Evidence map›Paper›PMID 33987182›Full record

ReviewFrontiers in cell and developmental biology2021

Zebrafish Cancer Predisposition Models.

Kim Kobar, Keon Collett, Sergey V Prykhozhij, Jason N Berman

Open access · goldAbstract readReview
In one paragraph

Review in Frontiers in cell and developmental biology, 2021. The graph could read no effect estimate from its abstract, so it casts no vote on the map. Cited by 20 papers.

0numbers the graph read from it
0cells of the map it votes in
20citing papers in PubMed
1.6field-weighted citation impact, top 16% of its field
1 · What the graph read from it

What it found

Each row is one number read from the abstract, on the scale the paper reported it, with its interval. Left of the dashed line favours the treatment, right favours the comparator. Under each row is the sentence it came from. New to these charts? A ten-minute tutorial.

The abstract states no effect estimate the extractor could read, or names no intervention and outcome on the map, so this paper lights no cell and moves no belief. It is still indexed, cited and linked below.

2 · The registry

The trial behind it

Trials whose registry record cites this paper, or whose number appears in the abstract. A trial that started after this paper was published is citing it as background, not reporting it.

Neither the registry nor the abstract names a trial number. If this is a trial report, that itself is worth knowing.

3 · Its place in the literature

Who cites it

20 citing papers in PubMed, 32 citations in OpenAlex.

  1. Review
  2. Bony fish genomes: Status and gaps.Journal of fish biology · 2026
    Review
  3. Article
  4. Review
  5. Review
  6. Review
  7. Consequences of Cancer on ZebrafishEcology and evolution · 2025
    Article
  8. Review
  9. Article
  10. Review
  11. Review
  12. Review
  13. Article
  14. Review
  15. Review
  16. Article
  17. Article
  18. Zebrafish-An Optimal Model in Experimental Oncology.Molecules (Basel, Switzerland) · 2022
    Review
  19. Zebrafish models of inflammation in hematopoietic development and disease.Frontiers in cell and developmental biology · 2022
    Review
  20. Article
4 · The record

Corrections and comments

PubMed lists nothing against this paper. Absence here is not a guarantee, only a check that was made.

5 · Who and what money

Authors and funding

4 authors at 2 institutions in 1 country.

Kim KobarChildren's Hospital of Eastern Ontario Research Institute, Ottawa, ON, Canada.
Keon CollettChildren's Hospital of Eastern Ontario Research Institute, Ottawa, ON, Canada.
Sergey V PrykhozhijChildren's Hospital of Eastern Ontario Research Institute, Ottawa, ON, Canada.
Jason N BermanChildren's Hospital of Eastern Ontario Research Institute, Ottawa, ON, Canada.
Children's Hospital of Eastern Ontario · CAUniversity of Ottawa · CA

Funding

No grant is acknowledged in the PubMed record.

6 · The paper itself

Abstract

Cancer predisposition syndromes are rare, typically monogenic disorders that result from germline mutations that increase the likelihood of developing cancer. Although these disorders are individually rare, resulting cancers collectively represent 5-10% of all malignancies. In addition to a greater incidence of cancer, affected individuals have an earlier tumor onset and are frequently subjected to long-term multi-modal cancer screening protocols for earlier detection and initiation of treatment.

Indexed as

cancercancer predispositiongenetic modelsmodel organismp53zebrafish

Identifiers

PMID33987182
PMCPMC8112447
OpenAlexW3158611454

What OpenQuestion holds

Textmetadata
LicenceCC BY
Read underepoch 390

Registered trials

None linked

Read under generation 80e0d062 · epoch 390. Bibliography from PubMed, PubMed Central and OpenAlex; grants from NIH RePORTER; trial links from ClinicalTrials.gov; estimates, votes and beliefs from the OpenQuestion graph.