Evidence map›Paper›PMID 34267322›Full record

ArticleCommunications biology2021

Essential roles of plexin-B3

Naomi Nihonmatsu-Kikuchi, Xiu-Jun Yu, Yoshiki Matsuda, Nobuyuki Ozawa, Taeko Ito, Kazuhito Satou, Tadashi Kaname, Yasushi Iwasaki, Akio Akagi, Mari Yoshida and 6 more

Erratum issuedOpen access · goldAbstract read
PubMed Full text at PMC Publisher
In one paragraph

Article in Communications biology, 2021. The graph could read no effect estimate from its abstract, so it casts no vote on the map. An erratum has been issued. Cited by 9 papers.

0numbers the graph read from it
0cells of the map it votes in
9citing papers in PubMed
1.6field-weighted citation impact, top 19% of its field
1 · What the graph read from it

What it found

Each row is one number read from the abstract, on the scale the paper reported it, with its interval. Left of the dashed line favours the treatment, right favours the comparator. Under each row is the sentence it came from. New to these charts? A ten-minute tutorial.

The abstract states no effect estimate the extractor could read, or names no intervention and outcome on the map, so this paper lights no cell and moves no belief. It is still indexed, cited and linked below.

2 · The registry

The trial behind it

Trials whose registry record cites this paper, or whose number appears in the abstract. A trial that started after this paper was published is citing it as background, not reporting it.

Neither the registry nor the abstract names a trial number. If this is a trial report, that itself is worth knowing.

3 · Its place in the literature

Who cites it

9 citing papers in PubMed, 16 citations in OpenAlex.

  1. Emerging Role of Oligodendrocytes Malfunction in the Progression of Alzheimer's Disease.Journal of neuroimmune pharmacology : the official journal of the Society on NeuroImmune Pharmacology · 2025
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4 · The record

Corrections and comments

5 · Who and what money

Authors and funding

16 authors at 6 institutions in 2 countries.

Naomi Nihonmatsu-KikuchiAffective Disorders Research Project, Tokyo Metropolitan Institute of Medical Science, Setagaya, Tokyo, Japan.
Xiu-Jun YuAffective Disorders Research Project, Tokyo Metropolitan Institute of Medical Science, Setagaya, Tokyo, Japan.
Yoshiki MatsudaAffective Disorders Research Project, Tokyo Metropolitan Institute of Medical Science, Setagaya, Tokyo, Japan.ORCID http://orcid.org/0000-0002-9517-151X
Nobuyuki OzawaAffective Disorders Research Project, Tokyo Metropolitan Institute of Medical Science, Setagaya, Tokyo, Japan.
Taeko ItoAffective Disorders Research Project, Tokyo Metropolitan Institute of Medical Science, Setagaya, Tokyo, Japan.
Kazuhito SatouDepartment of Genome Medicine, National Center for Child Health and Development, Setagaya, Tokyo, Japan.
Tadashi KanameDepartment of Genome Medicine, National Center for Child Health and Development, Setagaya, Tokyo, Japan.ORCID http://orcid.org/0000-0003-0281-9610
Yasushi IwasakiInstitute for Medical Science for Aging, Aichi Medical University, Nagakute, Aichi, Japan.
Akio AkagiInstitute for Medical Science for Aging, Aichi Medical University, Nagakute, Aichi, Japan.
Mari YoshidaInstitute for Medical Science for Aging, Aichi Medical University, Nagakute, Aichi, Japan.
Shuta ToruDepartment of Neurology, Nitobe Memorial Nakano General Hospital, Nakano, Tokyo, Japan.
Katsuiku HirokawaDepartment of Neurology, Nitobe Memorial Nakano General Hospital, Nakano, Tokyo, Japan.
Akihiko TakashimaDepartment of Life Science, Gakushuin University Graduate School of Science, Toshima, Tokyo, Japan.
Masato HasegawaDementia Research Project, Tokyo Metropolitan Institute of Medical Science, Setagaya, Tokyo, Japan.
Toshiki UchiharaAffective Disorders Research Project, Tokyo Metropolitan Institute of Medical Science, Setagaya, Tokyo, Japan.
Yoshitaka TatebayashiAffective Disorders Research Project, Tokyo Metropolitan Institute of Medical Science, Setagaya, Tokyo, Japan. tatebayashi-ys@igakuken.or.jp.ORCID http://orcid.org/0000-0002-4662-2281
Tokyo Metropolitan Institute of Medical Science · JPAichi Medical University · JPNational Center For Child Health and Development · JPNitobe Memorial Nakano General Hospital · JPGakushuin University · JPHebei Medical University · CN

Funding

No grant is acknowledged in the PubMed record.

6 · The paper itself

Abstract

The role of oligodendrocyte lineage cells, the largest glial population in the adult central nervous system (CNS), in the pathogenesis of Alzheimer's disease (AD) remains elusive. Here, we developed a culture method for adult oligodendrocyte progenitor cells (aOPCs). Fibroblast growth factor 2 (FGF2) promotes survival and proliferation of NG2

Indexed as

Alzheimer DiseaseAmyloid beta-PeptidesAnimalsAntigensBrainCells, CulturedChondroitin Sulfate Proteoglycan 4FemaleHumansMaleNerve Tissue ProteinsNeural Cell Adhesion MoleculesOligodendrocyte Precursor CellsOligodendrogliaPeptide FragmentsProteoglycansAmyloid beta-Peptidesamyloid beta-protein (1-42)AntigensChondroitin Sulfate Proteoglycan 4Nerve Tissue ProteinsNeural Cell Adhesion MoleculesPeptide FragmentsPLXNB3 protein, humanPlxnb3 protein, ratProteoglycans

Identifiers

PMID34267322
PMCPMC8282672
OpenAlexW3180973804

What OpenQuestion holds

Textmetadata
LicenceCC BY
Read underepoch 390

Registered trials

None linked

Read under generation 80e0d062 · epoch 390. Bibliography from PubMed, PubMed Central and OpenAlex; grants from NIH RePORTER; trial links from ClinicalTrials.gov; estimates, votes and beliefs from the OpenQuestion graph.