Evidence map›Paper›PMID 34094710›Full record

ArticleMolecular therapy. Nucleic acids2021

lncRNA MIR210HG promotes the progression of endometrial cancer by sponging miR-337-3p/137 via the HMGA2-TGF-β/Wnt pathway.

Jian Ma, Fan-Fei Kong, Di Yang, Hui Yang, Cuicui Wang, Rong Cong, Xiao-Xin Ma

Open access · goldAbstract read
In one paragraph

Article in Molecular therapy. Nucleic acids, 2021. The graph could read no effect estimate from its abstract, so it casts no vote on the map. Cited by 28 papers.

0numbers the graph read from it
0cells of the map it votes in
28citing papers in PubMed
3.2field-weighted citation impact, top 6% of its field
1 · What the graph read from it

What it found

Each row is one number read from the abstract, on the scale the paper reported it, with its interval. Left of the dashed line favours the treatment, right favours the comparator. Under each row is the sentence it came from. New to these charts? A ten-minute tutorial.

The abstract states no effect estimate the extractor could read, or names no intervention and outcome on the map, so this paper lights no cell and moves no belief. It is still indexed, cited and linked below.

2 · The registry

The trial behind it

Trials whose registry record cites this paper, or whose number appears in the abstract. A trial that started after this paper was published is citing it as background, not reporting it.

Neither the registry nor the abstract names a trial number. If this is a trial report, that itself is worth knowing.

3 · Its place in the literature

Who cites it

28 citing papers in PubMed, 43 citations in OpenAlex.

  1. Article
  2. LncRNAs signatures in gynecological cancers: ovarian and endometrial cancers.Clinical & translational oncology : official publication of the Federation of Spanish Oncology Societies and of the National Cancer Institute of Mexico · 2026
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4 · The record

Corrections and comments

PubMed lists nothing against this paper. Absence here is not a guarantee, only a check that was made.

5 · Who and what money

Authors and funding

7 authors at 1 institution in 1 country.

Jian MaDepartment of Obstetrics and Gynecology, Shengjing Hospital of China Medical University, Shenyang 110004, China.
Fan-Fei KongDepartment of Obstetrics and Gynecology, Shengjing Hospital of China Medical University, Shenyang 110004, China.
Di YangDepartment of Obstetrics and Gynecology, Shengjing Hospital of China Medical University, Shenyang 110004, China.
Hui YangDepartment of Obstetrics and Gynecology, Shengjing Hospital of China Medical University, Shenyang 110004, China.
Cuicui WangDepartment of Obstetrics and Gynecology, Shengjing Hospital of China Medical University, Shenyang 110004, China.
Rong CongDepartment of Obstetrics and Gynecology, Shengjing Hospital of China Medical University, Shenyang 110004, China.
Xiao-Xin MaDepartment of Obstetrics and Gynecology, Shengjing Hospital of China Medical University, Shenyang 110004, China.
China Medical University · CN

Funding

No grant is acknowledged in the PubMed record.

6 · The paper itself

Abstract

Epithelial-mesenchymal transition (EMT) promotes tumorigenesis and metastasis and increases tumor tolerance to treatment intervention. Abnormal activation of transforming growth factor β (TGF-β) and Wnt pathway induces EMT. Long non-coding RNAs (lncRNAs) significantly influence EMT regulation. Herein, we show that MIR210HG is overexpressed in endometrial cancer tissues, which is associated with poor prognosis. MIR210HG silencing significantly inhibited proliferation, migration, invasion, and EMT phenotype formation

Indexed as

EMTendometrial cancermiR-137MIR210HGprognosis

Identifiers

PMID34094710
PMCPMC8141672
OpenAlexW3156859226

What OpenQuestion holds

Textmetadata
LicenceCC BY-NC-ND
Read underepoch 390

Registered trials

None linked

Read under generation 80e0d062 · epoch 390. Bibliography from PubMed, PubMed Central and OpenAlex; grants from NIH RePORTER; trial links from ClinicalTrials.gov; estimates, votes and beliefs from the OpenQuestion graph.