke adenosine, endothelial autacoids, and K

Indexed as

AcetylcholineAdenosineAdenosine TriphosphateAnimalsArteriolesDilatationElectric StimulationKATP ChannelsMaleMiceMice, Inbred C57BLMuscle ContractionMuscle, SkeletalNitric OxideProstaglandinsVasodilationAcetylcholineAdenosineAdenosine TriphosphateKATP ChannelsNitric OxideProstaglandinsActive hyperemiaAdenosineEndothelial autacoidsGlibenclamideKATP channels

Identifiers

PMID34386847
PMCPMC8528760
OpenAlexW3190170795

What OpenQuestion holds

Textmetadata
LicenceCC BY
Read underepoch 390

Registered trials

None linked

Read under generation 80e0d062 · epoch 390. Bibliography from PubMed, PubMed Central and OpenAlex; grants from NIH RePORTER; trial links from ClinicalTrials.gov; estimates, votes and beliefs from the OpenQuestion graph.