Evidence map›Paper›PMID 34381982›Full record

ArticleiScience2021

Transgenic modeling of Ndr2 gene amplification reveals disturbance of hippocampus circuitry and function.

Deniz A Madencioglu, Gürsel Çalışkan, Pingan Yuanxiang, Kati Rehberg, Yunus E Demiray, Emre Kul, Alexander Engler, Hussam Hayani, Jorge R Bergado-Acosta, Anne Kummer and 6 more

Open access · goldAbstract read
In one paragraph

Article in iScience, 2021. The graph could read no effect estimate from its abstract, so it casts no vote on the map. Cited by 4 papers.

0numbers the graph read from it
0cells of the map it votes in
4citing papers in PubMed
0.9field-weighted citation impact, top 23% of its field
1 · What the graph read from it

What it found

Each row is one number read from the abstract, on the scale the paper reported it, with its interval. Left of the dashed line favours the treatment, right favours the comparator. Under each row is the sentence it came from. New to these charts? A ten-minute tutorial.

The abstract states no effect estimate the extractor could read, or names no intervention and outcome on the map, so this paper lights no cell and moves no belief. It is still indexed, cited and linked below.

2 · The registry

The trial behind it

Trials whose registry record cites this paper, or whose number appears in the abstract. A trial that started after this paper was published is citing it as background, not reporting it.

Neither the registry nor the abstract names a trial number. If this is a trial report, that itself is worth knowing.

3 · Its place in the literature

Who cites it

4 citing papers in PubMed, 8 citations in OpenAlex.

  1. Article
  2. Article
  3. The NDR family of kinases: essential regulators of aging.Frontiers in molecular neuroscience · 2024
    Review
  4. Article
4 · The record

Corrections and comments

PubMed lists nothing against this paper. Absence here is not a guarantee, only a check that was made.

5 · Who and what money

Authors and funding

16 authors at 5 institutions in 1 country.

Deniz A MadenciogluDepartment of Genetics and Molecular Neurobiology, Institute of Biology, Otto-von-Guericke-University, 39120 Magdeburg, Germany.
Gürsel ÇalışkanDepartment of Genetics and Molecular Neurobiology, Institute of Biology, Otto-von-Guericke-University, 39120 Magdeburg, Germany.
Pingan YuanxiangResearch Group Neuroplasticity, Leibniz Institute for Neurobiology, 39112Magdeburg, Germany.
Kati RehbergDepartment of Genetics and Molecular Neurobiology, Institute of Biology, Otto-von-Guericke-University, 39120 Magdeburg, Germany.
Yunus E DemirayDepartment of Genetics and Molecular Neurobiology, Institute of Biology, Otto-von-Guericke-University, 39120 Magdeburg, Germany.
Emre KulDepartment of Genetics and Molecular Neurobiology, Institute of Biology, Otto-von-Guericke-University, 39120 Magdeburg, Germany.
Alexander EnglerInstitute of Experimental Internal Medicine, Otto-von-Guericke-University, 39120Magdeburg, Germany.
Hussam HayaniMolecular Neuroplasticity Group, German Center for Neurodegenerative Diseases, 39120Magdeburg, Germany.
Jorge R Bergado-AcostaDepartment of Genetics and Molecular Neurobiology, Institute of Biology, Otto-von-Guericke-University, 39120 Magdeburg, Germany.
Anne KummerDepartment of Genetics and Molecular Neurobiology, Institute of Biology, Otto-von-Guericke-University, 39120 Magdeburg, Germany.
Iris MüllerDepartment of Genetics and Molecular Neurobiology, Institute of Biology, Otto-von-Guericke-University, 39120 Magdeburg, Germany.
Inseon SongMolecular Neuroplasticity Group, German Center for Neurodegenerative Diseases, 39120Magdeburg, Germany.
Alexander DityatevMolecular Neuroplasticity Group, German Center for Neurodegenerative Diseases, 39120Magdeburg, Germany.
Thilo KähneInstitute of Experimental Internal Medicine, Otto-von-Guericke-University, 39120Magdeburg, Germany.
Michael R KreutzResearch Group Neuroplasticity, Leibniz Institute for Neurobiology, 39112Magdeburg, Germany.
Oliver StorkDepartment of Genetics and Molecular Neurobiology, Institute of Biology, Otto-von-Guericke-University, 39120 Magdeburg, Germany.
Otto-von-Guericke-Universität Magdeburg · DECenter for Behavioral Brain Sciences · DEGerman Center for Neurodegenerative Diseases · DELeibniz Institute for Neurobiology · DEUniversität Hamburg · DE

Funding

Wellcome Trust
6 · The paper itself

Abstract

Duplications and deletions of short chromosomal fragments are increasingly recognized as the cause for rare neurodevelopmental conditions and disorders. The

Indexed as

geneticsneurosciencesensory neuroscience

Identifiers

PMID34381982
PMCPMC8340122
OpenAlexW3186221385

What OpenQuestion holds

Textmetadata
LicenceCC BY-NC-ND
Read underepoch 390

Registered trials

None linked

Read under generation 80e0d062 · epoch 390. Bibliography from PubMed, PubMed Central and OpenAlex; grants from NIH RePORTER; trial links from ClinicalTrials.gov; estimates, votes and beliefs from the OpenQuestion graph.