Evidence map›Paper›PMID 34237823›Full record

ReviewThe Korean journal of internal medicine2021

Clinical and genetic characteristics of Korean autosomal dominant polycystic kidney disease patients.

Yun Kyu Oh, Hayne Cho Park, Hyunjin Ryu, Yong-Chul Kim, Kook-Hwan Oh

Open access · goldAbstract readReview
In one paragraph

Review in The Korean journal of internal medicine, 2021. The graph could read no effect estimate from its abstract, so it casts no vote on the map. Cited by 14 papers.

0numbers the graph read from it
0cells of the map it votes in
14citing papers in PubMed
2.1field-weighted citation impact, top 12% of its field
1 · What the graph read from it

What it found

Each row is one number read from the abstract, on the scale the paper reported it, with its interval. Left of the dashed line favours the treatment, right favours the comparator. Under each row is the sentence it came from. New to these charts? A ten-minute tutorial.

The abstract states no effect estimate the extractor could read, or names no intervention and outcome on the map, so this paper lights no cell and moves no belief. It is still indexed, cited and linked below.

2 · The registry

The trial behind it

Trials whose registry record cites this paper, or whose number appears in the abstract. A trial that started after this paper was published is citing it as background, not reporting it.

Neither the registry nor the abstract names a trial number. If this is a trial report, that itself is worth knowing.

3 · Its place in the literature

Who cites it

14 citing papers in PubMed, 13 citations in OpenAlex.

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4 · The record

Corrections and comments

PubMed lists nothing against this paper. Absence here is not a guarantee, only a check that was made.

5 · Who and what money

Authors and funding

5 authors at 4 institutions in 1 country.

Yun Kyu OhDepartment of Internal Medicine, Seoul National University College of Medicine, Seoul, Korea.
Hayne Cho ParkDepartment of Internal Medicine, Hallym University Kangnam Sacred Heart Hospital, Seoul, Korea.
Hyunjin RyuDepartment of Internal Medicine, Seoul National University College of Medicine, Seoul, Korea.
Yong-Chul KimDepartment of Internal Medicine, Seoul National University College of Medicine, Seoul, Korea.
Kook-Hwan OhDepartment of Internal Medicine, Seoul National University College of Medicine, Seoul, Korea.
Seoul National University · KRHallym University Kangnam Sacred Heart Hospital · KRSeoul National University Hospital · KRSNUH SMG-SNU Boramae Medical Center · KR

Funding

Korea Disease Control and Prevention Agency 2019-ER-7304-00Korea Disease Control and Prevention Agency 2019-ER-7304-01
6 · The paper itself

Abstract

Autosomal dominant polycystic kidney disease (ADPKD) is the most common hereditary kidney disease. It is characterized by cyst growth in the kidneys, resulting in kidney enlargement and end-stage kidney disease. The polycystic kidney disease 1 (PKD1) and PKD2 have been identified as genes related to ADPKD and their significance in the molecular pathology of the disease has been studied. A disease-modifying drug has been approved; therefore, it has become important to identify patients at a high risk of kidney disease progression. Genetic tests, image analysis methods, and clinical factors for kidney disease progression prediction have been established. This review describes genetic and clinical characteristics, and discusses ongoing studies in Korean ADPKD patients.

Indexed as

Polycystic Kidney, Autosomal DominantPolycystic Kidney DiseasesHumansKidneyMutationRepublic of KoreaTRPP Cation ChannelsTRPP Cation ChannelsAutosomal dominant polycystic kidney diseaseDisease progressionPKD1PKD2Tolvaptan

Identifiers

PMID34237823
PMCPMC8273813
OpenAlexW3182122342

What OpenQuestion holds

Textmetadata
LicenceCC BY-NC
Read underepoch 390

Registered trials

None linked

Read under generation 80e0d062 · epoch 390. Bibliography from PubMed, PubMed Central and OpenAlex; grants from NIH RePORTER; trial links from ClinicalTrials.gov; estimates, votes and beliefs from the OpenQuestion graph.