Evidence map›Paper›PMID 34356361›Full record

ArticleAntioxidants (Basel, Switzerland)2021

GSNOR Contributes to Demethylation and Expression of Transposable Elements and Stress-Responsive Genes.

Eva Esther Rudolf, Patrick Hüther, Ignasi Forné, Elisabeth Georgii, Yongtao Han, Rüdiger Hell, Markus Wirtz, Axel Imhof, Claude Becker, Jörg Durner and 1 more

Open access · goldAbstract read
In one paragraph

Article in Antioxidants (Basel, Switzerland), 2021. The graph could read no effect estimate from its abstract, so it casts no vote on the map. Cited by 11 papers.

0numbers the graph read from it
0cells of the map it votes in
11citing papers in PubMed
2.7field-weighted citation impact, top 10% of its field
1 · What the graph read from it

What it found

Each row is one number read from the abstract, on the scale the paper reported it, with its interval. Left of the dashed line favours the treatment, right favours the comparator. Under each row is the sentence it came from. New to these charts? A ten-minute tutorial.

The abstract states no effect estimate the extractor could read, or names no intervention and outcome on the map, so this paper lights no cell and moves no belief. It is still indexed, cited and linked below.

2 · The registry

The trial behind it

Trials whose registry record cites this paper, or whose number appears in the abstract. A trial that started after this paper was published is citing it as background, not reporting it.

Neither the registry nor the abstract names a trial number. If this is a trial report, that itself is worth knowing.

3 · Its place in the literature

Who cites it

11 citing papers in PubMed, 17 citations in OpenAlex.

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  8. Nitric Oxide Implication in Potato Immunity toInternational journal of molecular sciences · 2022
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4 · The record

Corrections and comments

PubMed lists nothing against this paper. Absence here is not a guarantee, only a check that was made.

5 · Who and what money

Authors and funding

11 authors at 4 institutions in 2 countries.

Eva Esther RudolfInstitute of Biochemical Plant Pathology, Helmholtz Zentrum München, German Research Center for Environmental Health, 85764 Oberschleißheim, Germany.ORCID 0000-0002-1727-9525
Patrick HütherGregor Mendel Institute of Molecular Plant Biology, Austrian Academy Sciences, Vienna BioCenter (VBC), 1030 Wien, Austria.ORCID 0000-0003-3315-2484
Ignasi FornéProtein Analysis Unit, Ludwig-Maximilians-Universität München, 82152 Martinsried, Germany.ORCID 0000-0003-0309-907X
Elisabeth GeorgiiInstitute of Biochemical Plant Pathology, Helmholtz Zentrum München, German Research Center for Environmental Health, 85764 Oberschleißheim, Germany.ORCID 0000-0002-7511-5510
Yongtao HanInstitute of Biochemical Plant Pathology, Helmholtz Zentrum München, German Research Center for Environmental Health, 85764 Oberschleißheim, Germany.
Rüdiger HellCentre for Organismal Studies, Ruprecht-Karls-Universität Heidelberg, 69120 Heidelberg, Germany.ORCID 0000-0002-6238-4818
Markus WirtzCentre for Organismal Studies, Ruprecht-Karls-Universität Heidelberg, 69120 Heidelberg, Germany.ORCID 0000-0001-7790-4022
Axel ImhofProtein Analysis Unit, Ludwig-Maximilians-Universität München, 82152 Martinsried, Germany.ORCID 0000-0003-2993-8249
Claude BeckerGregor Mendel Institute of Molecular Plant Biology, Austrian Academy Sciences, Vienna BioCenter (VBC), 1030 Wien, Austria.
Jörg DurnerInstitute of Biochemical Plant Pathology, Helmholtz Zentrum München, German Research Center for Environmental Health, 85764 Oberschleißheim, Germany.
Christian LindermayrInstitute of Biochemical Plant Pathology, Helmholtz Zentrum München, German Research Center for Environmental Health, 85764 Oberschleißheim, Germany.
Helmholtz Zentrum München · DEGregor Mendel Institute of Molecular Plant Biology · ATHeidelberg University · DELudwig-Maximilians-Universität München · DE

Funding

Deutsche Forschungsgemeinschaft HE 1848/15-2Deutsche Forschungsgemeinschaft WI 3560/1-2Deutsche Forschungsgemeinschaft WI 3560/4-1Deutsche Forschungsgemeinschaft ZUK 49/2010-3009262
6 · The paper itself

Abstract

In the past, reactive nitrogen species (RNS) were supposed to be stress-induced by-products of disturbed metabolism that cause oxidative damage to biomolecules. However, emerging evidence demonstrates a substantial role of RNS as endogenous signals in eukaryotes. In plants, S-nitrosoglutathione (GSNO) is the dominant RNS and serves as the

Indexed as

DNA methylationhistone methylationmetaboloepigeneticnitric oxideS-adenosylhomocysteineS-nitrosoglutathioneS-nitrosoglutathione reductase

Identifiers

PMID34356361
PMCPMC8301139
OpenAlexW3182278987

What OpenQuestion holds

Textmetadata
LicenceCC BY
Read underepoch 390

Registered trials

None linked

Read under generation 80e0d062 · epoch 390. Bibliography from PubMed, PubMed Central and OpenAlex; grants from NIH RePORTER; trial links from ClinicalTrials.gov; estimates, votes and beliefs from the OpenQuestion graph.