Evidence map›Paper›PMID 34239739›Full record

ArticleCase reports in endocrinology2021

Hypokalemic Periodic Paralysis Precipitated by Thyrotoxicosis and Renal Tubular Acidosis.

Ian Jackson, Yazan Addasi, Moeed Ahmed, Bashar Ramadan, Karson Kalian, Noor Addasi, Ali Nayfeh, Jocelyn Taylor, Khalid Bashir, Bryan Krajicek

Open access · goldAbstract readCase Reports
In one paragraph

Article in Case reports in endocrinology, 2021. The graph could read no effect estimate from its abstract, so it casts no vote on the map. Cited by 9 papers.

0numbers the graph read from it
0cells of the map it votes in
9citing papers in PubMed
0.6field-weighted citation impact, top 36% of its field
1 · What the graph read from it

What it found

Each row is one number read from the abstract, on the scale the paper reported it, with its interval. Left of the dashed line favours the treatment, right favours the comparator. Under each row is the sentence it came from. New to these charts? A ten-minute tutorial.

The abstract states no effect estimate the extractor could read, or names no intervention and outcome on the map, so this paper lights no cell and moves no belief. It is still indexed, cited and linked below.

2 · The registry

The trial behind it

Trials whose registry record cites this paper, or whose number appears in the abstract. A trial that started after this paper was published is citing it as background, not reporting it.

Neither the registry nor the abstract names a trial number. If this is a trial report, that itself is worth knowing.

3 · Its place in the literature

Who cites it

9 citing papers in PubMed, 10 citations in OpenAlex.

  1. Review
  2. Thyrotoxic Hypokalemic Periodic Paralysis: Pathophysiological Mechanisms.Endocrinology and metabolism (Seoul, Korea) · 2025
    Review
  3. Article
  4. Article
  5. Review
  6. Article
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4 · The record

Corrections and comments

PubMed lists nothing against this paper. Absence here is not a guarantee, only a check that was made.

5 · Who and what money

Authors and funding

10 authors at 2 institutions in 1 country.

Ian JacksonDepartment of Medicine, Creighton University School of Medicine, Omaha, NE, USA.ORCID https://orcid.org/0000-0003-1878-1020
Yazan AddasiDepartment of Medicine, Creighton University School of Medicine, Omaha, NE, USA.ORCID https://orcid.org/0000-0001-7448-6507
Moeed AhmedDepartment of Medicine, Creighton University School of Medicine, Omaha, NE, USA.ORCID https://orcid.org/0000-0001-8280-486X
Bashar RamadanDepartment of Medicine, Creighton University School of Medicine, Omaha, NE, USA.ORCID https://orcid.org/0000-0003-2102-0900
Karson KalianDepartment of Medicine, Creighton University School of Medicine, Omaha, NE, USA.ORCID https://orcid.org/0000-0003-1469-5766
Noor AddasiDepartment of Endocrinology, University of Nebraska Medical Center, Omaha, NE, USA.ORCID https://orcid.org/0000-0003-3192-2297
Ali NayfehDepartment of Pulmonary and Critical Care, Creighton University School of Medicine, Omaha, NE, USA.ORCID https://orcid.org/0000-0001-7852-6203
Jocelyn TaylorDepartment of Medicine, Creighton University School of Medicine, Omaha, NE, USA.ORCID https://orcid.org/0000-0003-3441-2065
Khalid BashirDepartment of Nephrology, Creighton University School of Medicine, Omaha, NE, USA.ORCID https://orcid.org/0000-0003-1621-6212
Bryan KrajicekDepartment of Pulmonary and Critical Care, Creighton University School of Medicine, Omaha, NE, USA.ORCID https://orcid.org/0000-0002-5995-5982
Creighton University · USUniversity of Nebraska Medical Center · US

Funding

No grant is acknowledged in the PubMed record.

6 · The paper itself

Abstract

backgroundHypokalemic periodic paralysis is a rare neuromuscular disorder characterized by transient episodes of flaccid paralysis due to a defect in muscle ion channels. Most cases are hereditary, but it can be acquired. We present a case of acquired hypokalemic periodic paralysis associated with hyperthyroidism and renal tubular acidosis.

conclusionEarly recognition and treatment of thyrotoxic periodic paralysis are important to prevent cardiac complications. Management includes potassium replacement with careful monitoring to prevent rebound hyperkalemia. The definitive treatment is to achieve euthyroid status.

Identifiers

PMID34239739
PMCPMC8238611
OpenAlexW3174954520

What OpenQuestion holds

Textmetadata
LicenceCC BY
Read underepoch 390

Registered trials

None linked

Read under generation 80e0d062 · epoch 390. Bibliography from PubMed, PubMed Central and OpenAlex; grants from NIH RePORTER; trial links from ClinicalTrials.gov; estimates, votes and beliefs from the OpenQuestion graph.